Extracellular hepatitis C virus core protein activates STAT3 in human monocytes/macrophages/dendritic cells via an IL-6 autocrine pathway. - Wikidata - thesis-toulmin - TriplyDB

Extracellular hepatitis C virus core protein activates STAT3 in human monocytes/macrophages/dendritic cells via an IL-6 autocrine pathway.

Extracellular hepatitis C virus core protein activates STAT3 in human monocytes/macrophages/dendritic cells via an IL-6 autocrine pathway.

http://www.wikidata.org/entity/Q30427748

about

Dendritic cells: The warriors upfront-turned defunct in chronic hepatitis C infection The Complex Role of STAT3 in Viral Infections Direct effects of hepatitis C virus on the lymphoid cells Hepatitis C Virus-Induced Myeloid-Derived Suppressor Cells Suppress NK Cell IFN-γ Production by Altering Cellular Metabolism via Arginase-1.Myeloid-derived suppressor cells: the dark knight or the joker in viral infections?Hepatitis C virus promotes T-helper (Th)17 responses through thymic stromal lymphopoietin production by infected hepatocytes.Myeloid suppressor cells induced by hepatitis C virus suppress T-cell responses through the production of reactive oxygen species.Age, hepatitis C virus infection, and the risk of cancer: Unlocking a complex puzzle of interactions.Hepatitis C virus core protein inhibits interferon production by a human plasmacytoid dendritic cell line and dysregulates interferon regulatory factor-7 and signal transducer and activator of transcription (STAT) 1 protein expression.Paradoxical myeloid-derived suppressor cell reduction in the bone marrow of SIV chronically infected macaques.Hepcidin and the iron enigma in HCV infection.Production and pathogenicity of hepatitis C virus core gene products.An unbalanced PD-L1/CD86 ratio in CD14(++)CD16(+) monocytes is correlated with HCV viremia during chronic HCV infection.Hepatocarcinogenesis associated with hepatitis B, delta and C viruses.Hepatitis C virus core protein enhances HIV-1 replication in human macrophages through TLR2, JNK, and MEK1/2-dependent upregulation of TNF-α and IL-6.MicroRNA expression profiling in PBMCs: a potential diagnostic biomarker of chronic hepatitis C MicroRNA 21 is a homeostatic regulator of macrophage polarization and prevents prostaglandin E2-mediated M2 generation.Pathogenesis and prevention of hepatitis C virus-induced hepatocellular carcinoma.An HNF4α-miRNA inflammatory feedback circuit regulates hepatocellular oncogenesis Substitution in Amino Acid 70 of Hepatitis C Virus Core Protein Changes the Adipokine Profile via Toll-Like Receptor 2/4 Signaling.Hepatitis C virus-induced hepatocellular carcinoma Hepatitis C Virus Induces MDSCs-Like Monocytes through TLR2/PI3K/AKT/STAT3 Signaling.Deletion of interleukin-6 in monocytes/macrophages suppresses the initiation of hepatocellular carcinoma in mice.HCV core protein binds to gC1qR to induce A20 expression and inhibit cytokine production through MAPKs and NF-κB signaling pathways.Genetic association of interleukin-6 polymorphism (-174 G/C) with chronic liver diseases and hepatocellular carcinoma Insights into Myeloid-Derived Suppressor Cells in Inflammatory Diseases.The Role of Myeloid-Derived Suppressor Cells in Viral Infection.STAT3‑regulated long non‑coding RNAs lnc‑7SK and lnc‑IGF2‑AS promote hepatitis C virus replication A complex signaling network involving protein kinase CK2 is required for hepatitis C virus core protein-mediated modulation of the iron-regulatory hepcidin gene expression.Immune modulation by the hepatitis C virus core protein.STAT3 activation in infection and infection-associated cancer.Decline of miR-124 in myeloid cells promotes regulatory T-cell development in hepatitis C virus infection.Hepatitis C virus-induced myeloid-derived suppressor cells regulate T-cell differentiation and function via the signal transducer and activator of transcription 3 pathway.Differential association of STAT3 and HK-II expression in hepatitis B virus- and hepatitis C virus-related hepatocellular carcinoma.Hepatitis C virus core protein interacts with Snail and histone deacetylases to promote the metastasis of hepatocellular carcinoma.Functional polymorphisms of TLR8 are associated with hepatitis C virus infection.DNA methyltransferases 1 and 3b expression in Huh-7 cells expressing HCV core protein of different genotypes.Double filtration plasmapheresis could induce a favorable immune response for chronic hepatitis C therapy.Rapid reduction of hepatitis C virus-Core protein in the peripheral blood improve the immunological response in chronic hepatitis C patients.The IL-6/STAT3 pathway upregulates microRNA-125b expression in hepatitis C virus infection.

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Extracellular hepatitis C virus core protein activates STAT3 in human monocytes/macrophages/dendritic cells via an IL-6 autocrine pathway.

http://www.wikidata.org/entity/Q30427748

description

2011 nî lūn-bûn

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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հունվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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name

Extracellular hepatitis C viru ...... via an IL-6 autocrine pathway.

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Extracellular hepatitis C viru ...... via an IL-6 autocrine pathway.

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Extracellular hepatitis C viru ...... via an IL-6 autocrine pathway.

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Extracellular hepatitis C viru ...... via an IL-6 autocrine pathway.

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Extracellular hepatitis C viru ...... via an IL-6 autocrine pathway.

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Journal of Biological Chemistry

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Extracellular hepatitis C viru ...... via an IL-6 autocrine pathway.

@en

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Annie Tosello-Trampont

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Robert S Tacke

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Virginia Nguyen

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Young S Hahn

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P2860

Interaction between complement receptor gC1qR and hepatitis C virus core protein inhibits T-lymphocyte proliferation.

HCV core protein interaction with gC1q receptor inhibits Th1 differentiation of CD4+ T cells via suppression of dendritic cell IL-12 production

gC1q receptor ligation selectively down-regulates human IL-12 production through activation of the phosphoinositide 3-kinase pathway

Inhibition of dendritic cell differentiation and accumulation of myeloid-derived suppressor cells in cancer is regulated by S100A9 protein

Activation of STAT3 by the Hepatitis C Virus Core Protein Leads to Cellular Transformation

Myeloid-derived suppressor cells as regulators of the immune system

Gp130 and the interleukin-6 family of cytokines

STATs in cancer inflammation and immunity: a leading role for STAT3

Analysis of a successful immune response against hepatitis C virus

Adaptive immune responses in acute and chronic hepatitis C virus infection

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10847-10855

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David W. Mullins

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3060535

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