Hypoxia and the hypoxic response pathway protect against pore-forming toxins in C. elegans.
about
Multiple reciprocal adaptations and rapid genetic change upon experimental coevolution of an animal host and its microbial parasiteSignaling beyond Punching Holes: Modulation of Cellular Responses by Vibrio cholerae CytolysinModulation of translation and induction of autophagy by bacterial exoproductsDisruption of the C. elegans Intestinal Brush Border by the Fungal Lectin CCL2 Phenocopies Dietary Lectin Toxicity in MammalsGlobal functional analyses of cellular responses to pore-forming toxinsNeuronal Goα and CAPS regulate behavioral and immune responses to bacterial pore-forming toxinsSurveillance-Activated Defenses Block the ROS–Induced Mitochondrial Unfolded Protein ResponseEGL-9 controls C. elegans host defense specificity through prolyl hydroxylation-dependent and -independent HIF-1 pathwaysMonalysin, a novel ß-pore-forming toxin from the Drosophila pathogen Pseudomonas entomophila, contributes to host intestinal damage and lethalityAn ABC transporter mutation is correlated with insect resistance to Bacillus thuringiensis Cry1Ac toxinMechanistic and single-dose in vivo therapeutic studies of Cry5B anthelmintic action against hookwormsGene-trap mutagenesis identifies mammalian genes contributing to intoxication by Clostridium perfringens ε-toxinA subunit of eukaryotic translation initiation factor 2α-phosphatase (CreP/PPP1R15B) regulates membrane traffic.WWP-1 is a novel modulator of the DAF-2 insulin-like signaling network involved in pore-forming toxin cellular defenses in Caenorhabditis elegans.The Natural Biotic Environment of Caenorhabditis elegans.C. elegans SWAN-1 Binds to EGL-9 and regulates HIF-1-mediated resistance to the bacterial pathogen Pseudomonas aeruginosa PAO1.Discovery of a highly synergistic anthelmintic combination that shows mutual hypersusceptibilityProtist-type lysozymes of the nematode Caenorhabditis elegans contribute to resistance against pathogenic Bacillus thuringiensisTranscriptome profiling of the intoxication response of Tenebrio molitor larvae to Bacillus thuringiensis Cry3Aa protoxinRole of pore-forming toxins in bacterial infectious diseases.Feedback regulation via AMPK and HIF-1 mediates ROS-dependent longevity in Caenorhabditis elegans.Oligomerization of Clostridium perfringens epsilon toxin is dependent upon caveolins 1 and 2.Proteome response of Tribolium castaneum larvae to Bacillus thuringiensis toxin producing strains.RAB-5- and RAB-11-dependent vesicle-trafficking pathways are required for plasma membrane repair after attack by bacterial pore-forming toxin.Regulation of life span by mitochondrial respiration: the HIF-1 and ROS connection.Contrasting invertebrate immune defense behaviors caused by a single gene, the Caenorhabditis elegans neuropeptide receptor gene npr-1Genomic analysis of immune response against Vibrio cholerae hemolysin in Caenorhabditis elegans.Inhibition of elongin C promotes longevity and protein homeostasis via HIF-1 in C. elegansStatin-conferred enhanced cellular resistance against bacterial pore-forming toxins in airway epithelial cellsThe p38 MAP kinase pathway modulates the hypoxia response and glutamate receptor trafficking in aging neuronsNew role for DCR-1/dicer in Caenorhabditis elegans innate immunity against the highly virulent bacterium Bacillus thuringiensis DB27.HLH-30/TFEB-mediated autophagy functions in a cell-autonomous manner for epithelium intrinsic cellular defense against bacterial pore-forming toxin in C. elegans.HIF-1 modulates longevity and healthspan in a temperature-dependent manner.Role of MAPK p38 in the cellular responses to pore-forming toxins.The hypoxia-inducible factor HIF-1 functions as both a positive and negative modulator of aging.Bacillus thuringiensis: A story of a successful bioinsecticide.Microbial pathogenesis and host defense in the nematode C. elegans.The impact of hypoxia on bacterial infection.Current advances in the novel functions of hypoxia-inducible factor and prolyl hydroxylase in invertebrates.HIF-1-dependent regulation of lifespan in Caenorhabditis elegans by the acyl-CoA-binding protein MAA-1.
P2860
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P2860
Hypoxia and the hypoxic response pathway protect against pore-forming toxins in C. elegans.
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Hypoxia and the hypoxic respon ...... -forming toxins in C. elegans.
@ast
Hypoxia and the hypoxic respon ...... -forming toxins in C. elegans.
@en
Hypoxia and the hypoxic respon ...... -forming toxins in C. elegans.
@nl
type
label
Hypoxia and the hypoxic respon ...... -forming toxins in C. elegans.
@ast
Hypoxia and the hypoxic respon ...... -forming toxins in C. elegans.
@en
Hypoxia and the hypoxic respon ...... -forming toxins in C. elegans.
@nl
prefLabel
Hypoxia and the hypoxic respon ...... -forming toxins in C. elegans.
@ast
Hypoxia and the hypoxic respon ...... -forming toxins in C. elegans.
@en
Hypoxia and the hypoxic respon ...... -forming toxins in C. elegans.
@nl
P2860
P50
P1433
P1476
Hypoxia and the hypoxic respon ...... -forming toxins in C. elegans.
@en
P2093
Hediye N Cinar
Raffi V Aroian
P2860
P304
P356
10.1371/JOURNAL.PPAT.1000689
P577
2009-12-11T00:00:00Z