High mutability of the tumor suppressor genes RASSF1 and RBSP3 (CTDSPL) in cancer. - Wikidata - thesis-toulmin - TriplyDB

High mutability of the tumor suppressor genes RASSF1 and RBSP3 (CTDSPL) in cancer.

High mutability of the tumor suppressor genes RASSF1 and RBSP3 (CTDSPL) in cancer.

http://www.wikidata.org/entity/Q33455585

about

A RASSF1A polymorphism restricts p53/p73 activation and associates with poor survival and accelerated age of onset of soft tissue sarcoma.Understanding thrombocytopenia: physiological role of microRNA in survival of neonatal megakaryocytes.Simultaneous down-regulation of tumor suppressor genes RBSP3/CTDSPL, NPRL2/G21 and RASSF1A in primary non-small cell lung cancer.MiR-99a may serve as a potential oncogene in pediatric myeloid leukemia Somatic mutations, allele loss, and DNA methylation of the Cub and Sushi Multiple Domains 1 (CSMD1) gene reveals association with early age of diagnosis in colorectal cancer patients Damage-induced localized hypermutability.MiR-100 regulates cell differentiation and survival by targeting RBSP3, a phosphatase-like tumor suppressor in acute myeloid leukemia.RASSF1 Polymorphisms in Cancer.NotI microarrays: novel epigenetic markers for early detection and prognosis of high grade serous ovarian cancer.Clusters of Multiple Mutations: Incidence and Molecular Mechanisms Ras signaling through RASSF proteins.Integration of ALV into CTDSPL and CTDSPL2 genes in B-cell lymphomas promotes cell immortalization, migration and survival.Lentivirus-mediated silencing of USO1 inhibits cell proliferation and migration of human colon cancer cells.*K-means and cluster models for cancer signatures.Alterations of RASSF1A in premalignant cervical lesions: clinical and prognostic significance.The miR-181 family promotes cell cycle by targeting CTDSPL, a phosphatase-like tumor suppressor in uveal melanoma.Excess of a Rassf1-targeting microRNA, miR-193a-3p, perturbs cell division fidelity.

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P2860

High mutability of the tumor suppressor genes RASSF1 and RBSP3 (CTDSPL) in cancer.

http://www.wikidata.org/entity/Q33455585

description

2009 nî lūn-bûn

@nan

2009 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

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2009年論文

@zh-hk

2009年論文

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2009年論文

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2009年论文

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name

High mutability of the tumor suppressor genes RASSF1 and RBSP3 (CTDSPL) in cancer.

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High mutability of the tumor suppressor genes RASSF1 and RBSP3 (CTDSPL) in cancer.

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type

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High mutability of the tumor suppressor genes RASSF1 and RBSP3 (CTDSPL) in cancer.

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High mutability of the tumor suppressor genes RASSF1 and RBSP3 (CTDSPL) in cancer.

@en

prefLabel

High mutability of the tumor suppressor genes RASSF1 and RBSP3 (CTDSPL) in cancer.

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High mutability of the tumor suppressor genes RASSF1 and RBSP3 (CTDSPL) in cancer.

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High mutability of the tumor suppressor genes RASSF1 and RBSP3 (CTDSPL) in cancer.

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Alexei I Protopopov

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Alexey Kutsenko

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Eleonora Braga

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Eugene R Zabarovsky

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Fuli Wang

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George Klein

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Igor Kuzmin

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Ingemar Ernberg

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Jingfeng Li

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Julia Kobliakova

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P2860

Isolation, tissue distribution, and chromosomal localization of the human activation-induced cytidine deaminase (AID) gene

The RASSF1A tumor suppressor blocks cell cycle progression and inhibits cyclin D1 accumulation.

RBSP3 (HYA22) is a tumor suppressor gene implicated in major epithelial malignancies

The 630-kb lung cancer homozygous deletion region on human chromosome 3p21.3: identification and evaluation of the resident candidate tumor suppressor genes. The International Lung Cancer Chromosome 3p21.3 Tumor Suppressor Gene Consortium

DNA polymerase eta is an A-T mutator in somatic hypermutation of immunoglobulin variable genes

An integrated physical and gene map of the 3.5-Mb chromosome 3p21.3 (AP20) region implicated in major human epithelial malignancies

Hypermutation of multiple proto-oncogenes in B-cell diffuse large-cell lymphomas

Epigenetic inactivation of a RAS association domain family protein from the lung tumour suppressor locus 3p21.3.

Hypermutability of damaged single-strand DNA formed at double-strand breaks and uncapped telomeres in yeast Saccharomyces cerevisiae.

Mammalian DNA mismatch repair.

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Elvira Grigorieva

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