Oxidative stress induces mitochondrial dysfunction in a subset of autistic lymphoblastoid cell lines.
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Identification and Treatment of Pathophysiological Comorbidities of Autism Spectrum Disorder to Achieve Optimal Outcomes.Neuropathological Mechanisms of Seizures in Autism Spectrum DisorderTargeting Mitochondrial Dysfunction with L-Alpha GlycerylphosphorylcholineIncreased Paternal Age at Conception Is Associated with Transcriptomic Changes Involved in Mitochondrial Function in Elderly IndividualsModulation of mitochondrial function by the microbiome metabolite propionic acid in autism and control cell lines.Gastrointestinal dysfunction in autism spectrum disorder: the role of the mitochondria and the enteric microbiome.Treatments for biomedical abnormalities associated with autism spectrum disorder.Oxidative Stress Challenge Uncovers Trichloroacetaldehyde Hydrate-Induced Mitoplasticity in Autistic and Control Lymphoblastoid Cell Lines.Central pathways causing fatigue in neuro-inflammatory and autoimmune illnesses.Increased susceptibility to ethylmercury-induced mitochondrial dysfunction in a subset of autism lymphoblastoid cell lines.Modulation of the genome and epigenome of individuals susceptible to autism by environmental risk factorsPhenotyping, Etiological Factors, and Biomarkers: Toward Precision Medicine in Autism Spectrum Disorders.The Effect of Mitochondrial Supplements on Mitochondrial Activity in Children with Autism Spectrum Disorder.Epigenetic Treatment of Neuropsychiatric Disorders: Autism and Schizophrenia.Mitochondrial and redox abnormalities in autism lymphoblastoid cells: a sibling control study.Autistic Siblings with Novel Mutations in Two Different Genes: Insight for Genetic Workups of Autistic Siblings and Connection to Mitochondrial Dysfunction.Mitochondrial dysfunction in the gastrointestinal mucosa of children with autism: A blinded case-control study.Bioenergetic variation is related to autism symptomatology.Liver-specific deletion of RORα aggravates diet-induced nonalcoholic steatohepatitis by inducing mitochondrial dysfunction.Mitochondria As Sources and Targets of Methane.Modulation of Immunological Pathways in Autistic and Neurotypical Lymphoblastoid Cell Lines by the Enteric Microbiome Metabolite Propionic Acid.Decreased total antioxidant capacity has a larger effect size than increased oxidant levels in urine in individuals with autism spectrum disorder.Butyrate enhances mitochondrial function during oxidative stress in cell lines from boys with autism.Clinical and Molecular Characteristics of Mitochondrial Dysfunction in Autism Spectrum Disorder
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P2860
Oxidative stress induces mitochondrial dysfunction in a subset of autistic lymphoblastoid cell lines.
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2014 nî lūn-bûn
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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած
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2014 թվականի ապրիլին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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name
Oxidative stress induces mitoc ...... tic lymphoblastoid cell lines.
@ast
Oxidative stress induces mitoc ...... tic lymphoblastoid cell lines.
@en
Oxidative stress induces mitoc ...... tic lymphoblastoid cell lines.
@nl
type
label
Oxidative stress induces mitoc ...... tic lymphoblastoid cell lines.
@ast
Oxidative stress induces mitoc ...... tic lymphoblastoid cell lines.
@en
Oxidative stress induces mitoc ...... tic lymphoblastoid cell lines.
@nl
prefLabel
Oxidative stress induces mitoc ...... tic lymphoblastoid cell lines.
@ast
Oxidative stress induces mitoc ...... tic lymphoblastoid cell lines.
@en
Oxidative stress induces mitoc ...... tic lymphoblastoid cell lines.
@nl
P2093
P2860
P356
P1476
Oxidative stress induces mitoc ...... tic lymphoblastoid cell lines.
@en
P2093
J Slattery
P2860
P2888
P356
10.1038/TP.2014.15
P577
2014-04-01T00:00:00Z
P5875
P6179
1028908288