Painful stimuli evoke potentials recorded from the medial temporal lobe in humans.
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Neural mechanisms of pain and alcohol dependenceNasal application of neuropeptide S inhibits arthritis pain-related behaviors through an action in the amygdala.Attention to painful cutaneous laser stimuli evokes directed functional connectivity between activity recorded directly from human pain-related cortical structures.Painful laser stimuli induce directed functional interactions within and between the human amygdala and hippocampus.Dipole source analyses of laser evoked potentials obtained from subdural grid recordings from primary somatic sensory cortex.Attention to painful cutaneous laser stimuli evokes directed functional interactions between human sensory and modulatory pain-related cortical areas.Studies of properties of "Pain Networks" as predictors of targets of stimulation for treatment of pain.Fear conditioning is associated with dynamic directed functional interactions between and within the human amygdala, hippocampus, and frontal lobeN-methyl-D-aspartate receptor agonism and antagonism within the amygdaloid central nucleus suppresses pain affect: differential contribution of the ventrolateral periaqueductal gray.Differential effects of mGluR7 and mGluR8 activation on pain-related synaptic activity in the amygdalaReactive oxygen species mediate visceral pain-related amygdala plasticity and behaviors.Cross-frequency coupling in deep brain structures upon processing the painful sensory inputs.Functional role of induced gamma oscillatory responses in processing noxious and innocuous sensory events in humans.NMDA or non-NMDA receptor antagonism within the amygdaloid central nucleus suppresses the affective dimension of pain in rats: evidence for hemispheric synergy.Pain networks from the inside: Spatiotemporal analysis of brain responses leading from nociception to conscious perception.Oscillatory EEG activity induced by conditioning stimuli during fear conditioning reflects Salience and Valence of these stimuli more than Expectancy.Amygdala pain mechanisms.5-HT2CR blockade in the amygdala conveys analgesic efficacy to SSRIs in a rat model of arthritis pain.Regional gray matter volume and anxiety-related traits interact to predict somatic complaints in a non-clinical sampleTheta signal as the neural signature of social exclusion.Sex dimorphism in a mediatory role of the posterior midcingulate cortex in the association between anxiety and pain sensitivity.Increased Evoked Potentials and Behavioral Indices in Response to Pain Among Individuals with Intellectual Disability.
P2860
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P2860
Painful stimuli evoke potentials recorded from the medial temporal lobe in humans.
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Painful stimuli evoke potentials recorded from the medial temporal lobe in humans.
@ast
Painful stimuli evoke potentials recorded from the medial temporal lobe in humans.
@en
type
label
Painful stimuli evoke potentials recorded from the medial temporal lobe in humans.
@ast
Painful stimuli evoke potentials recorded from the medial temporal lobe in humans.
@en
prefLabel
Painful stimuli evoke potentials recorded from the medial temporal lobe in humans.
@ast
Painful stimuli evoke potentials recorded from the medial temporal lobe in humans.
@en
P2093
P2860
P1433
P1476
Painful stimuli evoke potentials recorded from the medial temporal lobe in humans
@en
P2093
P2860
P304
P356
10.1016/J.NEUROSCIENCE.2009.11.026
P407
P577
2009-11-17T00:00:00Z