Effect of noncompetitive blockade of N-methyl-D-aspartate receptors on the neurochemical sequelae of experimental brain injury.
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Current approaches to enhance glutamate transporter function and expressionMotor discoordination and increased susceptibility to cerebellar injury in GLAST mutant miceCurrent status of neuroprotection trials for traumatic brain injury: lessons from animal models and clinical studies.MK801 attenuates secondary injury in a mouse experimental compression model of spinal cord trauma.Melatonin, hydroxyl radical-mediated oxidative damage, and aging: a hypothesis.Traumatic brain injury: developmental differences in glutamate receptor response and the impact on treatment.N-methyl-D-aspartate receptor mechanosensitivity is governed by C terminus of NR2B subunit.In vitro stretch injury induces time- and severity-dependent alterations of STEP phosphorylation and proteolysis in neurons.Influence of alcohol on mortality in traumatic brain injury.Using anesthetics and analgesics in experimental traumatic brain injuryDrug targets for traumatic brain injury from poly(ADP-ribose)polymerase pathway modulation.Improvements in biomaterial matrices for neural precursor cell transplantationThe rationale for glutamate antagonists in the treatment of traumatic brain injury.Neurochemical mechanisms in brain injury and treatment: a review.The maxi-K channel opener BMS-204352 attenuates regional cerebral edema and neurologic motor impairment after experimental brain injury.Hippocampal CA3 lesion prevents postconcussive metabolic dysfunction in CA1.Effect of magnesium, MK-801 and combination of magnesium and MK-801 on blood-brain barrier permeability and brain edema after experimental traumatic diffuse brain injury.Alterations in ionized and total blood magnesium after experimental traumatic brain injury: relationship to neurobehavioral outcome and neuroprotective efficacy of magnesium chloride.Superoxide dismutase activity and the effect of N-methyl-D-aspartate antagonists on lipid peroxidation in the early phase of cold injury.Apoptotic neurodegeneration following trauma is markedly enhanced in the immature brain.Alterations in inducible 72-kDa heat shock protein and the chaperone cofactor BAG-1 in human brain after head injury.Calcium movements in traumatic brain injury: the role of glutamate receptor-operated ion channels.Attenuation of brain edema, blood-brain barrier breakdown, and injury volume by ifenprodil, a polyamine-site N-methyl-D-aspartate receptor antagonist, after experimental traumatic brain injury in rats.Prevention of trauma-induced neurodegeneration in infant rat brain.
P2860
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P2860
Effect of noncompetitive blockade of N-methyl-D-aspartate receptors on the neurochemical sequelae of experimental brain injury.
description
1990 nî lūn-bûn
@nan
1990 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
name
Effect of noncompetitive block ...... of experimental brain injury.
@ast
Effect of noncompetitive block ...... of experimental brain injury.
@en
type
label
Effect of noncompetitive block ...... of experimental brain injury.
@ast
Effect of noncompetitive block ...... of experimental brain injury.
@en
prefLabel
Effect of noncompetitive block ...... of experimental brain injury.
@ast
Effect of noncompetitive block ...... of experimental brain injury.
@en
P2093
P2860
P1476
Effect of noncompetitive block ...... of experimental brain injury.
@en
P2093
P2860
P304
P356
10.1111/J.1471-4159.1990.TB03122.X
P407
P577
1990-10-01T00:00:00Z