Adventitial fibroblasts induce a distinct proinflammatory/profibrotic macrophage phenotype in pulmonary hypertension. - Wikidata - thesis-toulmin - TriplyDB

Adventitial fibroblasts induce a distinct proinflammatory/profibrotic macrophage phenotype in pulmonary hypertension.

Adventitial fibroblasts induce a distinct proinflammatory/profibrotic macrophage phenotype in pulmonary hypertension.

http://www.wikidata.org/entity/Q33909537

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Macrophages in vascular inflammation--From atherosclerosis to vasculitis Contribution of metabolic reprogramming to macrophage plasticity and function Developmental origins of NAFLD: a womb with a clue.Hypoxia-Inducible Factor 1α Is a Critical Downstream Mediator for Hypoxia-Induced Mitogenic Factor (FIZZ1/RELMα)-Induced Pulmonary Hypertension.Macrophage arginase-1 controls bacterial growth and pathology in hypoxic tuberculosis granulomas.The role of inflammation in hypoxic pulmonary hypertension: from cellular mechanisms to clinical phenotypes.Hemoglobin-induced lung vascular oxidation, inflammation, and remodeling contribute to the progression of hypoxic pulmonary hypertension and is attenuated in rats with repeated-dose haptoglobin administration.Emerging translational approaches to target STAT3 signalling and its impact on vascular disease In Situ Characterization of Splenic Brucella melitensis Reservoir Cells during the Chronic Phase of Infection in Susceptible Mice Immune and Inflammatory Cell Composition of Human Lung Cancer Stroma.The Mycoplasma hyorhinis p37 Protein Rapidly Induces Genes in Fibroblasts Associated with Inflammation and Cancer.Mesenchymal stem cell-derived inflammatory fibroblasts promote monocyte transition into myeloid fibroblasts via an IL-6-dependent mechanism in the aging mouse heart.Macrophage depletion lowers blood pressure and restores sympathetic nerve α2-adrenergic receptor function in mesenteric arteries of DOCA-salt hypertensive rats Toll-like Receptors in the Vascular System: Sensing the Dangers Within.Raf/ERK drives the proliferative and invasive phenotype of BMPR2-silenced pulmonary artery endothelial cells.The Metabolic Prospective and Redox Regulation of Macrophage Polarization.Metabolic reprogramming and inflammation act in concert to control vascular remodeling in hypoxic pulmonary hypertension.Amino acid auxotrophy as a system of immunological control nodes.Constitutive Reprogramming of Fibroblast Mitochondrial Metabolism in Pulmonary Hypertension.Transcription factors, transcriptional coregulators, and epigenetic modulation in the control of pulmonary vascular cell phenotype: therapeutic implications for pulmonary hypertension (2015 Grover Conference series).Dimethyl Fumarate ameliorates pulmonary arterial hypertension and lung fibrosis by targeting multiple pathways Mesenchymal stem cell-derived inflammatory fibroblasts mediate interstitial fibrosis in the aging heart.Hallmarks of Pulmonary Hypertension: Mesenchymal and Inflammatory Cell Metabolic Reprogramming.Aortic adventitial fibroblast sensitivity to mitogen activated protein kinase inhibitors depends on substrate stiffness.Aberrant immune response with consequent vascular and connective tissue remodeling - causal to scleroderma and associated syndromes such as Raynaud phenomenon and other fibrosing syndromes?TNF Counterbalances the Emergence of M2 Tumor Macrophages M1-like Macrophage Polarization Promotes Orthodontic Tooth Movement.Pulmonary endothelial cell DNA methylation signature in pulmonary arterial hypertension.Safety and Efficacy of Immunoadsorption as an Add-On to Medical Treatment in Patients with Severe Idiopathic Pulmonary Arterial Hypertension.Metabolic Reprogramming Regulates the Proliferative and Inflammatory Phenotype of Adventitial Fibroblasts in Pulmonary Hypertension Through the Transcriptional Corepressor C-Terminal Binding Protein-1.Leukotriene B4 Activates Pulmonary Artery Adventitial Fibroblasts in Pulmonary Hypertension.The Roles of Immunity in the Prevention and Evolution of Pulmonary Arterial Hypertension.Pentose Shunt, Glucose-6-Phosphate Dehydrogenase, NADPH Redox, and Stem Cells in Pulmonary Hypertension.Niclosamide Prevents Systemic Sclerosis in a Reactive Oxygen Species-Induced Mouse Model.Metabolic Reprogramming and Redox Signaling in Pulmonary Hypertension.Metabolic and Proliferative State of Vascular Adventitial Fibroblasts in Pulmonary Hypertension Is Regulated Through a MicroRNA-124/PTBP1 (Polypyrimidine Tract Binding Protein 1)/Pyruvate Kinase Muscle Axis.A Time- and Compartment-Specific Activation of Lung Macrophages in Hypoxic Pulmonary Hypertension.Separate roles of IL-6 and oncostatin M in mouse macrophage polarization in vitro and in vivo.Nonresolving macrophage-mediated inflammation in malignancy.Cyp2c44 gene disruption is associated with increased hematopoietic stem cells: implication in chronic hypoxia-induced pulmonary hypertension.

P2860

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P2860

Adventitial fibroblasts induce a distinct proinflammatory/profibrotic macrophage phenotype in pulmonary hypertension.

http://www.wikidata.org/entity/Q33909537

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2014 nî lūn-bûn

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2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2014 թվականի հունիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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Adventitial fibroblasts induce ...... ype in pulmonary hypertension.

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Adventitial fibroblasts induce ...... ype in pulmonary hypertension.

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Adventitial fibroblasts induce ...... ype in pulmonary hypertension.

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Adventitial fibroblasts induce ...... ype in pulmonary hypertension.

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JIMMUNOL.1303048

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Adventitial fibroblasts induce ...... ype in pulmonary hypertension.

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Aimee L Anderson

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Brian B Graham

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Holger K Eltzschig

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Jacob E Friedman

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Jens M Poth

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Karim C El Kasmi

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Kurt R Stenmark

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Maria A Nagel

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Maria G Frid

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Mehdi A Fini

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P2860

Control of T(H)17/T(reg) balance by hypoxia-inducible factor 1

A CREB-C/EBPbeta cascade induces M2 macrophage-specific gene expression and promotes muscle injury repair

Targeted disruption of the mouse Stat3 gene leads to early embryonic lethality

Host responses in tissue repair and fibrosis

The adventitia: essential regulator of vascular wall structure and function

Alternative activation of macrophages

Enhancer-mediated control of macrophage-specific arginase I expression

Protective and pathogenic functions of macrophage subsets

Interferon and alternative activation of monocyte/macrophages in systemic sclerosis-associated pulmonary arterial hypertension.

Mice deficient in Mkp-1 develop more severe pulmonary hypertension and greater lung protein levels of arginase in response to chronic hypoxia.

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Soni Savai Pullamsetti

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24928992

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