Theoretical models for cooperative steady-state ATPase activity of myosin subfragment-1 on regulated actin.
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Effect of Ca2+ on cross-bridge turnover kinetics in skinned single rabbit psoas fibers: implications for regulation of muscle contraction.Kinetics of regulated actin transitions measured by probes on tropomyosin.Modulation of cross-bridge affinity for MgGTP by Ca2+ in skinned fibers of rabbit psoas muscle.Kinetics of thin filament activation probed by fluorescence of N-((2-(iodoacetoxy)ethyl)-N-methyl)amino-7-nitrobenz-2-oxa-1,3-diazole-labeled troponin I incorporated into skinned fibers of rabbit psoas muscle: implications for regulation of muscle cTroponin-tropomyosin: an allosteric switch or a steric blocker?Thin filament activation probed by fluorescence of N-((2-(iodoacetoxy)ethyl)-N-methyl)amino-7-nitrobenz-2-oxa-1,3-diazole-labeled troponin I incorporated into skinned fibers of rabbit psoas muscle.Ca2+ and ionic strength dependencies of S1-ADP binding to actin-tropomyosin-troponin: regulatory implications.Effects of the state of the succinimido-ring on the fluorescence and structural properties of pyrene maleimide-labeled alpha alpha-tropomyosin.Two elementary models for the regulation of skeletal muscle contraction by calciumThe spatial distribution of actin and mechanical cycle of myosin are different in right and left ventricles of healthy mouse heartsAcrylodan-labeled smooth muscle tropomyosin reports differences in the effects of troponin and caldesmon in the transition from the active state to the inactive stateKinetic diagram and free energy diagram for kinesin in microtubule-related motility.The Hill model for binding myosin S1 to regulated actin is not equivalent to the McKillop-Geeves modelThe C-terminus of troponin T is essential for maintaining the inactive state of regulated actinNegative charges at protein kinase C sites of troponin I stabilize the inactive state of actinA Novel Method of Determining the Functional Effects of a Minor Genetic Modification of a Protein.Some cardiomyopathy-causing troponin I mutations stabilize a functional intermediate actin state.Cooperative properties of cytochromes P450.Disease causing mutations of troponin alter regulated actin state distributions.Caldesmon and thin-filament regulation of muscle contraction.A theoretical analysis of binding to the Ca2+-specific sites on troponin incorporated into thin filamentsThe kinetics relating calcium and force in skeletal muscle.The Delta 14 mutation of human cardiac troponin T enhances ATPase activity and alters the cooperative binding of S1-ADP to regulated actin.Effect of caltropin on caldesmon-actin interaction.Caldesmon: binding to actin and myosin and effects on elementary steps in the ATPase cycle.
P2860
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P2860
Theoretical models for cooperative steady-state ATPase activity of myosin subfragment-1 on regulated actin.
description
1981 nî lūn-bûn
@nan
1981 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1981 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1981年の論文
@ja
1981年論文
@yue
1981年論文
@zh-hant
1981年論文
@zh-hk
1981年論文
@zh-mo
1981年論文
@zh-tw
1981年论文
@wuu
name
Theoretical models for coopera ...... fragment-1 on regulated actin.
@ast
Theoretical models for coopera ...... fragment-1 on regulated actin.
@en
Theoretical models for coopera ...... fragment-1 on regulated actin.
@nl
type
label
Theoretical models for coopera ...... fragment-1 on regulated actin.
@ast
Theoretical models for coopera ...... fragment-1 on regulated actin.
@en
Theoretical models for coopera ...... fragment-1 on regulated actin.
@nl
prefLabel
Theoretical models for coopera ...... fragment-1 on regulated actin.
@ast
Theoretical models for coopera ...... fragment-1 on regulated actin.
@en
Theoretical models for coopera ...... fragment-1 on regulated actin.
@nl
P2093
P2860
P1433
P1476
Theoretical models for coopera ...... fragment-1 on regulated actin.
@en
P2093
Chalovich JM
Eisenberg E
P2860
P304
P356
10.1016/S0006-3495(81)84777-7
P407
P577
1981-07-01T00:00:00Z