B-MYB is essential for normal cell cycle progression and chromosomal stability of embryonic stem cells. - Wikidata - thesis-toulmin - TriplyDB

B-MYB is essential for normal cell cycle progression and chromosomal stability of embryonic stem cells.

B-MYB is essential for normal cell cycle progression and chromosomal stability of embryonic stem cells.

http://www.wikidata.org/entity/Q33346222

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Novel Molecular Markers for Breast Cancer Delta40p53 controls the switch from pluripotency to differentiation by regulating IGF signaling in ESCs MicroRNA-302 increases reprogramming efficiency via repression of NR2F2.Enhanced proliferation of monolayer cultures of embryonic stem (ES) cell-derived cardiomyocytes following acute loss of retinoblastoma.Soaking RNAi in Bombyx mori BmN4-SID1 cells arrests cell cycle progression c-Myb binds MLL through menin in human leukemia cells and is an important driver of MLL-associated leukemogenesis.A scalable approach for discovering conserved active subnetworks across species.Activation of innate immunity is required for efficient nuclear reprogramming.Computational analysis of expression of human embryonic stem cell-associated signatures in tumors.Mybl2, downregulated during colon epithelial cell maturation, is suppressed by miR-365.The MuvB complex sequentially recruits B-Myb and FoxM1 to promote mitotic gene expression.G1 arrest and differentiation can occur independently of Rb family function.The B-MYB transcriptional network guides cell cycle progression and fate decisions to sustain self-renewal and the identity of pluripotent stem cells.B-Myb is critical for proper DNA duplication during an unperturbed S phase in mouse embryonic stem cells MYBL2, a link between proliferation and differentiation in maturing colon epithelial cells.LIN9, a subunit of the DREAM complex, regulates mitotic gene expression and proliferation of embryonic stem cells.Cellular senescence and aging: the role of B-MYB Cardiac stem/progenitor cells, secreted proteins, and proteomics Learning biomarkers of pluripotent stem cells in mouse.Embryonic stem cell-derived cardiomyocyte heterogeneity and the isolation of immature and committed cells for cardiac remodeling and regeneration.Addiction of MYCN amplified tumours to B-MYB underscores a reciprocal regulatory loop The CHR promoter element controls cell cycle-dependent gene transcription and binds the DREAM and MMB complexes.Stem cell pluripotency: a cellular trait that depends on transcription factors, chromatin state and a checkpoint deficient cell cycle.Regulation of CLU gene expression by oncogenes and epigenetic factors implications for tumorigenesis.Parallel Regulation of von Hippel-Lindau Disease by pVHL-Mediated Degradation of B-Myb and Hypoxia-Inducible Factor α.lin9 is required for mitosis and cell survival during early zebrafish development.B-myb is an essential regulator of hematopoietic stem cell and myeloid progenitor cell development.Examining the role of cyclin D1 in breast cancer.MYBL2 (B-Myb): a central regulator of cell proliferation, cell survival and differentiation involved in tumorigenesis.Early Evolution of Vertebrate Mybs: An Integrative Perspective Combining Synteny, Phylogenetic, and Gene Expression Analyses.Polo-like kinase 4 transcription is activated via CRE and NRF1 elements, repressed by DREAM through CDE/CHR sites and deregulated by HPV E7 protein.B-Myb promotes S-phase independently of its sequence-specific DNA binding activity and interacts with polymerase delta-interacting protein 1 (Pdip1)Lin9, a subunit of the mammalian DREAM complex, is essential for embryonic development, for survival of adult mice, and for tumor suppression.Microarray analyses of otospheres derived from the cochlea in the inner ear identify putative transcription factors that regulate the characteristics of otospheres.The DREAM complex through its subunit Lin37 cooperates with Rb to initiate quiescence.NonO binds to the CpG island of oct4 promoter and functions as a transcriptional activator of oct4 gene expression.B-Myb switches from Cyclin/Cdk-dependent to Jnk- and p38 kinase-dependent phosphorylation and associates with SC35 bodies after UV stress.Cell therapy from bench to bedside: Hepatocytes from fibroblasts - the truth and myth of transdifferentiation.Prognostic value of proliferation markers expression in breast cancer.Localization of c-MYB in differentiated cells during postnatal molar and alveolar bone development.

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B-MYB is essential for normal cell cycle progression and chromosomal stability of embryonic stem cells.

http://www.wikidata.org/entity/Q33346222

description

2008 nî lūn-bûn

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2008 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2008 թվականի հունիսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

B-MYB is essential for normal ...... ility of embryonic stem cells.

@ast

B-MYB is essential for normal ...... ility of embryonic stem cells.

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type

label

B-MYB is essential for normal ...... ility of embryonic stem cells.

@ast

B-MYB is essential for normal ...... ility of embryonic stem cells.

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prefLabel

B-MYB is essential for normal ...... ility of embryonic stem cells.

@ast

B-MYB is essential for normal ...... ility of embryonic stem cells.

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JOURNAL.PONE.0002478

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B-MYB is essential for normal ...... ility of embryonic stem cells.

@en

P2093

Anna M Wobus

http://www.w3.org/2001/XMLSchema#string

Bing Lim

http://www.w3.org/2001/XMLSchema#string

Colin L Stewart

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Daniel R Riordon

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David G Crider

http://www.w3.org/2001/XMLSchema#string

Gabriela Kania

http://www.w3.org/2001/XMLSchema#string

Gianluca Testa

http://www.w3.org/2001/XMLSchema#string

Jennifer E Van Eyk

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Jinliang Li

http://www.w3.org/2001/XMLSchema#string

Kenneth R Boheler

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P2860

The myb gene family in cell growth, differentiation and apoptosis

Degradation of B-Myb by ubiquitin-mediated proteolysis: involvement of the Cdc34-SCF(p45Skp2) pathway

Functional expression cloning of Nanog, a pluripotency sustaining factor in embryonic stem cells

Inhibition of cyclin A/Cdk2 phosphorylation impairs B-Myb transactivation function without affecting interactions with DNA or the CBP coactivator

Effects of B-Myb on gene transcription: phosphorylation-dependent activity ans acetylation by p300

Arrest of spermatogenesis and defective breast development in mice lacking A-myb

Quantitative expression of Oct-3/4 defines differentiation, dedifferentiation or self-renewal of ES cells

Reciprocal transcriptional regulation of Pou5f1 and Sox2 via the Oct4/Sox2 complex in embryonic stem cells

Rational siRNA design for RNA interference

The transcription factor B-Myb is maintained in an inhibited state in target cells through its interaction with the nuclear corepressors N-CoR and SMRT.

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e2478

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scholarly article

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10.1371/JOURNAL.PONE.0002478

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6

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3

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Kirill V Tarasov

P577

2008-06-25T00:00:00Z

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5279901

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18575582

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2423619

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