Hydrogen sulfide improves survival after cardiac arrest and cardiopulmonary resuscitation via a nitric oxide synthase 3-dependent mechanism in mice. - Wikidata - thesis-toulmin - TriplyDB

Hydrogen sulfide improves survival after cardiac arrest and cardiopulmonary resuscitation via a nitric oxide synthase 3-dependent mechanism in mice.

Hydrogen sulfide improves survival after cardiac arrest and cardiopulmonary resuscitation via a nitric oxide synthase 3-dependent mechanism in mice.

http://www.wikidata.org/entity/Q30491252

about

Hydrogen sulfide and nitric oxide are mutually dependent in the regulation of angiogenesis and endothelium-dependent vasorelaxation Hydrogen sulfide chemical biology: pathophysiological roles and detection A critical review of pharmacological significance of Hydrogen Sulfide in hypertension Role of cystathionine γ-lyase/hydrogen sulfide pathway in cardiovascular disease: a novel therapeutic strategy?Improving outcomes after cardiac arrest using NO inhalation H2S during circulatory shock: some unresolved questions Signaling molecules: hydrogen sulfide and polysulfide Pathophysiological Mechanisms in Gaseous Therapies for Severe Malaria Modes of physiologic H2S signaling in the brain and peripheral tissues Preventing ischemic brain injury after sudden cardiac arrest using NO inhalation Is pharmacological, H₂S-induced 'suspended animation' feasible in the ICU?Inhaled nitric oxide improves outcomes after successful cardiopulmonary resuscitation in mice A role for H2S in the microcirculation of newborns: the major metabolite of H2S (thiosulphate) is increased in preterm infants.Hydrogen sulfide and ischemia-reperfusion injury.Extracellular transsulfuration generates hydrogen sulfide from homocysteine and protects endothelium from redox stress Garlic extracts prevent oxidative stress, hypertrophy and apoptosis in cardiomyocytes: a role for nitric oxide and hydrogen sulfide Vascular complications of cystathionine β-synthase deficiency: future directions for homocysteine-to-hydrogen sulfide research Gaseous hydrogen sulfide protects against myocardial ischemia-reperfusion injury in mice partially independent from hypometabolism The quantitative significance of the transsulfuration enzymes for H2S production in murine tissues.Sarcoplasmic phospholamban protein is involved in the mechanisms of postresuscitation myocardial dysfunction and the cardioprotective effect of nitrite during resuscitation.Cardioprotective effects of hydrogen sulfide Beneficial effects of nitric oxide on outcomes after cardiac arrest and cardiopulmonary resuscitation in hypothermia-treated mice.Emergent role of gasotransmitters in ischemia-reperfusion injury.Hydrogen sulphide and angiogenesis: mechanisms and applications.Administration of hydrogen sulfide via extracorporeal membrane lung ventilation in sheep with partial cardiopulmonary bypass perfusion: a proof of concept study on metabolic and vasomotor effects.Farnesyltransferase inhibitor FTI-277 reduces mortality of septic mice along with improved bacterial clearance Hydrogen sulfide inhibits hypoxia- but not anoxia-induced hypoxia-inducible factor 1 activation in a von hippel-lindau- and mitochondria-dependent manner.Role of Hydrogen Sulfide in Ischemia-Reperfusion Injury.Cardioprotection by H2S engages a cGMP-dependent protein kinase G/phospholamban pathway.NaHS Protects Cochlear Hair Cells from Gentamicin-Induced Ototoxicity by Inhibiting the Mitochondrial Apoptosis Pathway Hydrogen sulfide inhibits high glucose-induced matrix protein synthesis by activating AMP-activated protein kinase in renal epithelial cells.Noninvasive detection of neural progenitor cells in living brains by MRI Hydrogen sulfide in biochemistry and medicine.Metabolic and cardiac signaling effects of inhaled hydrogen sulfide and low oxygen in male rats Hydrogen sulfide preconditioning protects against myocardial ischemia/reperfusion injury in rats through inhibition of endo/sarcoplasmic reticulum stress Hydrogen sulfide and polysulfides as signaling molecules.Hydrogen sulfide decreases reactive oxygen in a model of lung transplantation Nitric Oxide and Hydrogen Sulfide Interact When Modulating Gastric Physiological Functions in Rodents.Hydrogen sulfide stimulates ischemic vascular remodeling through nitric oxide synthase and nitrite reduction activity regulating hypoxia-inducible factor-1α and vascular endothelial growth factor-dependent angiogenesis.Additive cardioprotection by pharmacological postconditioning with hydrogen sulfide and nitric oxide donors in mouse heart: S-sulfhydration vs. S-nitrosylation.

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Hydrogen sulfide improves survival after cardiac arrest and cardiopulmonary resuscitation via a nitric oxide synthase 3-dependent mechanism in mice.

http://www.wikidata.org/entity/Q30491252

description

2009 nî lūn-bûn

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2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年学术文章

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2009年学术文章

@zh-cn

2009年学术文章

@zh-hans

2009年学术文章

@zh-my

2009年学术文章

@zh-sg

2009年學術文章

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name

Hydrogen sulfide improves surv ...... 3-dependent mechanism in mice.

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Hydrogen sulfide improves surv ...... 3-dependent mechanism in mice.

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type

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Hydrogen sulfide improves surv ...... 3-dependent mechanism in mice.

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Hydrogen sulfide improves surv ...... 3-dependent mechanism in mice.

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Hydrogen sulfide improves surv ...... 3-dependent mechanism in mice.

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Hydrogen sulfide improves surv ...... 3-dependent mechanism in mice

@en

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David J Lefer

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Fumito Ichinose

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Jia De Yu

http://www.w3.org/2001/XMLSchema#string

Kenneth D Bloch

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Masahiko Bougaki

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Shizuka Minamishima

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Yoji Andrew Minamishima

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P2860

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

H2S induces a suspended animation-like state in mice

Hydrogen sulfide attenuates myocardial ischemia-reperfusion injury by preservation of mitochondrial function

Inhaled hydrogen sulfide: a rapidly reversible inhibitor of cardiac and metabolic function in the mouse.

Protective effects of nitric oxide synthase 3 and soluble guanylate cyclase on the outcome of cardiac arrest and cardiopulmonary resuscitation in mice.

New directions for protecting the heart against ischaemia-reperfusion injury: targeting the Reperfusion Injury Salvage Kinase (RISK)-pathway.

From O2 to H2S: a landscape view of gas biology.

AMP-activated protein kinase: a key stress signaling pathway in the heart.

The emerging roles of hydrogen sulfide in the gastrointestinal tract and liver.

Role of the mitochondrial membrane permeability transition in cell death.

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