Epithelial cell-derived S100 calcium-binding proteins as key mediators in the hallmark acute neutrophil response during Candida vaginitis.
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The Host's Reply to Candida BiofilmCytokines in the host response to Candida vaginitis: Identifying a role for non-classical immune mediators, S100 alarminsExperimental Models of Vaginal Candidiasis and Their Relevance to Human CandidiasisIL-22 and IDO1 affect immunity and tolerance to murine and human vaginal candidiasisCandida vaginitis: when opportunism knocks, the host respondsDectin-1 isoforms contribute to distinct Th1/Th17 cell activation in mucosal candidiasis.Characterization of IL-22 and antimicrobial peptide production in mice protected against pulmonary Cryptococcus neoformans infection.Th17 cells and IL-17 in protective immunity to vaginal candidiasis.Candida albicans yeast and hyphae are discriminated by MAPK signaling in vaginal epithelial cells.The acute neutrophil response mediated by S100 alarmins during vaginal Candida infections is independent of the Th17-pathway.Prospects for development of a vaccine to prevent and control vaginal candidiasis.Candida albicans adhesion to and invasion and damage of vaginal epithelial cells: stage-specific inhibition by clotrimazole and bifonazole.Proteomics and pathway analyses of the milk fat globule in sheep naturally infected by Mycoplasma agalactiae provide indications of the in vivo response of the mammary epithelium to bacterial infection.Transcriptomic analysis of vulvovaginal candidiasis identifies a role for the NLRP3 inflammasome.A Murine Model of Candida glabrata Vaginitis Shows No Evidence of an Inflammatory Immunopathogenic Response.A lack of association between elevated serum levels of S100B protein and autoimmunity in autistic children.Protocols for vaginal inoculation and sample collection in the experimental mouse model of Candida vaginitisCandida albicans Pathogenesis: Fitting within the Host-Microbe Damage Response Framework.Adaptive immune responses to Candida albicans infection.Candida albicans-epithelial interactions and pathogenicity mechanisms: scratching the surface.The role of pattern recognition receptors in the innate recognition of Candida albicans.New approaches in the development of a vaccine for mucosal candidiasis: progress and challengesHost contributions to construction of three device-associated Candida albicans biofilmsPerforming vaginal lavage, crystal violet staining, and vaginal cytological evaluation for mouse estrous cycle staging identification.Urinary bladder hypersensitivity and dysfunction in female mice following early life and adult stressGenome-wide transcriptional analysis of differentially expressed genes in flagellin-pretreated mouse corneal epithelial cells in response to Pseudomonas aeruginosa: involvement of S100A8/A9.Clotrimazole dampens vaginal inflammation and neutrophil infiltration in response to Candida albicans infection.In vivo induction of neutrophil chemotaxis by secretory aspartyl proteinases of Candida albicans.Secretory aspartyl proteinases induce neutrophil chemotaxis in vivoVaginal epithelial cell-derived S100 alarmins induced by Candida albicans via pattern recognition receptor interactions are sufficient but not necessary for the acute neutrophil response during experimental vaginal candidiasis.Fungal morphogenetic pathways are required for the hallmark inflammatory response during Candida albicans vaginitis.Streptococcal co-infection augments Candida pathogenicity by amplifying the mucosal inflammatory response.Vaginal Heparan Sulfate Linked to Neutrophil Dysfunction in the Acute Inflammatory Response Associated with Experimental Vulvovaginal Candidiasis.IL-22 in antifungal immunity.Candida albicans morphogenesis and host defence: discriminating invasion from colonization.Interplay between Candida albicans and the mammalian innate host defense.Genetic basis for recurrent vulvo-vaginal candidiasis.Beyond Candida albicans: Mechanisms of immunity to non-albicans Candida speciesTh17 cells confer long-term adaptive immunity to oral mucosal Candida albicans infections.Recurrent Vulvovaginal Candidiasis: Could It Be Related to Cell-Mediated Immunity Defect in Response to Candida Antigen?
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P2860
Epithelial cell-derived S100 calcium-binding proteins as key mediators in the hallmark acute neutrophil response during Candida vaginitis.
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2010 nî lūn-bûn
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2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված
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2010年の論文
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年學術文章
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name
Epithelial cell-derived S100 c ...... onse during Candida vaginitis.
@ast
Epithelial cell-derived S100 c ...... onse during Candida vaginitis.
@en
Epithelial cell-derived S100 c ...... onse during Candida vaginitis.
@nl
type
label
Epithelial cell-derived S100 c ...... onse during Candida vaginitis.
@ast
Epithelial cell-derived S100 c ...... onse during Candida vaginitis.
@en
Epithelial cell-derived S100 c ...... onse during Candida vaginitis.
@nl
prefLabel
Epithelial cell-derived S100 c ...... onse during Candida vaginitis.
@ast
Epithelial cell-derived S100 c ...... onse during Candida vaginitis.
@en
Epithelial cell-derived S100 c ...... onse during Candida vaginitis.
@nl
P2093
P2860
P356
P1476
Epithelial cell-derived S100 c ...... onse during Candida vaginitis.
@en
P2093
Elizabeth Lilly
Junko Yano
Melissa Barousse
Paul L Fidel
P2860
P304
P356
10.1128/IAI.00388-10
P407
P577
2010-09-07T00:00:00Z