Uncoupling of expression of an intronic microRNA and its myosin host gene by exon skipping - Wikidata - thesis-toulmin - TriplyDB

Uncoupling of expression of an intronic microRNA and its myosin host gene by exon skipping

Uncoupling of expression of an intronic microRNA and its myosin host gene by exon skipping

http://www.wikidata.org/entity/Q33769212

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Functional shifts in insect microRNA evolution Myostatin: expanding horizons Does regulation of skeletal muscle function involve circulating microRNAs?MicroRNAs as biomarkers for myocardial infarction Helicase-like transcription factor (Hltf) regulates G2/M transition, Wt1/Gata4/Hif-1a cardiac transcription networks, and collagen biogenesis MiR-499 regulates cell proliferation and apoptosis during late-stage cardiac differentiation via Sox6 and cyclin D1 From Nutrient to MicroRNA: a Novel Insight into Cell Signaling Involved in Skeletal Muscle Development and Disease Myh7b/miR-499 gene expression is transcriptionally regulated by MRFs and Eos Developmental expression and cardiac transcriptional regulation of Myh7b, a third myosin heavy chain in the vertebrate heart miR-499 protects cardiomyocytes from H 2O 2-induced apoptosis via its effects on Pdcd4 and Pacs2.Elevated miR-499 levels blunt the cardiac stress response.Genome-wide mapping of Sox6 binding sites in skeletal muscle reveals both direct and indirect regulation of muscle terminal differentiation by Sox6.The myriad essential roles of microRNAs in cardiovascular homeostasis and disease.Posttranscriptional mechanisms involving microRNA-27a and b contribute to fast-specific and glucocorticoid-mediated myostatin expression in skeletal muscle.MicroRNAs in cardiac disease Evolution of the myosin heavy chain gene MYH14 and its intronic microRNA miR-499: muscle-specific miR-499 expression persists in the absence of the ancestral host gene.Egfl7 is differentially expressed in arteries and veins during retinal vascular development.Increased expression of GDF-15 may mediate ICU-acquired weakness by down-regulating muscle microRNAs.MicroRNA-499 expression distinctively correlates to target genes sox6 and rod1 profiles to resolve the skeletal muscle phenotype in Nile tilapia.Evolution and genomic organization of muscle microRNAs in fish genomes Influence of microRNA-related polymorphisms on clinical outcomes in coronary artery disease MicroRNAs: Processing, Maturation, Target Recognition and Regulatory Functions A human 3' miR-499 mutation alters cardiac mRNA targeting and function.Mechanical Stretch Inhibits MicroRNA499 via p53 to Regulate Calcineurin-A Expression in Rat Cardiomyocytes Identification of Targets of CUG-BP, Elav-Like Family Member 1 (CELF1) Regulation in Embryonic Heart Muscle.Evolution and Distribution of Teleost myomiRNAs: Functionally Diversified myomiRs in Teleosts.Direct and indirect involvement of microRNA-499 in clinical and experimental cardiomyopathy.Hsa-mir-499 rs3746444 T/C Polymorphism is Associated with Increased Risk of Coronary Artery Disease in a Chinese Population.MicroRNA-208b progressively declines after spinal cord injury in humans and is inversely related to myostatin expression.Sulforaphane causes a major epigenetic repression of myostatin in porcine satellite cells The role of microRNAs in skeletal muscle health and disease Trip12, a HECT domain E3 ubiquitin ligase, targets Sox6 for proteasomal degradation and affects fiber type-specific gene expression in muscle cells.Nuclear receptor/microRNA circuitry links muscle fiber type to energy metabolism.Early de novo DNA methylation and prolonged demethylation in the muscle lineage.The MyomiR network in skeletal muscle plasticity.Mechanisms of muscle gene regulation in the electric organ of Sternopygus macrurus.Coupling of mitochondrial function and skeletal muscle fiber type by a miR-499/Fnip1/AMPK circuit.A genome-wide association study of congenital cardiovascular left-sided lesions shows association with a locus on chromosome 20.Role of microRNAs in skeletal muscle hypertrophy.Negative auto-regulation of myostatin expression is mediated by Smad3 and microRNA-27.

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Uncoupling of expression of an intronic microRNA and its myosin host gene by exon skipping

http://www.wikidata.org/entity/Q33769212

description

2010 nî lūn-bûn

@nan

2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Uncoupling of expression of an ...... sin host gene by exon skipping

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Uncoupling of expression of an ...... sin host gene by exon skipping

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Uncoupling of expression of an ...... sin host gene by exon skipping

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Uncoupling of expression of an ...... sin host gene by exon skipping

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Uncoupling of expression of an ...... sin host gene by exon skipping

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Molecular and Cellular Biology

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Uncoupling of expression of an ...... sin host gene by exon skipping

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Leslie A Leinwand

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Massimo Buvoli

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Matthew L Bell

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P2860

Comparative sequence analysis of the complete human sarcomeric myosin heavy chain family: implications for functional diversity

MicroRNA-145 regulates OCT4, SOX2, and KLF4 and represses pluripotency in human embryonic stem cells

Microarray profiling of microRNAs reveals frequent coexpression with neighboring miRNAs and host genes

A feedback regulatory loop involving microRNA-9 and nuclear receptor TLX in neural stem cell fate determination

Sox6 regulation of cardiac myocyte development

Processing of intronic microRNAs

Sox6 is a candidate gene for p100H myopathy, heart block, and sudden neonatal death

Rapid turnover of extracellular signal-regulated kinase 3 by the ubiquitin-proteasome pathway defines a novel paradigm of mitogen-activated protein kinase regulation during cellular differentiation

Prediction of the coding sequences of unidentified human genes. XVII. The complete sequences of 100 new cDNA clones from brain which code for large proteins in vitro

Myosin gene mutation correlates with anatomical changes in the human lineage

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1937-1945

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scholarly article

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10.1128/MCB.01370-09

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8

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30

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2010-02-12T00:00:00Z

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20154144

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2849460

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