A molecular signature of proteinuria in glomerulonephritis.
about
Anti-GBM glomerulonephritis involves IL-1 but is independent of NLRP3/ASC inflammasome-mediated activation of caspase-1.Identification of nephropathy candidate genes by comparing sclerosis-prone and sclerosis-resistant mouse strain kidney transcriptomes.The primary glomerulonephritides: a systems biology approach.Defining nephrotic syndrome from an integrative genomics perspective.Osteoprotegerin in exosome-like vesicles from human cultured tubular cells and urineA 3-biomarker-panel predicts renal outcome in patients with proteinuric renal diseases.Epithelial-to-mesenchymal transition induces cell cycle arrest and parenchymal damage in renal fibrosis.Lipoxins attenuate renal fibrosis by inducing let-7c and suppressing TGFβR1.Tissue transcriptome-driven identification of epidermal growth factor as a chronic kidney disease biomarkerAmelioration of albuminuria in ROCK1 knockout mice with streptozotocin-induced diabetic kidney diseaseDefining cell-type specificity at the transcriptional level in human diseaseThe genetics of diabetic nephropathy.Tet3-mediated hydroxymethylation of epigenetically silenced genes contributes to bone morphogenic protein 7-induced reversal of kidney fibrosis.Notch-3 receptor activation drives inflammation and fibrosis following tubulointerstitial kidney injury.NLRP3 Localizes to the Tubular Epithelium in Human Kidney and Correlates With Outcome in IgA NephropathyIdentification of potential biomarkers and therapeutic targets for human IgA nephropathy and hypertensive nephropathy by bioinformatics analysis.Bcl3: a regulator of NF-κB inducible by TWEAK in acute kidney injury with anti-inflammatory and antiapoptotic properties in tubular cells.Non-canonical NFκB activation promotes chemokine expression in podocytesNovel avenues for drug discovery in diabetic kidney disease.Canadians Seeking Solutions and Innovations to Overcome Chronic Kidney Disease (Can-SOLVE CKD): Form and Function.Transcriptomics in kidney biopsy is an untapped resource for precision therapy in nephrology: a systematic review.Pharmacological induction of hypoxia-inducible transcription factor ARNT attenuates chronic kidney failure.Adiponectin attenuates angiotensin II-induced oxidative stress in renal tubular cells through AMPK and cAMP-Epac signal transduction pathways.
P2860
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P2860
A molecular signature of proteinuria in glomerulonephritis.
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
A molecular signature of proteinuria in glomerulonephritis.
@ast
A molecular signature of proteinuria in glomerulonephritis.
@en
type
label
A molecular signature of proteinuria in glomerulonephritis.
@ast
A molecular signature of proteinuria in glomerulonephritis.
@en
prefLabel
A molecular signature of proteinuria in glomerulonephritis.
@ast
A molecular signature of proteinuria in glomerulonephritis.
@en
P2093
P2860
P1433
P1476
A molecular signature of proteinuria in glomerulonephritis.
@en
P2093
Andrew M Herzenberg
Anissa Boucherot
Anna Henger
Celine C Berthier
Clemens D Cohen
Daniel C Cattran
David Tritchler
Felix Eichinger
Heather N Reich
James W Scholey
P2860
P304
P356
10.1371/JOURNAL.PONE.0013451
P407
P577
2010-10-18T00:00:00Z