Major reduction of atherosclerosis in fractalkine (CX3CL1)-deficient mice is at the brachiocephalic artery, not the aortic root.
about
An oxidized lipid-peroxisome proliferator-activated receptor gamma-chemokine pathway in the regulation of macrophage-vascular smooth muscle cell adhesionRegulation of atherogenesis by chemokines and chemokine receptorsHydrogen sulfide inhibits the development of atherosclerosis with suppressing CX3CR1 and CX3CL1 expressionInflammatory cell recruitment in cardiovascular disease: murine models and potential clinical applicationsSevere Atherosclerosis and Hypercholesterolemia in Mice Lacking Both the Melanocortin Type 4 Receptor and Low Density Lipoprotein ReceptorGreater fractalkine expression in mesenteric arteries of female spontaneously hypertensive rats compared with malesDo not let death do us part: 'find-me' signals in communication between dying cells and the phagocytes.Influence of phthalates on glucose homeostasis and atherosclerosis in hyperlipidemic miceVascular cells contribute to atherosclerosis by cytokine- and innate-immunity-related inflammatory mechanisms.Diabetic conditions promote binding of monocytes to vascular smooth muscle cells and their subsequent differentiationCX3CR1 deficiency does not influence trafficking of adipose tissue macrophages in mice with diet-induced obesity.Aortic arch curvature and atherosclerosis have overlapping quantitative trait loci in a cross between 129S6/SvEvTac and C57BL/6J apolipoprotein E-null miceIndoxyl sulfate (IS)-mediated immune dysfunction provokes endothelial damage in patients with end-stage renal disease (ESRD).Fractalkine (CX3CL1), GM-CSF and VEGF-a levels are reduced by statins in adult patients.Identifying novel genes for atherosclerosis through mouse-human comparative genetics.Wilson disease at a single cell level: intracellular copper trafficking activates compartment-specific responses in hepatocytes.Atherosclerosis quantitative trait loci are sex- and lineage-dependent in an intercross of C57BL/6 and FVB/N low-density lipoprotein receptor-/- miceEvaluation of foam cell formation in cultured macrophages: an improved method with Oil Red O staining and DiI-oxLDL uptake.Fractalkine is expressed in early and advanced atherosclerotic lesions and supports monocyte recruitment via CX3CR1.Quantitative trait loci mapping of the mouse plasma proteome (pQTL)Fractalkine promotes human monocyte survival via a reduction in oxidative stressChemokine receptor CCR1 disruption in bone marrow cells enhances atherosclerotic lesion development and inflammation in miceNo reduction of atherosclerosis in C-reactive protein (CRP)-deficient mice.The Role of Efferocytosis in Atherosclerosis.Suppressed monocyte recruitment drives macrophage removal from atherosclerotic plaques of Apoe-/- mice during disease regressionInnate immunity and monocyte-macrophage activation in atherosclerosis.Monocyte subsets differentially employ CCR2, CCR5, and CX3CR1 to accumulate within atherosclerotic plaques.Shear stress-induced changes in atherosclerotic plaque composition are modulated by chemokines.Cytokines in atherosclerosis: Key players in all stages of disease and promising therapeutic targets.NADPH oxidase 4 mediates monocyte priming and accelerated chemotaxis induced by metabolic stress.A disintegrin and metalloprotease 17 mediates neointimal hyperplasia in vasculature.In vivo structure/function and expression analysis of the CX3C chemokine fractalkineCD40-TRAF Signaling Upregulates CX3CL1 and TNF-α in Human Aortic Endothelial Cells but Not in Retinal Endothelial Cells.Anti-beta2GPI-antibody-induced endothelial cell gene expression profiling reveals induction of novel pro-inflammatory genes potentially involved in primary antiphospholipid syndrome.Thematic review series: The immune system and atherogenesis. Cytokines affecting endothelial and smooth muscle cells in vascular disease.Chemokine binding protein 'M3' limits atherosclerosis in apolipoprotein E-/- miceSexual dimorphism in rodent models of hypertension and atherosclerosis.Fractalkine deficiency markedly reduces macrophage accumulation and atherosclerotic lesion formation in CCR2-/- mice: evidence for independent chemokine functions in atherogenesis.CX3CL1 up-regulation is associated with recruitment of CX3CR1+ mononuclear phagocytes and T lymphocytes in the lungs during cigarette smoke-induced emphysema.CX3CL1 and CX3CR1 in the GL261 murine model of glioma: CX3CR1 deficiency does not impact tumor growth or infiltration of microglia and lymphocytes.
P2860
Q24300694-73FAD33E-5C48-4F51-8544-ED7DE28D9253Q27003124-4DF34359-A290-4D79-A533-C386844FFA42Q27332432-20968590-1416-4CC6-86AA-A15FA4A92F9AQ28275259-325DA18E-BEF2-4BE1-A1F7-D238326B86FAQ28468525-5A6016F1-2961-4E80-BFFC-FCEB7BA477E3Q28571899-355B13B9-2F59-4949-9D16-78868DC64B9AQ30277318-214E6F56-82E8-42B1-BBEE-2B4910987A0BQ30300182-53193333-C87C-4C79-8BB8-095F919523ECQ30319434-94E2AE51-E9DA-4D66-80C5-00C80703167CQ30433016-6094B385-EF13-4BF0-9D62-6A5493A8C24FQ33554073-E70D5E9A-9FFA-41CE-BA07-815CBC8C8E07Q33767143-A9AC1D74-A7A7-4CF2-BB3B-86600F5BE115Q33780032-D93C76CE-C7B6-4006-8D1B-C77EF32C9CD5Q33794941-5A5F962C-5EB0-4665-AC51-894C6A6A141CQ34021005-30589F01-17E1-475F-BA8D-75568AD302A1Q34155396-18863BD4-9100-474B-A254-0A3653047725Q34248316-A83A0D0E-28CC-498D-897D-0F27F04CAFF5Q34361591-9C0C468F-329F-4F5E-871F-D4647AC83F24Q34391705-4A2F93FE-0F46-455F-9EC7-5133A46D581DQ34485032-8BE792DD-00E8-42DE-9E2C-9F30857B8665Q34536235-CA91DAB2-6993-46C3-A5D0-930BF6699B78Q34572318-AA5DE037-68DA-49D9-94A6-F256909C4FA9Q34675880-E0D83C93-351C-40E6-888D-C02005326F46Q34680660-A7968129-DABA-48D7-81E5-9EA6BAE5E6EBQ34876397-4E73DD54-1C7D-4AE9-B210-4A3B42443508Q34980214-30B88338-07C4-45BD-913F-84537ADC1FD3Q35250952-053464AC-6A46-4E56-94BA-C0780780552EQ35629011-08747FA3-0560-4514-8180-A0C5256DE350Q35640438-69B30335-0352-446B-8535-547EC1358BB3Q35683209-92B6AFCC-A8AE-4345-B125-1912BB2F71E4Q35848839-D93000A0-8AF1-49BA-941D-45EEC952023EQ35865080-0F855288-5B12-4918-990E-2034A5C12568Q35879683-7346D611-FFCF-4E58-B99C-8CD3C1D31BD4Q35953541-CB375877-BAE0-4462-97CC-B72ECD9B982EQ36099980-729F73B0-28D8-482E-B171-AC79A6714B0BQ36304258-B9FD0DDE-DA7C-4E1D-A2AF-0046E03EA3CAQ36346910-90EFC854-A716-49A3-A5E2-FB2EACD40FB1Q36660105-83321F63-5D09-4BB3-8C43-A79BD4E1AE74Q36891487-65FAD880-C78F-4BCD-803D-249D57B5720CQ36922514-D450E141-BC2B-49E7-9C74-0030E4A86EF5
P2860
Major reduction of atherosclerosis in fractalkine (CX3CL1)-deficient mice is at the brachiocephalic artery, not the aortic root.
description
2004 nî lūn-bûn
@nan
2004 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Major reduction of atheroscler ...... c artery, not the aortic root.
@ast
Major reduction of atheroscler ...... c artery, not the aortic root.
@en
Major reduction of atheroscler ...... c artery, not the aortic root.
@nl
type
label
Major reduction of atheroscler ...... c artery, not the aortic root.
@ast
Major reduction of atheroscler ...... c artery, not the aortic root.
@en
Major reduction of atheroscler ...... c artery, not the aortic root.
@nl
prefLabel
Major reduction of atheroscler ...... c artery, not the aortic root.
@ast
Major reduction of atheroscler ...... c artery, not the aortic root.
@en
Major reduction of atheroscler ...... c artery, not the aortic root.
@nl
P2093
P2860
P356
P1476
Major reduction of atheroscler ...... c artery, not the aortic root.
@en
P2093
Jan L Breslow
Jose-Carlos Gutierrez-Ramos
Marietta Tan
Roland Kolbeck
Stephanos Pavlides
P2860
P304
17795-17800
P356
10.1073/PNAS.0408096101
P407
P577
2004-12-13T00:00:00Z