Loss of Thr286 phosphorylation disrupts synaptic CaMKIIα targeting, NMDAR activity and behavior in pre-adolescent mice.
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Evidence against dopamine D1/D2 receptor heteromers.Serotonin 5-HT3 receptor-mediated vomiting occurs via the activation of Ca2+/CaMKII-dependent ERK1/2 signaling in the least shrew (Cryptotis parva)Protection of α-CaMKII from Dephosphorylation by GluN2B Subunit of NMDA Receptor Is Abolished by Mutation of Glu96 or His282 of α-CaMKIIMechanisms and Consequences of Dopamine Depletion-Induced Attenuation of the Spinophilin/Neurofilament Medium Interaction.Age-dependent targeting of protein phosphatase 1 to Ca2+/calmodulin-dependent protein kinase II by spinophilin in mouse striatum.Haploinsufficiency of the E3 ubiquitin ligase C-terminus of heat shock cognate 70 interacting protein (CHIP) produces specific behavioral impairments.A significant but rather mild contribution of T286 autophosphorylation to Ca2+/CaM-stimulated CaMKII activity.Changes in the Adult GluN2B Associated Proteome following Adolescent Intermittent Ethanol ExposureDifferential CaMKII regulation by voltage-gated calcium channels in the striatum.Subtle biobehavioral effects produced by paternal cocaine exposureCaMKII regulates diacylglycerol lipase-α and striatal endocannabinoid signalingProteomic Analysis of Postsynaptic Protein Complexes Underlying Neuronal Plasticity.A Novel Human CAMK2A Mutation Disrupts Dendritic Morphology and Synaptic Transmission, and Causes ASD-Related Behaviors.A cluster of protein kinases and phosphatases modulated in fetal Down syndrome (trisomy 21) brain.Chronic intermittent alcohol disrupts the GluN2B-associated proteome and specifically regulates group I mGlu receptor-dependent long-term depression.Quantitative proteomics analysis of CaMKII phosphorylation and the CaMKII interactome in the mouse forebrain.Biochemical principles underlying the stable maintenance of LTP by the CaMKII/NMDAR complex.Deletion of Gαq in the telencephalon alters specific neurobehavioral outcomes.Differential association of postsynaptic signaling protein complexes in striatum and hippocampus.Development of hyperactivity and anxiety responses in dopamine transporter-deficient mice.Role of Striatal Direct Pathway 2-Arachidonoylglycerol Signaling in Sociability and Repetitive Behavior.The CaMKII/NMDA receptor complex controls hippocampal synaptic transmission by kinase-dependent and independent mechanisms.Attention and working memory deficits in a perinatal nicotine exposure mouse model.
P2860
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P2860
Loss of Thr286 phosphorylation disrupts synaptic CaMKIIα targeting, NMDAR activity and behavior in pre-adolescent mice.
description
2011 nî lūn-bûn
@nan
2011 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Loss of Thr286 phosphorylation ...... havior in pre-adolescent mice.
@ast
Loss of Thr286 phosphorylation ...... havior in pre-adolescent mice.
@en
type
label
Loss of Thr286 phosphorylation ...... havior in pre-adolescent mice.
@ast
Loss of Thr286 phosphorylation ...... havior in pre-adolescent mice.
@en
prefLabel
Loss of Thr286 phosphorylation ...... havior in pre-adolescent mice.
@ast
Loss of Thr286 phosphorylation ...... havior in pre-adolescent mice.
@en
P2093
P2860
P1476
Loss of Thr286 phosphorylation ...... havior in pre-adolescent mice.
@en
P2093
Brian C Shonesy
Danny G Winder
Gregg D Stanwood
Nidhi Jalan-Sakrikar
Richard M Gustin
Stacey L Robinson
Tyler J Rentz
P2860
P304
P356
10.1016/J.MCN.2011.05.006
P577
2011-05-24T00:00:00Z