Altered ultrasonic vocalizations in a tuberous sclerosis mouse model of autism - Wikidata - thesis-toulmin - TriplyDB

Altered ultrasonic vocalizations in a tuberous sclerosis mouse model of autism

Altered ultrasonic vocalizations in a tuberous sclerosis mouse model of autism

http://www.wikidata.org/entity/Q30480422

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Clinical and Neurobiological Relevance of Current Animal Models of Autism Spectrum Disorders.The molecular basis of cognitive deficits in pervasive developmental disorders Potential for treatment of severe autism in tuberous sclerosis complex Mice with Dab1 or Vldlr insufficiency exhibit abnormal neonatal vocalization patterns Structure and function of neonatal social communication in a genetic mouse model of autism Altered ultrasonic vocalization and impaired learning and memory in Angelman syndrome mouse model with a large maternal deletion from Ube3a to Gabrb3.Negative allosteric modulation of the mGluR5 receptor reduces repetitive behaviors and rescues social deficits in mouse models of autism.Deletion of JMJD2B in neurons leads to defective spine maturation, hyperactive behavior and memory deficits in mouse Mourning Dr. Alfred G. Knudson: the two-hit hypothesis, tumor suppressor genes, and the tuberous sclerosis complex Found in translation: Understanding the biology and behavior of experimental traumatic brain injury Communication impairments in mice lacking Shank1: reduced levels of ultrasonic vocalizations and scent marking behavior.Autism-related deficits via dysregulated eIF4E-dependent translational control.Evaluation of Pax6 mutant rat as a model for autism.Cry, baby, cry: Expression of Distress as a Biomarker and Modulator in Autism Spectrum Disorder Deficits in social behavioral tests in a mouse model of alternating hemiplegia of childhood.An Overrepresentation of High Frequencies in the Mouse Inferior Colliculus Supports the Processing of Ultrasonic Vocalizations.Both maternal and pup genotype influence ultrasonic vocalizations and early developmental milestones in tsc2 (+/-) mice.Developmental delays and reduced pup ultrasonic vocalizations but normal sociability in mice lacking the postsynaptic cell adhesion protein neuroligin2.Sociosexual and communication deficits after traumatic injury to the developing murine brain.Transient receptor potential ankyrin 1 mediates chronic pancreatitis pain in mice.Temporal vocal features suggest different call-pattern generating mechanisms in mice and bats.Conditional knockout of tumor overexpressed gene in mouse neurons affects RNA granule assembly, granule translation, LTP and short term habituation.Maternal immune activation yields offspring displaying mouse versions of the three core symptoms of autism Automated classification of mouse pup isolation syllables: from cluster analysis to an Excel-based "mouse pup syllable classification calculator".Translational animal models of autism and neurodevelopmental disorders.Comprehensive analysis of ultrasonic vocalizations in a mouse model of fragile X syndrome reveals limited, call type specific deficits The differential effects of prenatal and/or postnatal rapamycin on neurodevelopmental defects and cognition in a neuroglial mouse model of tuberous sclerosis complex.Excitatory/inhibitory synaptic imbalance leads to hippocampal hyperexcitability in mouse models of tuberous sclerosis.Epilepsy and Autism.Rapamycin for treating Tuberous sclerosis and Autism spectrum disorders Overview of mouse models of autism spectrum disorders Absence of preference for social novelty and increased grooming in integrin β3 knockout mice: initial studies and future directions.Sera from children with autism induce autistic features which can be rescued with a CNTF small peptide mimetic in rats Mutations causing syndromic autism define an axis of synaptic pathophysiology.Graded loss of tuberin in an allelic series of brain models of TSC correlates with survival, and biochemical, histological and behavioral features.Pregnancy at Advanced Maternal Age Affects Behavior and Hippocampal Gene Expression in Mouse Offspring.Synaptic plasticity in mouse models of autism spectrum disorders.Decreased reelin expression and organophosphate pesticide exposure alters mouse behaviour and brain morphology Autistic-like behaviour and cerebellar dysfunction in Purkinje cell Tsc1 mutant mice.α5GABAA receptor deficiency causes autism-like behaviors

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Altered ultrasonic vocalizations in a tuberous sclerosis mouse model of autism

http://www.wikidata.org/entity/Q30480422

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2010 nî lūn-bûn

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Altered ultrasonic vocalizations in a tuberous sclerosis mouse model of autism

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Altered ultrasonic vocalizations in a tuberous sclerosis mouse model of autism

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Altered ultrasonic vocalizations in a tuberous sclerosis mouse model of autism

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Altered ultrasonic vocalizations in a tuberous sclerosis mouse model of autism

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Altered ultrasonic vocalizations in a tuberous sclerosis mouse model of autism

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Altered ultrasonic vocalizations in a tuberous sclerosis mouse model of autism

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A Katrin Schenk

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David M Young

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Lily Yeh Jan

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Shi-Bing Yang

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Yuh Nung Jan

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P2860

Pervasive social deficits, but normal parturition, in oxytocin receptor-deficient mice

Abnormal behavior in a chromosome-engineered mouse model for human 15q11-13 duplication seen in autism

Reversal of learning deficits in a Tsc2+/- mouse model of tuberous sclerosis

TOR signaling in growth and metabolism

Tsc2(+/-) mice develop tumors in multiple sites that express gelsolin and are influenced by genetic background

Unusual repertoire of vocalizations in the BTBR T+tf/J mouse model of autism

Reduced ultrasonic vocalizations in vasopressin 1b knockout mice.

Acquisition and expression of a socially mediated separation response

The psychopathologies of children and adolescents with tuberous sclerosis complex (TSC): a postal survey of UK families.

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