Critical role of methylglyoxal and AGE in mycobacteria-induced macrophage apoptosis and activation. - Wikidata - thesis-toulmin - TriplyDB

Critical role of methylglyoxal and AGE in mycobacteria-induced macrophage apoptosis and activation.

Critical role of methylglyoxal and AGE in mycobacteria-induced macrophage apoptosis and activation.

http://www.wikidata.org/entity/Q33267290

about

Cell death and autophagy in tuberculosis An oligopeptide transporter of Mycobacterium tuberculosis regulates cytokine release and apoptosis of infected macrophages Role of the DinB homologs Rv1537 and Rv3056 in Mycobacterium tuberculosis Tmem26 is dynamically expressed during palate and limb development but is not required for embryonic survival.Uptake and accumulation of oxidized low-density lipoprotein during Mycobacterium tuberculosis infection in guinea pigs.Diabetes and immunity to tuberculosis.Non-diabetic hyperglycemia exacerbates disease severity in Mycobacterium tuberculosis infected guinea pigs.Endoplasmic reticulum stress pathway-mediated apoptosis in macrophages contributes to the survival of Mycobacterium tuberculosis.An unconventional hexacoordinated flavohemoglobin from Mycobacterium tuberculosis.Tannerella forsythia infection-induced calvarial bone and soft tissue transcriptional profiles.Host-directed therapy targeting the Mycobacterium tuberculosis granuloma: a review Characterisation of the Mycobacterium tuberculosis alternative sigma factor SigG: its operon and regulon Cell death paradigms in the pathogenesis of Mycobacterium tuberculosis infection.Methylglyoxal and glyoxalase I in atherosclerosis.Cells producing their own nemesis: understanding methylglyoxal metabolism.Defence against methylglyoxal in Group A Streptococcus: a role for Glyoxylase I in bacterial virulence and survival in neutrophils?The Mycobacterium tuberculosis relBE toxin:antitoxin genes are stress-responsive modules that regulate growth through translation inhibition.Integrated stress response of Escherichia coli to methylglyoxal: transcriptional readthrough from the nemRA operon enhances protection through increased expression of glyoxalase I.Intracellular accumulation of advanced glycation end products induces apoptosis via endoplasmic reticulum stress in chondrocytes.Untargeted metabolomics reveals a new mode of action of pretomanid (PA-824).

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Critical role of methylglyoxal and AGE in mycobacteria-induced macrophage apoptosis and activation.

http://www.wikidata.org/entity/Q33267290

description

2006 nî lūn-bûn

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2006 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2006 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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Critical role of methylglyoxal ...... hage apoptosis and activation.

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Critical role of methylglyoxal ...... hage apoptosis and activation.

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Critical role of methylglyoxal ...... hage apoptosis and activation.

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Critical role of methylglyoxal ...... hage apoptosis and activation.

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Critical role of methylglyoxal ...... hage apoptosis and activation.

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Critical role of methylglyoxal ...... hage apoptosis and activation.

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Critical role of methylglyoxal ...... hage apoptosis and activation.

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Critical role of methylglyoxal ...... hage apoptosis and activation.

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Critical role of methylglyoxal ...... hage apoptosis and activation.

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Critical role of methylglyoxal ...... hage apoptosis and activation.

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Ali Nasser Eddine

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George A Kosmiadi

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Helmy Rachman

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Jong Seok Lee

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Lydia Pradl

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Marion Rother

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Ralf-Jürgen Kuban

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Robert Hurwitz

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P2860

Overexpression of glyoxalase-I in bovine endothelial cells inhibits intracellular advanced glycation endproduct formation and prevents hyperglycemia-induced increases in macromolecular endocytosis

A cytoplasmic inhibitor of the JNK signal transduction pathway

Type IV collagen is transcriptionally regulated by Smad1 under advanced glycation end product (AGE) stimulation.

Qualitative determination of specific protein glycation products by matrix-assisted laser desorption/ionization mass spectrometry Peptide mapping.

Methylglyoxal in living organisms: chemistry, biochemistry, toxicology and biological implications.

Methylglyoxal modification of protein. Chemical and immunochemical characterization of methylglyoxal-arginine adducts.

Unique transcriptome signature of Mycobacterium tuberculosis in pulmonary tuberculosis.

Advanced glycation end products and the progressive course of renal disease.

Induction of apoptotic cell death by methylglyoxal and 3-deoxyglucosone in macrophage-derived cell lines.

Synthesis and secretion of tumour necrosis factor-alpha by human monocytic THP-1 cells and chemotaxis induced by human serum albumin derivatives modified with methylglyoxal and glucose-derived advanced glycation endproducts.

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