Grp78 promotes the invasion of hepatocellular carcinoma. - Wikidata - thesis-toulmin - TriplyDB

Grp78 promotes the invasion of hepatocellular carcinoma.

Grp78 promotes the invasion of hepatocellular carcinoma.

http://www.wikidata.org/entity/Q33524918

about

Melanoma and the Unfolded Protein Response Glucose-regulated protein 78 demonstrates antiviral effects but is more suitable for hepatocellular carcinoma prevention in hepatitis B.Lack of association between the GRP78 polymorphisms in the promoter and 3' UTR and susceptibility to chronic HBV infection in a Chinese Han population.Antitumor agent 25-epi Ritterostatin GN1N induces endoplasmic reticulum stress and autophagy mediated cell death in melanoma cells Granulin-epithelin precursor interacts with 78-kDa glucose-regulated protein in hepatocellular carcinoma LyGDI, a novel SHIP-interacting protein, is a negative regulator of FcγR-mediated phagocytosis.Endoplasmic reticulum stress and cancer.Glucose regulated protein 78 promotes cell invasion via regulation of uPA production and secretion in colon cancer cells.Clusterin protects hepatocellular carcinoma cells from endoplasmic reticulum stress induced apoptosis through GRP78.Proteomic differences between hepatocellular carcinoma and nontumorous liver tissue investigated by a combined gel-based and label-free quantitative proteomics study.The 3' UTR variants in the GRP78 are not associated with overall survival in resectable hepatocellular carcinoma.De-acetylation and degradation of HSPA5 is critical for E1A metastasis suppression in breast cancer cells.Endoplasmic reticulum stress in endometrial cancer.Autoantibodies against cell surface GRP78 promote tumor growth in a murine model of melanoma Role of prostate apoptosis response 4 in translocation of GRP78 from the endoplasmic reticulum to the cell surface of trophoblastic cells.JNK contributes to the tumorigenic potential of human cholangiocarcinoma cells through the mTOR pathway regulated GRP78 induction.Upregulation of heat shock proteins (HSPA12A, HSP90B1, HSPA4, HSPA5 and HSPA6) in tumour tissues is associated with poor outcomes from HBV-related early-stage hepatocellular carcinoma.Unfolding the Role of Stress Response Signaling in Endocrine Resistant Breast Cancers.Melatonin Activates Endoplasmic Reticulum Stress and Apoptosis in Rats with Diethylnitrosamine-Induced Hepatocarcinogenesis Elevated GRP78 expression is associated with poor prognosis in patients with pancreatic cancer 3'3-Diindolylmethane inhibits migration, invasion and metastasis of hepatocellular carcinoma by suppressing FAK signaling.GRP78 confers the resistance to 5-FU by activating the c-Src/LSF/TS axis in hepatocellular carcinoma Autoantibodies against glucose-regulated protein 78 as serological diagnostic biomarkers in hepatocellular carcinoma The cell surface GRP78 facilitates the invasion of hepatocellular carcinoma cells.High expression of glucose-regulated protein 78 (GRP78) is associated with metastasis and poor prognosis in patients with esophageal squamous cell carcinoma.GRP78 as a regulator of liver steatosis and cancer progression mediated by loss of the tumor suppressor PTEN.The biological and therapeutic relevance of mRNA translation in cancer.Targeting GRP78 and antiestrogen resistance in breast cancer.Expression of ER stress and autophagy-related molecules in human non-small cell lung cancer and premalignant lesions.Oncolytic vaccinia virus inhibits human hepatocellular carcinoma MHCC97-H cell proliferation via endoplasmic reticulum stress, autophagy and Wnt pathways.Grp78 Is Critical for Amelogenin-Induced Cell Migration in a Multipotent Clonal Human Periodontal Ligament Cell Line.High Expression of GRP78 Promotes Invasion and Metastases in Patients with Esophageal Squamous Cell Carcinoma.Nanoparticles inhibit cancer cell invasion and enhance antitumor efficiency by targeted drug delivery via cell surface-related GRP78.GRP78 promotes the invasion of pancreatic cancer cells by FAK and JNK.Knockdown of glucose-regulated protein 78 decreases the invasion, metalloproteinase expression and ECM degradation in hepatocellular carcinoma cells.Silencing of glucose-regulated protein 78 (GRP78) enhances cell migration through the upregulation of vimentin in hepatocellular carcinoma cells.Glucose-regulated protein 58 modulates cell invasiveness and serves as a prognostic marker for cervical cancer.Secreted GRP78 activates EGFR-SRC-STAT3 signaling and confers the resistance to sorafeinib in HCC cells.Deficiency of protein-L-isoaspartate (D-aspartate) O-methyl-transferase expression under endoplasmic reticulum stress promotes epithelial mesenchymal transition in lung adenocarcinoma.Mouse models for investigating the underlying mechanisms of nonalcoholic steatohepatitis-derived hepatocellular carcinoma.

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P2860

Grp78 promotes the invasion of hepatocellular carcinoma.

http://www.wikidata.org/entity/Q33524918

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

@zh-tw

2010年论文

@wuu

name

Grp78 promotes the invasion of hepatocellular carcinoma.

@ast

Grp78 promotes the invasion of hepatocellular carcinoma.

@en

Grp78 promotes the invasion of hepatocellular carcinoma.

@nl

type

label

Grp78 promotes the invasion of hepatocellular carcinoma.

@ast

Grp78 promotes the invasion of hepatocellular carcinoma.

@en

Grp78 promotes the invasion of hepatocellular carcinoma.

@nl

prefLabel

Grp78 promotes the invasion of hepatocellular carcinoma.

@ast

Grp78 promotes the invasion of hepatocellular carcinoma.

@en

Grp78 promotes the invasion of hepatocellular carcinoma.

@nl

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1471-2407-10-20

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Grp78 promotes the invasion of hepatocellular carcinoma.

@en

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Cuifen Bao

http://www.w3.org/2001/XMLSchema#string

Hongdan Li

http://www.w3.org/2001/XMLSchema#string

Huijuan Song

http://www.w3.org/2001/XMLSchema#string

Jia Wei

http://www.w3.org/2001/XMLSchema#string

Liufang Cheng

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Rongjian Su

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Zhen Li

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P2860

Signaling from Rho to the actin cytoskeleton through protein kinases ROCK and LIM-kinase

Integrin-regulated FAK-Src signaling in normal and cancer cells

GRP78 and Raf-1 cooperatively confer resistance to endoplasmic reticulum stress-induced apoptosis

Homocysteine-induced endoplasmic reticulum stress causes dysregulation of the cholesterol and triglyceride biosynthetic pathways

Rho and Rac take center stage

Focal adhesion kinase: in command and control of cell motility

FAK-Src signalling through paxillin, ERK and MLCK regulates adhesion disassembly

A randomized phase III study of doxorubicin versus cisplatin/interferon alpha-2b/doxorubicin/fluorouracil (PIAF) combination chemotherapy for unresectable hepatocellular carcinoma.

The glucose-regulated proteins: stress induction and clinical applications.

Suppression of RhoA activity by focal adhesion kinase-induced activation of p190RhoGAP: role in regulation of endothelial permeability.

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20082722

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2821360

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