Skeletal actin mRNA increases in the human heart during ontogenic development and is the major isoform of control and failing adult hearts.
about
Nemaline myopathy caused by mutations in the muscle alpha-skeletal-actin gene.Alpha-cardiac actin mutations produce atrial septal defectsMyosin heavy chain gene expression in human heart failureSarcomeric protein isoform transitions in cardiac muscle: a journey to heart failureModelling sarcomeric cardiomyopathies in the dish: from human heart samples to iPSC cardiomyocytesRescue of cardiac alpha-actin-deficient mice by enteric smooth muscle gamma-actinCellular disorganization and extensive apoptosis in the developing heart of mice that lack cardiac muscle alpha-actin: apparent cause of perinatal deathAnnexin expressions are temporally and spatially regulated during rat hepatocyte differentiationCloning and characterization of a novel gene, striamin, that interacts with the tumor suppressor protein p53Heart Failure in Pediatric Patients With Congenital Heart Disease.Alpha-cardiac actin is a novel disease gene in familial hypertrophic cardiomyopathyMyofilament calcium regulation in human myocardiumThe vascular smooth muscle alpha-actin gene is reactivated during cardiac hypertrophy provoked by load.Pathological situations characterized by altered actin isoform expression.Molecular defects in cardiac myofibrillar proteins due to thyroid hormone imbalance and diabetes.Mice expressing mutant myosin heavy chains are a model for familial hypertrophic cardiomyopathyMyofibrillar remodeling in cardiac hypertrophy, heart failure and cardiomyopathies.A gain-of-function ACTC1 3'UTR mutation that introduces a miR-139-5p target site may be associated with a dominant familial atrial septal defect.Effective top-down LC/MS+ method for assessing actin isoforms as a potential cardiac disease markerRe-expression of alpha skeletal actin as a marker for dedifferentiation in cardiac pathologies.EST-based identification of genes expressed in skeletal muscle of the mandarin fish (Siniperca chuatsi).Cardiogenic differentiation and transdifferentiation of progenitor cellsβ-myosin heavy chain is induced by pressure overload in a minor subpopulation of smaller mouse cardiac myocytes.Harnessing fetal and adult genetic reprograming for therapy of heart disease.The W-loop of alpha-cardiac actin is critical for heart function and endocardial cushion morphogenesis in zebrafish.Myocardial phenotype changes in heart failure: cellular and subcellular adaptations and their functional significance.
P2860
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P2860
Skeletal actin mRNA increases in the human heart during ontogenic development and is the major isoform of control and failing adult hearts.
description
1991 nî lūn-bûn
@nan
1991 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1991 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
name
Skeletal actin mRNA increases ...... trol and failing adult hearts.
@ast
Skeletal actin mRNA increases ...... trol and failing adult hearts.
@en
Skeletal actin mRNA increases ...... trol and failing adult hearts.
@nl
type
label
Skeletal actin mRNA increases ...... trol and failing adult hearts.
@ast
Skeletal actin mRNA increases ...... trol and failing adult hearts.
@en
Skeletal actin mRNA increases ...... trol and failing adult hearts.
@nl
prefLabel
Skeletal actin mRNA increases ...... trol and failing adult hearts.
@ast
Skeletal actin mRNA increases ...... trol and failing adult hearts.
@en
Skeletal actin mRNA increases ...... trol and failing adult hearts.
@nl
P2093
P2860
P356
P1476
Skeletal actin mRNA increases ...... trol and failing adult hearts.
@en
P2093
Boheler KR
Mercadier JJ
Schwartz K
de la Bastie D
P2860
P304
P356
10.1172/JCI115295
P407
P577
1991-07-01T00:00:00Z