Reduced striatal dopamine underlies the attention system dysfunction in neurofibromatosis-1 mutant mice. - Wikidata - thesis-toulmin - TriplyDB

Reduced striatal dopamine underlies the attention system dysfunction in neurofibromatosis-1 mutant mice.

Reduced striatal dopamine underlies the attention system dysfunction in neurofibromatosis-1 mutant mice.

http://www.wikidata.org/entity/Q30497049

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Roles of mTOR Signaling in Brain Development RASopathies: unraveling mechanisms with animal models Neurofibromatosis type 1 and chronic neurological conditions in the United States: an administrative claims analysis.Comprehensive functional characterization of murine infantile Batten disease including Parkinson-like behavior and dopaminergic markers.Optimizing biologically targeted clinical trials for neurofibromatosis.Neurofibromatosis type 1 alternative splicing is a key regulator of Ras signaling in neurons The effects of methylphenidate on cerebral activations to salient stimuli in healthy adults.Sex Is a major determinant of neuronal dysfunction in neurofibromatosis type 1.Neurofibromatosis type 1: modeling CNS dysfunction.Skeletal muscle and motor deficits in Neurofibromatosis Type 1.Behavioral abnormalities in female mice following administration of aluminum adjuvants and the human papillomavirus (HPV) vaccine Gardasil.Motivational disturbances and effects of L-dopa administration in neurofibromatosis-1 model mice.PET imaging for attention deficit preclinical drug testing in neurofibromatosis-1 mice.Neurofibromatosis-1 heterozygosity impairs CNS neuronal morphology in a cAMP/PKA/ROCK-dependent manner Elucidating the impact of neurofibromatosis-1 germline mutations on neurofibromin function and dopamine-based learning.Behavioral and cognitive outcomes for clinical trials in children with neurofibromatosis type 1 Dopamine deficiency underlies learning deficits in neurofibromatosis-1 mice ADHD-associated dopamine transporter, latrophilin and neurofibromin share a dopamine-related locomotor signature in Drosophila Modeling cognitive dysfunction in neurofibromatosis-1 Conflict processing in juvenile patients with neurofibromatosis type 1 (NF1) and healthy controls - Two pathways to success.Developmental neurotoxicity of alcohol and anesthetic drugs is augmented by co-exposure to caffeine.Epilepsy Mechanisms in Neurocutaneous Disorders: Tuberous Sclerosis Complex, Neurofibromatosis Type 1, and Sturge-Weber Syndrome.A neuropsychological perspective on attention problems in neurofibromatosis type 1.Cognitive and behavioral problems in children with neurofibromatosis type 1: challenges and future directions.The role of the thalamus in ADHD symptomatology and treatment.Characterization of early communicative behavior in mouse models of neurofibromatosis type 1.Attention deficit hyperactivity disorder (ADHD) in phenotypically similar neurogenetic conditions: Turner syndrome and the RASopathies Human stem cell modeling in neurofibromatosis type 1 (NF1).Modeling RASopathies with Genetically Modified Mouse Models.Dopaminergic dysfunction in neurodevelopmental disorders: recent advances and synergistic technologies to aid basic research.Multivariate pattern analysis reveals subtle brain anomalies relevant to the cognitive phenotype in neurofibromatosis type 1.Relationship between cognitive dysfunction, gait, and motor impairment in children and adolescents with neurofibromatosis type 1.Neurofibromatosis 2011: a report of the Children's Tumor Foundation annual meeting.The impact of ADHD on the cognitive and academic functioning of children with NF1.The adverse influence of attention-deficit disorder with or without hyperactivity on cognition in neurofibromatosis type 1.Does attention-deficit-hyperactivity disorder exacerbate executive dysfunction in children with neurofibromatosis type 1?Neurodevelopmental disorders in children with neurofibromatosis type 1.Neurofibromatosis Clinical Trial Consortium.Oligodendrocyte Nf1 Controls Aberrant Notch Activation and Regulates Myelin Structure and Behavior.The genetic and neuroanatomical basis of social dysfunction: lessons from neurofibromatosis type 1.

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Reduced striatal dopamine underlies the attention system dysfunction in neurofibromatosis-1 mutant mice.

http://www.wikidata.org/entity/Q30497049

description

2010 nî lūn-bûn

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2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Reduced striatal dopamine unde ...... urofibromatosis-1 mutant mice.

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Reduced striatal dopamine unde ...... urofibromatosis-1 mutant mice.

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type

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Reduced striatal dopamine unde ...... urofibromatosis-1 mutant mice.

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Reduced striatal dopamine unde ...... urofibromatosis-1 mutant mice.

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Reduced striatal dopamine unde ...... urofibromatosis-1 mutant mice.

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Reduced striatal dopamine unde ...... urofibromatosis-1 mutant mice.

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Human Molecular Genetics

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Reduced striatal dopamine unde ...... eurofibromatosis-1 mutant mice

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P2093

Carla M Yuede

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Crystal R White

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David F Wozniak

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Karen L O'Malley

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Ryan J Emnett

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Sara B Conyers

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P2860

Evidence for neurogenesis in the adult mammalian substantia nigra.

Targeted disruption of the neurofibromatosis type-1 gene leads to developmental abnormalities in heart and various neural crest-derived tissues

Activity, non-selective attention and emotionality in dopamine D2/D3 receptor knock-out mice

The HMG-CoA reductase inhibitor lovastatin reverses the learning and attention deficits in a mouse model of neurofibromatosis type 1

A mouse model for the learning and memory deficits associated with neurofibromatosis type I

A robust and high-throughput Cre reporting and characterization system for the whole mouse brain

Aberrant sprouting and downregulation of tyrosine hydroxylase in lesioned nigrostriatal dopamine neurons induced by long-lasting overexpression of glial cell line derived neurotrophic factor in the striatum by lentiviral gene transfer.

Quantification of MPTP-induced dopaminergic neurodegeneration in the mouse substantia nigra by laser capture microdissection.

Adult-generated hippocampal neurons allow the flexible use of spatially precise learning strategies.

Adult hippocampal neurogenesis is functionally important for stress-induced social avoidance.

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4515-4528

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10.1093/HMG/DDQ382

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22

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19

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David H Gutmann

Jacquelyn Brown

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2010-09-07T00:00:00Z

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46180933

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20826448

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corpus striatum

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2957316

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