Matrix metalloprotease 9 mediates neutrophil migration into the airways in response to influenza virus-induced toll-like receptor signaling. - Wikidata - thesis-toulmin - TriplyDB

Matrix metalloprotease 9 mediates neutrophil migration into the airways in response to influenza virus-induced toll-like receptor signaling.

Matrix metalloprotease 9 mediates neutrophil migration into the airways in response to influenza virus-induced toll-like receptor signaling.

http://www.wikidata.org/entity/Q34229687

about

The Role of the Immune Response in Chlamydia trachomatis Infection of the Male Genital Tract: A Double-Edged Sword Neutrophil trails guide influenza-specific CD8⁺ T cells in the airways.Plasminogen controls inflammation and pathogenesis of influenza virus infections via fibrinolysis ADAMTS5 Is a Critical Regulator of Virus-Specific T Cell Immunity A Role for Neutrophils in Viral Respiratory Disease.Heterogeneous pathological outcomes after experimental pH1N1 influenza infection in ferrets correlate with viral replication and host immune responses in the lung.Dengue virus infection increases microglial cell migration.Biomarkers of lung epithelial injury and inflammation distinguish severe sepsis patients with acute respiratory distress syndrome.Differential protein expression in chicken macrophages and heterophils in vivo following infection with Salmonella Enteritidis.Early local immune defences in the respiratory tract Neutrophils are needed for an effective immune response against pulmonary rat coronavirus infection, but also contribute to pathology.Effect of ozone exposure and infection on bronchoalveolar lavage: sex differences in response patterns Hematopoietic but not endothelial cell MyD88 contributes to host defense during gram-negative pneumonia derived sepsis.Patient-based transcriptome-wide analysis identify interferon and ubiquination pathways as potential predictors of influenza A disease severity.Identification of small molecules that interfere with H1N1 influenza A viral replication.P. gingivalis lipopolysaccharide intensifies inflammation post-myocardial infarction through matrix metalloproteinase-9 Type-I interferons induce lung protease responses following respiratory syncytial virus infection via RIG-I-like receptors.Differential host response, rather than early viral replication efficiency, correlates with pathogenicity caused by influenza viruses.Therapy with plasma purified alpha1-antitrypsin (Prolastin®) induces time-dependent changes in plasma levels of MMP-9 and MPO.Inflammatory impact of IFN-γ in CD8+ T cell-mediated lung injury is mediated by both Stat1-dependent and -independent pathways.Mast cells play an important role in chlamydia pneumoniae lung infection by facilitating immune cell recruitment into the airway Dysregulated mechanisms underlying Duchenne muscular dystrophy from co-expression network preservation analysis Matrix Metalloproteinase 9 Exerts Antiviral Activity against Respiratory Syncytial Virus Testing multiple hypotheses through IMP weighted FDR based on a genetic functional network with application to a new zebrafish transcriptome study Glioma-associated microglial MMP9 expression is upregulated by TLR2 signaling and sensitive to minocycline.Building a better infarct: Modulation of collagen cross-linking to increase infarct stiffness and reduce left ventricular dilation post-myocardial infarction.The Influenza Virus Protein PB1-F2 Increases Viral Pathogenesis through Neutrophil Recruitment and NK Cells Inhibition.Involvement of the different lung compartments in the pathogenesis of pH1N1 influenza virus infection in ferrets.Biomechanics of Cardiac Function Metalloproteinases: a Functional Pathway for Myeloid Cells.Depletion of alveolar macrophages during influenza infection facilitates bacterial superinfections Circulating Porphyromonas gingivalis lipopolysaccharide resets cardiac homeostasis in mice through a matrix metalloproteinase-9-dependent mechanism.Adjuvant-induced Human Monocyte Secretome Profiles Reveal Adjuvant- and Age-specific Protein Signatures.Inhibition of NF-κB-mediated inflammation in severe acute respiratory syndrome coronavirus-infected mice increases survival Differential neutrophil activation in viral infections: Enhanced TLR-7/8-mediated CXCL8 release in asthma.Genital Chlamydia trachomatis: understanding the roles of innate and adaptive immunity in vaccine research.Chlamydia muridarum infection-induced destruction of male germ cells and sertoli cells is partially prevented by Chlamydia major outer membrane protein-specific immune CD4 cells.In the eye of the neutrophil swarm-navigation signals that bring neutrophils together in inflamed and infected tissues.Cannabinoid Receptor 2 Modulates Neutrophil Recruitment in a Murine Model of Endotoxemia.Immunohistochemical expression of TGF-β1 and MMP-9 in periapical lesions.

P2860

Q26851537-10A05BCF-5683-4C44-8F8E-54DF61D293A7 Q27331481-3B31D299-3A01-4273-8DC2-EF9CD35E2B19 Q27335814-D1D37EF7-23E9-41BD-8EA3-440C2B99E4FC Q28553566-4E76FB9C-3E58-459F-B841-DF014F3279E6 Q30234336-043EB9F0-8A34-44C2-AFD8-81FB6DD4ABCE Q30366138-B490BFAC-A28F-4ED8-94C2-A0665F0C684E Q33668917-99AD7DE8-309F-4FC8-BF8A-8A1C4ED35657 Q33750995-648B090A-C732-46E5-BD0E-789B27F2DFF9 Q33808364-0434616C-D39B-4215-B5F1-441FFFC9404B Q33824093-C0D59896-CB25-4514-B783-02F3D42260C8 Q33887347-2DD03BC8-86DE-4F5C-9D8A-1DA0AC9457E6 Q34217008-91A68880-D5AC-4AAC-9BB4-3923B991898E Q34257461-8368A7E2-C9E4-4705-8EE4-C2B853DB7129 Q34445640-9E417FE5-EF07-4F64-9270-023C9A3B4098 Q34471507-043B2C51-63FC-4873-B848-4476B0B679E4 Q34603609-AEC70ED3-836D-4EC3-B799-3701B3D71128 Q34716819-636D5258-F33F-4E8A-8045-0010D9D4802E Q35000731-557C275C-EA7B-4A0F-AFD2-38F881AAE80C Q35031693-9B5FA58F-AD98-4356-B057-534539A6DB5B Q35275348-CB50AF36-9ACB-4803-8E08-6AC18B723318 Q35329165-06924377-E2EE-4666-A40C-5C2E3FA0A057 Q35583817-AD448B97-EF41-471E-9F84-7C0A12D01F98 Q35748831-00AE85F7-4C30-4524-A3AF-44C351A2828A Q35761262-2D920451-5F5F-42B1-A1D2-8A35DE49BBD1 Q35901927-CC30AE4B-8BF2-4B5F-B81C-0A2FFDDFA423 Q35931621-0A5F97A6-F025-4C9A-90E4-231801F9C8B2 Q36178669-E219354E-AC93-4F6D-B780-39DE96B2BFC0 Q36186133-92EBF53E-C273-4EE0-B3C5-26D0CD6A2DA2 Q36343041-29C8A3F4-FAEB-4476-89A8-122CD83C8AA3 Q36955864-C7D4ABB8-0B09-4C04-B680-4E1B200085F9 Q37002972-334CEDBB-A756-4F25-BFFF-A7E570242196 Q37244775-9A92B53E-41D3-4C18-9A67-4BBA090749A4 Q37372321-21D9B6D2-99E5-4752-9472-9883A3746F12 Q37547432-7D102DF8-EECA-43D8-8C2E-1040F13DEDFA Q37663881-1E8644C4-85E0-45EB-A524-5420E5CFE2B4 Q37712799-93D9D0A1-579B-43A8-BD72-FEE0EE45FE8E Q37842770-1DFC87D0-EB80-4F46-A647-62D6DAA64A90 Q38594922-5D50DC69-4702-431B-B11C-F4F0566E34E6 Q38602925-70327C2A-428A-4FA6-9121-C4EF5E6A650B Q38692749-7597B5BE-56B7-4490-AC9D-99F8ED5FA5D7

P2860

Matrix metalloprotease 9 mediates neutrophil migration into the airways in response to influenza virus-induced toll-like receptor signaling.

http://www.wikidata.org/entity/Q34229687

description

2012 nî lūn-bûn

@nan

2012 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2012年の論文

@ja

2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

name

Matrix metalloprotease 9 media ...... toll-like receptor signaling.

@ast

Matrix metalloprotease 9 media ...... toll-like receptor signaling.

@en

Matrix metalloprotease 9 media ...... toll-like receptor signaling.

@nl

type

label

Matrix metalloprotease 9 media ...... toll-like receptor signaling.

@ast

Matrix metalloprotease 9 media ...... toll-like receptor signaling.

@en

Matrix metalloprotease 9 media ...... toll-like receptor signaling.

@nl

prefLabel

Matrix metalloprotease 9 media ...... toll-like receptor signaling.

@ast

Matrix metalloprotease 9 media ...... toll-like receptor signaling.

@en

Matrix metalloprotease 9 media ...... toll-like receptor signaling.

@nl

P1433

Q34229687-673559A3-3A5F-4810-8BF4-6E0B637DB5EF

P1476

Q34229687-66672650-4930-4C60-B368-B67AB275894B

P2093

Q34229687-290BF5E0-A19E-43FC-8688-23DE0B8F1A18

Q34229687-433A95B3-F680-4AA0-8A75-EBF82646750A

Q34229687-9F07E37A-A3A2-4B31-AA48-3DEE0C64758B

Q34229687-BC6ACE55-93E0-4C5A-A4A4-1140FA6FEA37

Q34229687-CA65AE19-3E29-4739-AD09-54885A2C3B46

P2860

Q34229687-0DA9EB0D-62F1-4A90-9A98-95062594C135

Q34229687-17872708-0684-4DD5-992D-CCC28E1AB3CA

Q34229687-1ABF0EEA-D972-402F-B3F2-B32ACF00B556

Q34229687-22777737-F995-4F4B-951A-3A16FC78B0D3

Q34229687-29D495C7-3428-45E3-A1F2-56DC9E120768

Q34229687-347A8315-A0F3-48F7-ADCD-30004F21CD80

Q34229687-3B73884F-02BC-4190-B38C-37EBB58A6D27

Q34229687-475BFBC6-9606-4BF6-85DD-27A8DBC84EF0

Q34229687-478CE041-8FEB-44FC-9BD5-5DC2395CD320

Q34229687-65BD3242-B460-4662-B1A5-A07DA3AE3022

P304

Q34229687-093361A9-0F71-46B9-B332-762DFEC3DF09

P31

Q34229687-42AC65B2-7AA3-48D8-9F3F-1C94CECDE9C3

P356

Q34229687-DCE90E61-B297-451D-A902-AA00396373E2

P433

Q34229687-E8D26657-C747-4098-9089-A74D72C61034

P478

Q34229687-43FE10AE-9705-4ECA-B1FA-A1D852D3A6A9

P577

Q34229687-D2CFC022-D96A-4DEE-BCB5-4EC2450DFE26

P5875

Q34229687-9FC83786-BB12-4D7F-BDA6-A8D88BFDE2EF

P698

Q34229687-ECA39660-0498-445E-883B-98F23EA86E1F

P921

Q34229687-94E5F5A9-B7EC-4249-BCCF-CABF05248D7B

P932

Q34229687-9D971F91-EE4B-4DC5-BA3A-165D855C9A58

P356

JOURNAL.PPAT.1002641

P1433

P1476

Matrix metalloprotease 9 media ...... toll-like receptor signaling.

@en

P2093

Bas J G Baaten

http://www.w3.org/2001/XMLSchema#string

Dhrubamitra Chatterjee

http://www.w3.org/2001/XMLSchema#string

Linda M Bradley

http://www.w3.org/2001/XMLSchema#string

Mia F Douglass

http://www.w3.org/2001/XMLSchema#string

Shizuo Akira

http://www.w3.org/2001/XMLSchema#string

P2860

MMP-9/gelatinase B is a key regulator of growth plate angiogenesis and apoptosis of hypertrophic chondrocytes

Matrix metalloproteinases in lung biology

Toll-like receptor-mediated activation of neutrophils by influenza A virus

Fatal outcome of human influenza A (H5N1) is associated with high viral load and hypercytokinemia

Innate antiviral responses by means of TLR7-mediated recognition of single-stranded RNA

Mortality associated with influenza and respiratory syncytial virus in the United States

Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function

Pathogenicity of influenza viruses with genes from the 1918 pandemic virus: functional roles of alveolar macrophages and neutrophils in limiting virus replication and mortality in mice

A question of self-preservation: immunopathology in influenza virus infection.

H5N1 and 1918 pandemic influenza virus infection results in early and excessive infiltration of macrophages and neutrophils in the lungs of mice.

P304

e1002641

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1371/JOURNAL.PPAT.1002641

http://www.w3.org/2001/XMLSchema#string

P433

4

http://www.w3.org/2001/XMLSchema#string

P478

8

http://www.w3.org/2001/XMLSchema#string

P577

2012-04-05T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

223977920

http://www.w3.org/2001/XMLSchema#string

P698

22496659

http://www.w3.org/2001/XMLSchema#string

P921

Toll-like receptor

P932

3320598

http://www.w3.org/2001/XMLSchema#string