Infectious molecular clones with the nonhomologous dimer initiation sequences found in different subtypes of human immunodeficiency virus type 1 can recombine and initiate a spreading infection in vitro.
about
The retroviral RNA dimer linkage: different structures may reflect different rolesHIV-1 reverse transcriptionCation-dependent cleavage of the duplex form of the subtype-B HIV-1 RNA dimerization initiation siteElucidation and characterization of oligonucleotide-accessible sites on HIV-2 leader region RNA.Effect of the murine leukemia virus extended packaging signal on the rates and locations of retroviral recombination.Identification of a major restriction in HIV-1 intersubtype recombination.Retroviral vectors for analysis of viral mutagenesis and recombination.Effects of varying sequence similarity on the frequency of repeat deletion during reverse transcription of a human immunodeficiency virus type 1 vector.Dimerization of HIV-1 genomic RNA of subtypes A and B: RNA loop structure and magnesium bindingConvergence of natural and artificial evolution on an RNA loop-loop interaction: the HIV-1 dimerization initiation site.Dimeric RNA recognition regulates HIV-1 genome packaging.HIV-1 RNA dimerization: It takes two to tangoMultiple barriers to recombination between divergent HIV-1 variants revealed by a dual-marker recombination assayDimerization and template switching in the 5' untranslated region between various subtypes of human immunodeficiency virus type 1.Human immunodeficiency virus type 1 genetic recombination is more frequent than that of Moloney murine leukemia virus despite similar template switching rates.Mechanisms of human immunodeficiency virus type 2 RNA packaging: efficient trans packaging and selection of RNA copackaging partners.The dimer initiation sequence stem-loop of human immunodeficiency virus type 1 is dispensable for viral replication in peripheral blood mononuclear cells.Dimer initiation signal of human immunodeficiency virus type 1: its role in partner selection during RNA copackaging and its effects on recombination.Conformations of flanking bases in HIV-1 RNA DIS kissing complexes studied by molecular dynamics.High rates of human immunodeficiency virus type 1 recombination: near-random segregation of markers one kilobase apart in one round of viral replicationPrimary T-lymphocytes rescue the replication of HIV-1 DIS RNA mutants in part by facilitating reverse transcription.Antiviral Stratagems Against HIV-1 Using RNA Interference (RNAi) Technology.The remarkable frequency of human immunodeficiency virus type 1 genetic recombinationDissecting structural transitions in the HIV-1 dimerization initiation site RNA using 2-aminopurine fluorescence.A short sequence motif in the 5' leader of the HIV-1 genome modulates extended RNA dimer formation and virus replication.Opening of the TAR hairpin in the HIV-1 genome causes aberrant RNA dimerization and packaging.Moloney murine sarcoma virus genomic RNAs dimerize via a two-step process: a concentration-dependent kissing-loop interaction is driven by initial contact between consecutive guanines.In vivo selection of Rous sarcoma virus mutants with randomized sequences in the packaging signal.Mutations of the kissing-loop dimerization sequence influence the site specificity of murine leukemia virus recombination in vivo.Nonrandom dimerization of murine leukemia virus genomic RNAs.A human immunodeficiency virus type 1-infected individual with low viral load harbors a virus variant that exhibits an in vitro RNA dimerization defect.Short-hairpin RNAs synthesized by T7 phage polymerase do not induce interferon.Coadaptive stability of interfering particles with HIV-1 when there is an evolutionary conflict.In cell mutational interference mapping experiment (in cell MIME) identifies the 5' polyadenylation signal as a dual regulator of HIV-1 genomic RNA production and packaging.
P2860
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P2860
Infectious molecular clones with the nonhomologous dimer initiation sequences found in different subtypes of human immunodeficiency virus type 1 can recombine and initiate a spreading infection in vitro.
description
1998 nî lūn-bûn
@nan
1998 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Infectious molecular clones wi ...... spreading infection in vitro.
@ast
Infectious molecular clones wi ...... spreading infection in vitro.
@en
type
label
Infectious molecular clones wi ...... spreading infection in vitro.
@ast
Infectious molecular clones wi ...... spreading infection in vitro.
@en
prefLabel
Infectious molecular clones wi ...... spreading infection in vitro.
@ast
Infectious molecular clones wi ...... spreading infection in vitro.
@en
P2093
P2860
P1433
P1476
Infectious molecular clones wi ...... a spreading infection in vitro
@en
P2093
D C St Louis
D W Ritchey
E Sanders-Buell
F E McCutchan
M O Salminen
P2860
P304
P577
1998-05-01T00:00:00Z