Toll-like receptor-4 inhibits enterocyte proliferation via impaired beta-catenin signaling in necrotizing enterocolitis. - Wikidata - thesis-toulmin - TriplyDB

Toll-like receptor-4 inhibits enterocyte proliferation via impaired beta-catenin signaling in necrotizing enterocolitis.

Toll-like receptor-4 inhibits enterocyte proliferation via impaired beta-catenin signaling in necrotizing enterocolitis.

http://www.wikidata.org/entity/Q33618589

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Inflammatory signaling in necrotizing enterocolitis Intestinal stem cell growth and differentiation on a tubular scaffold with evaluation in small and large animals.Necrotizing enterocolitis: new insights into pathogenesis and mechanisms TLR4 activates the β-catenin pathway to cause intestinal neoplasia Discovery and validation of a new class of small molecule Toll-like receptor 4 (TLR4) inhibitors WNT1-inducible signaling pathway protein 1 contributes to ventilator-induced lung injury Intestinal dysbiosis in preterm infants preceding necrotizing enterocolitis: a systematic review and meta-analysis.Damage-associated molecular patterns generated in osteoarthritis directly excite murine nociceptive neurons through Toll-like receptor 4.Commensal and probiotic bacteria may prevent NEC by maturing intestinal host defenses.Intestinal stem cells and their roles during mucosal injury and repair.Genetic deletion of toll-like receptor 4 on platelets attenuates experimental pulmonary hypertension.The Research Progress on Intestinal Stem Cells and Its Relationship with Intestinal Microbiota.Paneth-cell-disruption-induced necrotizing enterocolitis in mice requires live bacteria and occurs independently of TLR4 signaling Circulating fibrocytes are involved in inflammation and leukocyte trafficking in neonates with necrotizing enterocolitis Lactobacillus reuteri DSM 17938 differentially modulates effector memory T cells and Foxp3+ regulatory T cells in a mouse model of necrotizing enterocolitis.Evidence-based feeding strategies before and after the development of necrotizing enterocolitis.Update in pathogenesis and prospective in treatment of necrotizing enterocolitis.Nucleotide-binding oligomerization domain-2 inhibits toll-like receptor-4 signaling in the intestinal epithelium.Novel role for the innate immune receptor Toll-like receptor 4 (TLR4) in the regulation of the Wnt signaling pathway and photoreceptor apoptosis Transactivation of EGFR by LPS induces COX-2 expression in enterocytes.Bifidobacterium bifidum reduces apoptosis in the intestinal epithelium in necrotizing enterocolitis.Enteric bacteria and cancer stem cells.Host-microbiota interaction and intestinal stem cells in chronic inflammation and colorectal cancer.The mechanism of excessive intestinal inflammation in necrotizing enterocolitis: an immature innate immune response.DNA attenuates enterocyte Toll-like receptor 4-mediated intestinal mucosal injury after remote trauma.Antimicrobial peptides and gut microbiota in homeostasis and pathology.Necrotizing enterocolitis in newborns: update in pathophysiology and newly emerging therapeutic strategies.Differential effects of β-catenin and NF-κB interplay in the regulation of cell proliferation, inflammation and tumorigenesis in response to bacterial infection Worms, flies and four-legged friends: the applicability of biological models to the understanding of intestinal inflammatory diseases.New insights into the pathogenesis and treatment of necrotizing enterocolitis: Toll-like receptors and beyond How the Intricate Interaction among Toll-Like Receptors, Microbiota, and Intestinal Immunity Can Influence Gastrointestinal Pathology Intracellular heat shock protein-70 negatively regulates TLR4 signaling in the newborn intestinal epithelium.TLR4 Activation Promotes Bone Marrow MSC Proliferation and Osteogenic Differentiation via Wnt3a and Wnt5a Signaling Breast milk protects against the development of necrotizing enterocolitis through inhibition of Toll-like receptor 4 in the intestinal epithelium via activation of the epidermal growth factor receptor Amniotic fluid inhibits Toll-like receptor 4 signaling in the fetal and neonatal intestinal epithelium.Gut mucosal injury in neonates is marked by macrophage infiltration in contrast to pleomorphic infiltrates in adult: evidence from an animal model Elevated lipopolysaccharide in the colon evokes intestinal inflammation, aggravated in immune modulator-impaired mice.Toll-like receptor 4 is expressed on intestinal stem cells and regulates their proliferation and apoptosis via the p53 up-regulated modulator of apoptosis High mobility group box 1 contributes to the pathogenesis of experimental pulmonary hypertension via activation of Toll-like receptor 4 Intestinal epithelial Toll-like receptor 4 regulates goblet cell development and is required for necrotizing enterocolitis in mice.

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P2860

Toll-like receptor-4 inhibits enterocyte proliferation via impaired beta-catenin signaling in necrotizing enterocolitis.

http://www.wikidata.org/entity/Q33618589

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2009 nî lūn-bûn

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2009 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2009 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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Toll-like receptor-4 inhibits ...... in necrotizing enterocolitis.

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J.GASTRO.2009.09.045

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Gastroenterology

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Toll-like receptor-4 inhibits ...... in necrotizing enterocolitis.

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Anthony Russo

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Chhinder P Sodhi

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Congrong Ma

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David J Hackam

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Maria Branca

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Richard A Shapiro

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Steven C Gribar

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Thomas Prindle

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Ward M Richardson

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Xia-Hua Shi

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P2860

Is NF-kappaB a good target for cancer therapy? Hopes and pitfalls

GSK-3: tricks of the trade for a multi-tasking kinase

Specificity and mechanism of action of some commonly used protein kinase inhibitors

Inducible Cre-mediated control of gene expression in the murine gastrointestinal tract: effect of loss of beta-catenin

Tyrosine residues 654 and 670 in beta-catenin are crucial in regulation of Met-beta-catenin interactions

Toll-like receptor-4 promotes the development of colitis-associated colorectal tumors.

The roles of bacteria and TLR4 in rat and murine models of necrotizing enterocolitis

Differential alteration in intestinal epithelial cell expression of toll-like receptor 3 (TLR3) and TLR4 in inflammatory bowel disease.

Wnt and beyond Wnt: multiple mechanisms control the transcriptional property of beta-catenin.

Impaired enterocyte proliferation in aquaporin-3 deficiency in mouse models of colitis.

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185-196

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10.1053/J.GASTRO.2009.09.045

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1

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26852979

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19786028

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Toll-like receptor

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