Blockade of self-reactive IgM significantly reduces injury in a murine model of acute myocardial infarction - Wikidata - thesis-toulmin - TriplyDB

Blockade of self-reactive IgM significantly reduces injury in a murine model of acute myocardial infarction

Blockade of self-reactive IgM significantly reduces injury in a murine model of acute myocardial infarction

http://www.wikidata.org/entity/Q34062698

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Emerging Functions of Natural IgM and Its Fc Receptor FCMR in Immune Homeostasis Effects of complement activation on allograft injury Natural IgM anti-leukocyte autoantibodies attenuate excess inflammation mediated by innate and adaptive immune mechanisms involving Th-17 B lymphocytes trigger monocyte mobilization and impair heart function after acute myocardial infarction.Activation of complement factor B contributes to murine and human myocardial ischemia/reperfusion injury.Membrane lipid interactions in intestinal ischemia/reperfusion-induced Injury Oxidative stress sensitizes retinal pigmented epithelial (RPE) cells to complement-mediated injury in a natural antibody-, lectin pathway-, and phospholipid epitope-dependent manner.TLR2 modulates antibodies required for intestinal ischemia/reperfusion-induced damage and inflammation Targeting pathogenic postischemic self-recognition by natural IgM to protect against posttransplantation cardiac reperfusion injury Pathogenic natural antibodies propagate cerebral injury following ischemic stroke in mice.Acute Liver Injury Is Independent of B Cells or Immunoglobulin M.The Fifth Domain of Beta 2 Glycoprotein I Protects from Natural IgM Mediated Cardiac Ischaemia Reperfusion Injury IgM exacerbates glomerular disease progression in complement-induced glomerulopathy Small β2-glycoprotein I peptides protect from intestinal ischemia reperfusion injury.Natural IgM Blockade Limits Infarct Expansion and Left Ventricular Dysfunction in a Swine Myocardial Infarct Model Endothelial pentraxin 3 contributes to murine ischemic acute kidney injury.Humoral innate immune response and disease.IgM contributes to glomerular injury in FSGS Mouse mast cell proteases 4 and 5 mediate epidermal injury through disruption of tight junctions Role of complement and perspectives for intervention in ischemia-reperfusion damage.Nitrosative Stress, Hypernitrosylation, and Autoimmune Responses to Nitrosylated Proteins: New Pathways in Neuroprogressive Disorders Including Depression and Chronic Fatigue Syndrome.The Emerging Importance of Non-HLA Autoantibodies in Kidney Transplant Complications.Prevention of intestinal ischemia-reperfusion injury in humanized mice.Blockade of IgM-Mediated Inflammation Alters Wound Progression in a Swine Model of Partial-Thickness Burn.Natural IgM initiates an inflammatory response important for both hepatic ischemia reperfusion injury and regeneration.CD6 Receptor Regulates Intestinal Ischemia/Reperfusion-induced Injury by Modulating Natural IgM-producing B1a Cell Self-renewal.Complete B Cell Deficiency Reduces Allograft Inflammation and Intragraft Macrophages in a Rat Kidney Transplant Model.Lymphocytes at the Heart of Wound Healing.

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P2860

Blockade of self-reactive IgM significantly reduces injury in a murine model of acute myocardial infarction

http://www.wikidata.org/entity/Q34062698

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Blockade of self-reactive IgM ...... of acute myocardial infarction

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Blockade of self-reactive IgM ...... of acute myocardial infarction

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Blockade of self-reactive IgM ...... of acute myocardial infarction

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type

label

Blockade of self-reactive IgM ...... of acute myocardial infarction

@ast

Blockade of self-reactive IgM ...... of acute myocardial infarction

@en

Blockade of self-reactive IgM ...... of acute myocardial infarction

@nl

prefLabel

Blockade of self-reactive IgM ...... of acute myocardial infarction

@ast

Blockade of self-reactive IgM ...... of acute myocardial infarction

@en

Blockade of self-reactive IgM ...... of acute myocardial infarction

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P1433

Cardiovascular Research

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Blockade of self-reactive IgM ...... of acute myocardial infarction

@en

P2093

Elisabeth M Alicot

http://www.w3.org/2001/XMLSchema#string

Francis D Moore

http://www.w3.org/2001/XMLSchema#string

Franziska Schuerpf

http://www.w3.org/2001/XMLSchema#string

Jinan Li

http://www.w3.org/2001/XMLSchema#string

Michael C Carroll

http://www.w3.org/2001/XMLSchema#string

Michael S Haas

http://www.w3.org/2001/XMLSchema#string

P2860

Heart Disease and Stroke Statistics--2010 Update: A Report From the American Heart Association

Identification of splenic reservoir monocytes and their deployment to inflammatory sites

Myocardial reperfusion: a double-edged sword?

Pexelizumab in ischemic heart disease: a systematic review and meta-analysis on 15,196 patients

The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions

Human serum IgM glycosylation: identification of glycoforms that can bind to mannan-binding lectin.

Attenuation of skeletal muscle reperfusion injury with intravenous 12 amino acid peptides that bind to pathogenic IgM.

Targeting C-reactive protein for the treatment of cardiovascular disease.

Reactive species mechanisms of cellular hypoxia-reoxygenation injury.

Myocardial reperfusion injury.

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618-627

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scholarly article

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10.1093/CVR/CVQ141

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4

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87

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2010-05-11T00:00:00Z

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44592481

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20462867

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2920809

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