SaeR binds a consensus sequence within virulence gene promoters to advance USA300 pathogenesis.
about
Dual RNA regulatory control of a Staphylococcus aureus virulence factorThe SaeRS Two-Component System of Staphylococcus aureusStaphylococcus aureus Nuc2 is a functional, surface-attached extracellular nucleaseSaeRS-dependent inhibition of biofilm formation in Staphylococcus aureus NewmanThe Staphylococcus aureus Global Regulator MgrA Modulates Clumping and Virulence by Controlling Surface Protein ExpressionThe Staphylococcal Biofilm: Adhesins, Regulation, and Host ResponseDifferential regulation of staphylococcal virulence by the sensor kinase SaeS in response to neutrophil-derived stimuli.Evolutionary blueprint for host- and niche-adaptation in Staphylococcus aureus clonal complex CC30.An in vivo reporter assay for sRNA-directed gene control in Gram-positive bacteria: identifying a novel sRNA target in Staphylococcus aureus.The SaeRS Two-Component System Controls Survival of Staphylococcus aureus in Human Blood through Regulation of Coagulase.The bicomponent pore-forming leucocidins of Staphylococcus aureusImportance of the global regulators Agr and SaeRS in the pathogenesis of CA-MRSA USA300 infection.Ketamine inhibits tumor necrosis factor secretion by RAW264.7 murine macrophages stimulated with antibiotic-exposed strains of community-associated, methicillin-resistant Staphylococcus aureusHaemin represses the haemolytic activity of Staphylococcus aureus in an Sae-dependent manner.Epic Immune Battles of History: Neutrophils vs. Staphylococcus aureus.Synthesis and Evaluation of In Vitro Antibacterial and Antitumor Activities of Novel N,N-Disubstituted Schiff BasesVirulence strategies of the dominant USA300 lineage of community-associated methicillin-resistant Staphylococcus aureus (CA-MRSA)The SaeR/S gene regulatory system induces a pro-inflammatory cytokine response during Staphylococcus aureus infection.Nuclease modulates biofilm formation in community-associated methicillin-resistant Staphylococcus aureusAlpha-toxin induces programmed cell death of human T cells, B cells, and monocytes during USA300 infection.Temporal and stochastic control of Staphylococcus aureus biofilm development.A genetic resource for rapid and comprehensive phenotype screening of nonessential Staphylococcus aureus genesLocal inflammation exacerbates the severity of Staphylococcus aureus skin infection.Methionine sulfoxide reductases protect against oxidative stress in Staphylococcus aureus encountering exogenous oxidants and human neutrophils.Phagocytosis escape by a Staphylococcus aureus protein that connects complement and coagulation proteins at the bacterial surfaceIdentification of the P3 promoter and distinct roles of the two promoters of the SaeRS two-component system in Staphylococcus aureus.Staphylococcus aureus regulates the expression and production of the staphylococcal superantigen-like secreted proteins in a Rot-dependent manner.The role of innate immunity in promoting SaeR/S-mediated virulence in Staphylococcus aureus.Colonization of epidermal tissue by Staphylococcus aureus produces localized hypoxia and stimulates secretion of antioxidant and caspase-14 proteins.The Sbi Protein Contributes to Staphylococcus aureus Inflammatory Response during Systemic Infection.Proteomic Identification of saeRS-Dependent Targets Critical for Protective Humoral Immunity against Staphylococcus aureus Skin InfectionStaphylococcus aureus Inhibits Neutrophil-derived IL-8 to Promote Cell Death.Organizational requirements of the SaeR binding sites for a functional P1 promoter of the sae operon in Staphylococcus aureusInteraction of Staphylococci with Human B cells.Rot and SaeRS cooperate to activate expression of the staphylococcal superantigen-like exoproteins.CodY deletion enhances in vivo virulence of community-associated methicillin-resistant Staphylococcus aureus clone USA300.TSS-EMOTE, a refined protocol for a more complete and less biased global mapping of transcription start sites in bacterial pathogens.The WalKR system controls major staphylococcal virulence genes and is involved in triggering the host inflammatory responseThe auxiliary protein complex SaePQ activates the phosphatase activity of sensor kinase SaeS in the SaeRS two-component system of Staphylococcus aureus.18β-Glycyrrhetinic acid inhibits methicillin-resistant Staphylococcus aureus survival and attenuates virulence gene expression.
P2860
Q24567581-F97942B1-6EDF-4A79-BAED-F5E5E068DC1BQ28073824-6E1A3F30-4600-43B4-9C1F-22C1ED9200BDQ28538089-7B3A64F0-6AE5-4976-9338-7247789907FDQ28545930-1F1C9F6A-0ACC-4DDC-B144-F948DB02B29BQ28551807-D5704ED1-CF40-41CC-9A97-8078E4152F62Q28829672-E2B44C77-2B39-44E0-B04C-74BF2FF69ACCQ30361924-AF9EA7E3-04FD-443E-81FD-4DC0170E813EQ30420650-CD7C619D-6E03-4F49-AD1A-976979669BE8Q33635956-B9D6C5D8-8F20-430A-BBF4-417D956D7A38Q33734889-2DB0EB7B-CC77-447F-885E-3D63D819FE5BQ33743499-DA6A1154-AEDC-4798-B403-0F15575C0D7DQ33769704-79889A94-0732-4FD5-A06E-98EB5ECA129FQ33803430-E01111A1-3A64-470D-AD42-3EFC7227DEB3Q33852326-3BC7A852-ADC9-4C88-BAD9-4EAF169EF3B7Q33854487-E6AB41D1-5BC4-435B-9C35-9207054B3145Q33872727-CBFBAB4A-9F50-46EC-A958-5E6B709F3D8DQ33874939-59B8368F-EC3C-4AD3-8466-C8CCADDE658CQ33908572-9C3749AE-4AB5-420B-8CC8-E8FED468305FQ34077679-D0DD71EE-9121-4FF2-98DD-A7CA76306EF2Q34264762-3ABB779D-B17F-4AA5-B464-1130AC2E3DCEQ34384809-B7DF20C9-0C3B-405B-B2FE-85F937B63E71Q34583671-A1088A8F-DDDD-4044-8AE0-A20DED2D6C40Q34828601-AFC3A16D-B498-491F-815B-7C94A058E683Q35046066-8322AB2A-55BF-43F9-A598-DE934ED82B6CQ35069186-DD45A4AF-2389-4DAF-8146-9B548C17478CQ35192376-3266C718-D6DC-483D-A110-B88470720056Q35552227-889EB956-A304-4411-BCE3-862A55737C44Q35619910-EDE0D40D-B690-452A-94B5-330035CD8C86Q35634849-4BD29EB1-9757-44F4-B267-C5821B10852CQ35678811-06BBF21D-C0E6-4E74-BB89-263337FB12B8Q35947359-4611FF61-57B4-4B06-B48D-755CF1107D3BQ35993459-90B04115-2F3C-477D-A486-40C884738B8EQ36018289-2AD0E9BA-7F6C-4E0D-BB69-BD8A30DD7785Q36155037-1D38AC53-A049-4EE2-83AE-D0E045AA0F50Q36155287-1D974294-3E28-4BB2-BD34-AEA2E827FAB7Q36155969-6CE2D626-B3C8-4065-8A0C-5B2F4A7B3B4DQ36180648-BE2FC271-CFE7-4B80-BDB4-B6D1A70ED285Q36277416-001335DD-28B7-402D-B684-CAB3CD6049EDQ36309811-011272B9-837F-400D-8ABC-891560EFFF16Q36505342-D584122F-9F59-4585-A8E0-186A19E14BDA
P2860
SaeR binds a consensus sequence within virulence gene promoters to advance USA300 pathogenesis.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
SaeR binds a consensus sequenc ...... o advance USA300 pathogenesis.
@ast
SaeR binds a consensus sequenc ...... o advance USA300 pathogenesis.
@en
SaeR binds a consensus sequenc ...... o advance USA300 pathogenesis.
@nl
type
label
SaeR binds a consensus sequenc ...... o advance USA300 pathogenesis.
@ast
SaeR binds a consensus sequenc ...... o advance USA300 pathogenesis.
@en
SaeR binds a consensus sequenc ...... o advance USA300 pathogenesis.
@nl
prefLabel
SaeR binds a consensus sequenc ...... o advance USA300 pathogenesis.
@ast
SaeR binds a consensus sequenc ...... o advance USA300 pathogenesis.
@en
SaeR binds a consensus sequenc ...... o advance USA300 pathogenesis.
@nl
P2093
P2860
P356
P1476
SaeR binds a consensus sequenc ...... o advance USA300 pathogenesis.
@en
P2093
Cuong Vuong
Jovanka M Voyich
Kyler B Pallister
Peter Ruzevich
Shannon Griffith
Tyler K Nygaard
P2860
P304
P356
10.1086/649570
P407
P577
2010-01-01T00:00:00Z