Liver cancer-derived hepatitis C virus core proteins shift TGF-beta responses from tumor suppression to epithelial-mesenchymal transition - Wikidata - thesis-toulmin - TriplyDB

Liver cancer-derived hepatitis C virus core proteins shift TGF-beta responses from tumor suppression to epithelial-mesenchymal transition

Liver cancer-derived hepatitis C virus core proteins shift TGF-beta responses from tumor suppression to epithelial-mesenchymal transition

http://www.wikidata.org/entity/Q33405240

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Hepatitis C virus infection and the risk of intrahepatic cholangiocarcinoma and extrahepatic cholangiocarcinoma: evidence from a systematic review and meta-analysis of 16 case-control studies Viruses in cancer cell plasticity: the role of hepatitis C virus in hepatocellular carcinoma Hepatitis C Virus (HCV) Proteins Induce NADPH Oxidase 4 Expression in a Transforming Growth Factor -Dependent Manner: a New Contributor to HCV-Induced Oxidative Stress Hepatitis viruses infection and risk of intrahepatic cholangiocarcinoma: evidence from a meta-analysis.Targeting the transforming growth factor-beta signaling pathway in human cancer.Epithelial-mesenchymal transitions and hepatocarcinogenesis.Hepcidin and the iron enigma in HCV infection.Production and pathogenicity of hepatitis C virus core gene products.Hepatitis C virus regulates transforming growth factor beta1 production through the generation of reactive oxygen species in a nuclear factor kappaB-dependent manner.Enhancement of canonical Wnt/β-catenin signaling activity by HCV core protein promotes cell growth of hepatocellular carcinoma cells Viral factors induce Hedgehog pathway activation in humans with viral hepatitis, cirrhosis, and hepatocellular carcinoma.Focal adhesion kinase (FAK) mediates the induction of pro-oncogenic and fibrogenic phenotypes in hepatitis C virus (HCV)-infected cells.Epithelial-to-mesenchymal transition of murine liver tumor cells promotes invasion Viral hepatocarcinogenesis.Variations in DEPDC5 gene and its association with chronic hepatitis C virus infection in Saudi Arabia.Oncogenic potential of hepatitis C virus proteins Slit-Robo signaling induces malignant transformation through Hakai-mediated E-cadherin degradation during colorectal epithelial cell carcinogenesis TLR9 differentiates rapidly from slowly progressing forms of idiopathic pulmonary fibrosis.Sulfatase 1 and sulfatase 2 in hepatocellular carcinoma: associated signaling pathways, tumor phenotypes, and survival.Gamma-smooth muscle actin expression is associated with epithelial-mesenchymal transition and stem-like properties in hepatocellular carcinoma Hepatitis C virus infection: establishment of chronicity and liver disease progression Microbes-induced EMT at the crossroad of inflammation and cancer.Hepatitis C virus core protein modulates pRb2/p130 expression in human hepatocellular carcinoma cell lines through promoter methylation ESC reverses epithelial mesenchymal transition induced by transforming growth factor-β via inhibition of Smad signal pathway in HepG2 liver cancer cells.dsRNA-dependent protein kinase PKR and its role in stress, signaling and HCV infection.Virus-specific mechanisms of carcinogenesis in hepatitis C virus associated liver cancer SHIP2 regulates epithelial cell polarity through its lipid product, which binds to Dlg1, a pathway subverted by hepatitis C virus core protein Cyclophilin A as a New Therapeutic Target for Hepatitis C Virus-induced Hepatocellular Carcinoma Epithelial-mesenchymal transition in hepatocellular carcinoma.Tumor initiation and progression in hepatocellular carcinoma: risk factors, classification, and therapeutic targets.Intersecting pathways in inflammation and cancer: Hepatocellular carcinoma as a paradigm.Epithelial-mesenchymal transition: molecular pathways of hepatitis viruses-induced hepatocellular carcinoma progression.The rationale for targeting TGF-β in chronic liver diseases.Hepatitis C virus core protein epigenetically silences SFRP1 and enhances HCC aggressiveness by inducing epithelial-mesenchymal transition.HAb18G/CD147 promotes epithelial-mesenchymal transition through TGF-β signaling and is transcriptionally regulated by Slug.Hepatitis C virus core protein activates Wnt/β-catenin signaling through multiple regulation of upstream molecules in the SMMC-7721 cell line.Comprehensive analysis of the independent effect of twist and snail in promoting metastasis of hepatocellular carcinoma.Hepatic stellate cells as key target in liver fibrosis.Hepatocellular carcinoma: from hepatocyte to liver cancer stem cell Hepatitis C virus core protein interacts with Snail and histone deacetylases to promote the metastasis of hepatocellular carcinoma.

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Liver cancer-derived hepatitis C virus core proteins shift TGF-beta responses from tumor suppression to epithelial-mesenchymal transition

http://www.wikidata.org/entity/Q33405240

description

2009 nî lūn-bûn

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2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի փետրվարին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Liver cancer-derived hepatitis ...... thelial-mesenchymal transition

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Liver cancer-derived hepatitis ...... thelial-mesenchymal transition

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type

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Liver cancer-derived hepatitis ...... thelial-mesenchymal transition

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Liver cancer-derived hepatitis ...... thelial-mesenchymal transition

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Liver cancer-derived hepatitis ...... thelial-mesenchymal transition

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Liver cancer-derived hepatitis ...... thelial-mesenchymal transition

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Liver cancer-derived hepatitis ...... thelial-mesenchymal transition

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Anna Linda Zignego

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Azeddine Atfi

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Christian Bréchot

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Didier Samuel

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Nassima Benzoubir

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Pierre Charneau

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Serena Battaglia

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Soizic Nobilet

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P2860

Twist and epithelial-mesenchymal transition are induced by the EBV oncoprotein latent membrane protein 1 and are associated with metastatic nasopharyngeal carcinoma

Hepatitis C viral proteins interact with Smad3 and differentially regulate TGF-beta/Smad3-mediated transcriptional activation

Hepatitis C virus core variants isolated from liver tumor but not from adjacent non-tumor tissue interact with Smad3 and inhibit the TGF-beta pathway

The epithelial-mesenchymal transition generates cells with properties of stem cells

Hepatitis C virus core protein binds to the cytoplasmic domain of tumor necrosis factor (TNF) receptor 1 and enhances TNF-induced apoptosis.

Inhibition of transforming growth factor (TGF)-beta1-induced extracellular matrix with a novel inhibitor of the TGF-beta type I receptor kinase activity: SB-431542

Smad-dependent and Smad-independent pathways in TGF-beta family signalling

Hematopoiesis controlled by distinct TIF1gamma and Smad4 branches of the TGFbeta pathway

Smads: transcriptional activators of TGF-beta responses

Jun N-terminal kinase 1 regulates epithelial-to-mesenchymal transition induced by TGF-beta1

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10.1371/JOURNAL.PONE.0004355

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Marie-Françoise Bourgeade

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2009-02-03T00:00:00Z

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23974990

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2629560

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