HIV and HCV activate the inflammasome in monocytes and macrophages via endosomal Toll-like receptors without induction of type 1 interferon. - Wikidata - thesis-toulmin - TriplyDB

HIV and HCV activate the inflammasome in monocytes and macrophages via endosomal Toll-like receptors without induction of type 1 interferon.

HIV and HCV activate the inflammasome in monocytes and macrophages via endosomal Toll-like receptors without induction of type 1 interferon.

http://www.wikidata.org/entity/Q33553764

about

Innate Sensing of the Gut Microbiota: Modulation of Inflammatory and Autoimmune Diseases Interleukin-1 Family Cytokines in Liver Diseases Virological Mechanisms in the Coinfection between HIV and HCV Regulation of the Host Antiviral State by Intercellular Communications Interplay between Inflammation and Cellular Stress Triggered by Flaviviridae Viruses MAIT cells are activated during human viral infections Inflammasomes and Their Role in Innate Immunity of Sexually Transmitted Infections Insufficient natural killer cell responses against retroviruses: how to improve NK cell killing of retrovirus-infected cells Integrating Inflammasome Signaling in Sexually Transmitted Infections Systematic review and meta-analysis of hepatitis C virus infection and HIV viral load: new insights into epidemiologic synergy Inflammasome control of viral infection Natural Killer Cells in Viral Hepatitis.Toll-like receptor 3 signaling inhibits simian immunodeficiency virus replication in macrophages from rhesus macaques Nucleoside reverse transcriptase inhibitors possess intrinsic anti-inflammatory activity.IFNL4 and IFNL3 associated polymorphisms strongly influence the spontaneous IFN-alpha receptor-1 expression in HCV-infected patients.SHP-1-dependent macrophage differentiation exacerbates virus-induced myositis Hepatitis C Virus. Strategies to Evade Antiviral Responses Human Immunodeficiency Virus Type 1 Nef Inhibits Autophagy through Transcription Factor EB Sequestration HCV dsRNA-Activated Macrophages Inhibit HCV Replication in Hepatocytes.A Cross-sectional Study of the Association Between Chronic Hepatitis C Virus Infection and Subclinical Coronary Atherosclerosis Among Participants in the Multicenter AIDS Cohort Study The Hepatitis C Virus-induced NLRP3 Inflammasome Activates the Sterol Regulatory Element-binding Protein (SREBP) and Regulates Lipid Metabolism.NLRX1 Sequesters STING to Negatively Regulate the Interferon Response, Thereby Facilitating the Replication of HIV-1 and DNA Viruses.Hepatitis C Virus-Induced Monocyte Differentiation Into Polarized M2 Macrophages Promotes Stellate Cell Activation via TGF-β Continued Elevation of Interleukin-18 and Interferon-γ After Initiation of Antiretroviral Therapy and Clinical Failure in a Diverse Multicountry Human Immunodeficiency Virus Cohort.TLR7/8 agonist induces a post-entry SAMHD1-independent block to HIV-1 infection of monocytes.HIV cell-to-cell transmission: effects on pathogenesis and antiretroviral therapy Hepatitis regulation by the inflammasome signaling pathway.Innate immunity against HIV-1 infection.Inflammasome activation and function in liver disease.Interplay between HIV-1 and Toll-like receptors in human myeloid cells: friend or foe in HIV-1 pathogenesis?Sensing of HIV-1 Infection in Tzm-bl Cells with Reconstituted Expression of STING.HIV-1-Mediated BAFF Secretion in Macrophages Does Not Require Endosomal TLRs, Type-I IFN, and Nef, but Depends on the Cellular Phenotype Status.Potent NLRP3 Inflammasome Activation by the HIV Reverse Transcriptase Inhibitor Abacavir.Proteomics of Human Dendritic Cell Subsets Reveals Subset-Specific Surface Markers and Differential Inflammasome Function.Dose of Retroviral Infection Determines Induction of Antiviral NK Cell Responses.Intra-hepatic Depletion of Mucosal Associated Invariant T cells in Hepatitis C Virus-induced Liver Inflammation.Group A streptococcal M protein activates the NLRP3 inflammasome.Dendritic Cell Response to HIV-1 Is Controlled by Differentiation Programs in the Cells and Strain-Specific Properties of the Virus.Hepatitis C virus-induced CCL5 secretion from macrophages activates hepatic stellate cells.Genetic basis for variation in plasma IL-18 levels in persons with chronic hepatitis C virus and human immunodeficiency virus-1 infections.

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HIV and HCV activate the inflammasome in monocytes and macrophages via endosomal Toll-like receptors without induction of type 1 interferon.

http://www.wikidata.org/entity/Q33553764

description

2014 nî lūn-bûn

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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

HIV and HCV activate the infla ...... nduction of type 1 interferon.

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HIV and HCV activate the infla ...... nduction of type 1 interferon.

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HIV and HCV activate the infla ...... nduction of type 1 interferon.

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HIV and HCV activate the infla ...... nduction of type 1 interferon.

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HIV and HCV activate the infla ...... nduction of type 1 interferon.

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HIV and HCV activate the infla ...... nduction of type 1 interferon.

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HIV and HCV activate the infla ...... nduction of type 1 interferon.

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HIV and HCV activate the infla ...... nduction of type 1 interferon.

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HIV and HCV activate the infla ...... nduction of type 1 interferon.

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HIV and HCV activate the infla ...... nduction of type 1 interferon.

@en

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Andrea L Cox

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Drew Pardoll

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Joel N Blankson

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Matthew E Winter

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Michael A Chattergoon

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Rachel Latanich

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Robert W Buckheit

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P2860

Cell death by pyroptosis drives CD4 T-cell depletion in HIV-1 infection

Species-specific recognition of single-stranded RNA via toll-like receptor 7 and 8

IFI16 DNA sensor is required for death of lymphoid CD4 T cells abortively infected with HIV

Innate immunity induced by composition-dependent RIG-I recognition of hepatitis C virus RNA

TLR8: the forgotten relative revindicated

Robust hepatitis C virus infection in vitro.

Production of infectious genotype 1a hepatitis C virus (Hutchinson strain) in cultured human hepatoma cells

Elevation of interleukin-18 in chronic hepatitis C: implications for hepatitis C virus pathogenesis

HIV-1 evades innate immune recognition through specific cofactor recruitment

In vitro isolation and identification of human immunodeficiency virus (HIV) variants with reduced sensitivity to C-2 symmetrical inhibitors of HIV type 1 protease

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e1004082

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10.1371/JOURNAL.PPAT.1004082

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5

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10

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24788318

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Toll-like receptor

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4006909

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