Filaggrin loss-of-function mutations are associated with food allergy in childhood and adolescence - Wikidata - thesis-toulmin - TriplyDB

Filaggrin loss-of-function mutations are associated with food allergy in childhood and adolescence

Filaggrin loss-of-function mutations are associated with food allergy in childhood and adolescence

http://www.wikidata.org/entity/Q34294641

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The role of dietary interventions in the prevention of IgE-mediated food allergy in children.Intradermal administration of IL-33 induces allergic airway inflammation Genome-wide association study identifies peanut allergy-specific loci and evidence of epigenetic mediation in US children The role of gene-environment interactions in the development of food allergy.Molecular and cellular mechanisms of food allergy and food tolerance.The Role of Skin Barrier in the Pathogenesis of Food Allergy Food allergy: immune mechanisms, diagnosis and immunotherapy Recent advances in epidemiology and prevention of atopic eczema.The future of biologics: applications for food allergy.Unravelling the complex genetic background of atopic dermatitis: from genetic association results towards novel therapeutic strategies.Extended implementation of educational programs for atopic dermatitis in childhood.Epicutaneous immunotherapy for food allergy as a novel pathway for oral tolerance induction.Prevalence of Immediate-Type Food Allergy in Korean Schoolchildren in 2015: A Nationwide, Population-based Study.The atopic march: current insights into skin barrier dysfunction and epithelial cell-derived cytokines.IL-33 promotes gastrointestianl allergy in a TSLP-independent manner.Are genetic tests informative in predicting food allergy?The skin barrier function gene SPINK5 is associated with challenge-proven IgE-mediated food allergy in infants.The barrier hypothesis and Oncostatin M: Restoration of epithelial barrier function as a novel therapeutic strategy for the treatment of type 2 inflammatory disease.Genome-wide association study identifies the SERPINB gene cluster as a susceptibility locus for food allergy.Oral Tolerance Development and Maintenance.Transplant-acquired food allergy: current perspectives.Association of FLG single nucleotide variations with clinical phenotypes of atopic dermatitis.Filaggrin mutations increase allergic airway disease in childhood and adolescence through interactions with eczema and aeroallergen sensitization.The Implications of DNA Methylation on Food Allergy.Component-resolved diagnosis and beyond: Multivariable regression models to predict severity of hazelnut allergy.The role of environmental exposure to peanut in the development of clinical allergy to peanut.Identification and analysis of peanut-specific T effector and T regulatory cells in children allergic and tolerant to peanut.Prevalence and longitudinal trends of food allergy during childhood and adolescence: Results of the Isle of Wight Birth Cohort study.Genomewide association study of peanut allergy reproduces association with amino acid polymorphisms in HLA-DRB1.Oral immunotherapy for food allergy.Cohort Profile: The Isle Of Wight Whole Population Birth Cohort (IOWBC)Food allergy

P2860

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P2860

Filaggrin loss-of-function mutations are associated with food allergy in childhood and adolescence

http://www.wikidata.org/entity/Q34294641

description

2014 nî lūn-bûn

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2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2014 թվականի օգոստոսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Filaggrin loss-of-function mut ...... y in childhood and adolescence

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Filaggrin loss-of-function mut ...... y in childhood and adolescence

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Filaggrin loss-of-function mut ...... y in childhood and adolescence

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Filaggrin loss-of-function mut ...... y in childhood and adolescence

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Filaggrin loss-of-function mut ...... y in childhood and adolescence

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Filaggrin loss-of-function mut ...... y in childhood and adolescence

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Filaggrin loss-of-function mut ...... y in childhood and adolescence

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Filaggrin loss-of-function mut ...... y in childhood and adolescence

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Filaggrin loss-of-function mut ...... y in childhood and adolescence

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J.JACI.2014.07.033

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The Journal of Allergy and Clinical Immunology

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Filaggrin loss-of-function mut ...... y in childhood and adolescence

@en

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Devasmitha Venkataraman

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Mich Erlewyn-Lajeunesse

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Nelís Soto-Ramírez

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Ramesh J Kurukulaaratchy

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S Hasan Arshad

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Susan L Ewart

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P2860

Gene-environment interaction in the onset of eczema in infancy: filaggrin loss-of-function mutations enhanced by neonatal cat exposure

Factors associated with the development of peanut allergy in childhood

Common loss-of-function variants of the epidermal barrier protein filaggrin are a major predisposing factor for atopic dermatitis

Abnormal skin barrier in the etiopathogenesis of atopic dermatitis.

A homozygous frameshift mutation in the mouse Flg gene facilitates enhanced percutaneous allergen priming.

Filaggrin-deficient mice exhibit TH17-dominated skin inflammation and permissiveness to epicutaneous sensitization with protein antigen

Interplay of filaggrin loss-of-function variants, allergic sensitization, and eczema in a longitudinal study covering infancy to 18 years of age.

Loss-of-function mutations in the gene encoding filaggrin cause ichthyosis vulgaris.

Moisturization and skin barrier function.

Epidemiologic risks for food allergy.

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John W. Holloway

Wilfried Karmaus

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2014-08-28T00:00:00Z

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