Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis - Wikidata - thesis-toulmin - TriplyDB

Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis

Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis

http://www.wikidata.org/entity/Q33932297

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Protein microarrays discover angiotensinogen and PRKRIP1 as novel targets for autoantibodies in chronic renal disease Fibroblast growth factor 23 inhibits extrarenal synthesis of 1,25-dihydroxyvitamin D in human monocytes Phosphate and FGF-23 The FGF23-Klotho axis: endocrine regulation of phosphate homeostasis Hyperphosphatemia Management in Patients with Chronic Kidney Disease Vascular calcification, bone and mineral metabolism after kidney transplantation Left Ventricular Hypertrophy in Chronic Kidney Disease Patients: From Pathophysiology to Treatment.FGF23 in Acute and Chronic Illness The role of phosphorus in the development and progression of vascular calcification Miscellaneous non-inflammatory musculoskeletal conditions. Hyperphosphatemic familial tumoral calcinosis (FGF23, GALNT3 and αKlotho)The rise of multiple imputation: a review of the reporting and implementation of the method in medical research Fibroblast growth factor 23 Hyperphosphatemia in patients with ESRD: assessing the current evidence linking outcomes with treatment adherence Skeletal secretion of FGF-23 regulates phosphate and vitamin D metabolism Phosphate control in end-stage renal disease: barriers and opportunities Regulation and function of the FGF23/klotho endocrine pathways Calcium regulation and bone mineral metabolism in elderly patients with chronic kidney disease Bone disease in pediatric chronic kidney disease Fibroblast Growth Factor 23: A New Dimension to Diseases of Calcium-Phosphorus Metabolism Phosphorus and the kidney: What is known and what is needed Role of FGF23 in vitamin D and phosphate metabolism: implications in chronic kidney disease Is there an association between elevated or low serum levels of phosphorus, parathyroid hormone, and calcium and mortality in patients with end stage renal disease? A meta-analysis Evidence for the prevention and treatment of stroke in dialysis patients Cardiovascular Risk Factors and Chronic Kidney Disease-FGF23: A Key Molecule in the Cardiovascular Disease Arterial stiffness, vascular calcification and bone metabolism in chronic kidney disease FGF23 signaling impairs neutrophil recruitment and host defense during CKD.Calcium and phosphate impact cardiovascular risk.FGF23 antagonism: the thin line between adaptation and maladaptation in chronic kidney disease.Novel biological markers of bone: from bone metabolism to bone physiology Vascular calcification: When should we interfere in chronic kidney disease patients and how?Fibroblast Growth Factor-23-A Potential Uremic Toxin Cardiovascular biomarkers in chronic kidney disease: state of current research and clinical applicability Biomarkers in chronic kidney disease, from kidney function to kidney damage Circulating FGF21 levels are progressively increased from the early to end stages of chronic kidney diseases and are associated with renal function in Chinese FGF23 fails to inhibit uremic parathyroid glands Intravenous phosphate loading increases fibroblast growth factor 23 in uremic rats Regulation of renal phosphate transport by FGF23 is mediated by FGFR1 and FGFR4 Evolution of Cardiovascular Disease During the Transition to End-Stage Renal Disease.Potential pathophysiological role for the vitamin D deficiency in essential hypertension.Elevated fibroblast growth factor 23 is a risk factor for kidney transplant loss and mortality.

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Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis

http://www.wikidata.org/entity/Q33932297

description

2008 nî lūn-bûn

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2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի օգոստոսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis

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Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis

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type

label

Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis

@ast

Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis

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prefLabel

Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis

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Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis

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The New England Journal of Medicine

P1476

Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis

@en

P2093

Anand Shah

http://www.w3.org/2001/XMLSchema#string

Hang Lee

http://www.w3.org/2001/XMLSchema#string

Harald Jüppner

http://www.w3.org/2001/XMLSchema#string

Hector Tamez

http://www.w3.org/2001/XMLSchema#string

Jose Alejandro Rauh-Hain

http://www.w3.org/2001/XMLSchema#string

Kelsey Smith

http://www.w3.org/2001/XMLSchema#string

Michael Mannstadt

http://www.w3.org/2001/XMLSchema#string

Myles Wolf

http://www.w3.org/2001/XMLSchema#string

Orlando M Gutiérrez

http://www.w3.org/2001/XMLSchema#string

Ravi Thadhani

http://www.w3.org/2001/XMLSchema#string

P2860

Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23

Evidence for alteration of the vitamin D-endocrine system in blacks

Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia

The autosomal dominant hypophosphatemic rickets (ADHR) gene is a secreted polypeptide overexpressed by tumors that cause phosphate wasting

Chronic kidney disease and the risks of death, cardiovascular events, and hospitalization

The parathyroid is a target organ for FGF23 in rats

Circulating FGF-23 is regulated by 1alpha,25-dihydroxyvitamin D3 and phosphorus in vivo

Targeted ablation of Fgf23 demonstrates an essential physiological role of FGF23 in phosphate and vitamin D metabolism

Circulating concentration of FGF-23 increases as renal function declines in patients with chronic kidney disease, but does not change in response to variation in phosphate intake in healthy volunteers.

Analysis of the biochemical mechanisms for the endocrine actions of fibroblast growth factor-23

P304

584-592

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scholarly article

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10.1056/NEJMOA0706130

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P433

6

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359

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2008-08-01T00:00:00Z

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18687639

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2890264

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