Control of neuronal ion channel function by glycogen synthase kinase-3: new prospective for an old kinase.
about
Intraneuronal Aβ accumulation induces hippocampal neuron hyperexcitability through A-type K(+) current inhibition mediated by activation of caspases and GSK-3.Gsk3 Signalling and Redox Status in Bipolar Disorder: Evidence from Lithium EfficacyRole of the axonal initial segment in psychiatric disorders: function, dysfunction, and interventionThe Fibroblast Growth Factor signaling pathwayThe fibroblast growth factor 14·voltage-gated sodium channel complex is a new target of glycogen synthase kinase 3 (GSK3).Activation of GSK-3β and caspase-3 occurs in Nigral dopamine neurons during the development of apoptosis activated by a striatal injection of 6-hydroxydopamineQuantitative proteomics reveals protein-protein interactions with fibroblast growth factor 12 as a component of the voltage-gated sodium channel 1.2 (nav1.2) macromolecular complex in Mammalian brain.Herpes Simplex Virus type-1 infection induces synaptic dysfunction in cultured cortical neurons via GSK-3 activation and intraneuronal amyloid-β protein accumulationCK2 activity is required for the interaction of FGF14 with voltage-gated sodium channels and neuronal excitabilityWnt signaling: role in Alzheimer disease and schizophrenia.Wnt and lithium: a common destiny in the therapy of nervous system pathologies?PPARgamma agonists rescue increased phosphorylation of FGF14 at S226 in the Tg2576 mouse model of Alzheimer's disease.Modulation of Kv7 channels and excitability in the brain.The Nav1.2 channel is regulated by GSK3.Glycogen synthase kinase 3 beta alters anxiety-, depression-, and addiction-related behaviors and neuronal activity in the nucleus accumbens shell.Membrane-tethered AKT kinase regulates basal synaptic transmission and early phase LTP expression by modulation of post-synaptic AMPA receptor level.Hippocampal neurophysiology is modified by a disease-associated C-terminal fragment of tau protein.Disparate Effects of Lithium and a GSK-3 Inhibitor on Neuronal Oscillatory Activity in Prefrontal Cortex and Hippocampus.Juvenile treatment with mGluR2/3 agonist prevents schizophrenia-like phenotypes in adult by acting through GSK3β.
P2860
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P2860
Control of neuronal ion channel function by glycogen synthase kinase-3: new prospective for an old kinase.
description
2012 nî lūn-bûn
@nan
2012 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Control of neuronal ion channe ...... prospective for an old kinase.
@ast
Control of neuronal ion channe ...... prospective for an old kinase.
@en
type
label
Control of neuronal ion channe ...... prospective for an old kinase.
@ast
Control of neuronal ion channe ...... prospective for an old kinase.
@en
prefLabel
Control of neuronal ion channe ...... prospective for an old kinase.
@ast
Control of neuronal ion channe ...... prospective for an old kinase.
@en
P2860
P356
P1476
Control of neuronal ion channe ...... prospective for an old kinase.
@en
P2093
Norelle C Wildburger
P2860
P356
10.3389/FNMOL.2012.00080
P577
2012-07-16T00:00:00Z