Bioavailability of VEGF in tumor-shed vesicles depends on vesicle burst induced by acidic pH. - Wikidata - thesis-toulmin - TriplyDB

Bioavailability of VEGF in tumor-shed vesicles depends on vesicle burst induced by acidic pH.

Bioavailability of VEGF in tumor-shed vesicles depends on vesicle burst induced by acidic pH.

http://www.wikidata.org/entity/Q35052151

about

Role of exosomes/microvesicles in the nervous system and use in emerging therapies Glioblastoma microvesicles transport RNA and proteins that promote tumour growth and provide diagnostic biomarkers Microvesicles: mediators of extracellular communication during cancer progression Extracellular Vesicles in Physiology, Pathology, and Therapy of the Immune and Central Nervous System, with Focus on Extracellular Vesicles Derived from Mesenchymal Stem Cells as Therapeutic Tools Microvesicles as a potential biomarker of neoplastic diseases and their role in development and progression of neoplasm Exosomes: a novel pathway of local and distant intercellular communication that facilitates the growth and metastasis of neoplastic lesions Tumor-derived microvesicles: shedding light on novel microenvironment modulators and prospective cancer biomarkers Rab27 GTPases distribute extracellular nanomaps for invasive growth and metastasis: implications for prognosis and treatment The new deal: a potential role for secreted vesicles in innate immunity and tumor progression Blebbishields, the emergency program for cancer stem cells: sphere formation and tumorigenesis after apoptosis.Neoplasia: Where We Have Been and Where We Are Going A hybrid model for three-dimensional simulations of sprouting angiogenesis.Neoplasia: the second decade.A class of extracellular vesicles from breast cancer cells activates VEGF receptors and tumour angiogenesis.Gelatinases, endonuclease and Vascular Endothelial Growth Factor during development and regression of swine luteal tissue.Interactions of human monocytes with TMVs (tumour-derived microvesicles).Deciphering the role of ectosomes in cancer development and progression: focus on the proteome.Extracellular Membrane Vesicles Derived from 143B Osteosarcoma Cells Contain Pro-Osteoclastogenic Cargo: A Novel Communication Mechanism in Osteosarcoma Bone Microenvironment.Microparticles mediated cross-talk between tumoral and endothelial cells promote the constitution of a pro-metastatic vascular niche through Arf6 up regulation.ARF6-regulated shedding of tumor cell-derived plasma membrane microvesicles Live-cell imaging of tumor proteolysis: impact of cellular and non-cellular microenvironment Role of exosomes released by chronic myelogenous leukemia cells in angiogenesis.Biological properties of extracellular vesicles and their physiological functions.Tumor vesicle-associated CD147 modulates the angiogenic capability of endothelial cells.Exosomes serve as tumour markers for personalized diagnostics owing to their important role in cancer metastasis.Paracrine induction of endothelium by tumor exosomes.RNA interference-mediated targeting of urokinase plasminogen activator receptor and matrix metalloproteinase-9 gene expression in the IOMM-lee malignant meningioma cell line inhibits tumor growth, tumor cell invasion and angiogenesis.Matrix metalloproteinase-2-mediated occludin degradation and caveolin-1-mediated claudin-5 redistribution contribute to blood-brain barrier damage in early ischemic stroke stage.Release of Matrix Metalloproteinases-2 and 9 by S-Nitrosylated Caveolin-1 Contributes to Degradation of Extracellular Matrix in tPA-Treated Hypoxic Endothelial Cells.Oncogenic extracellular vesicles in brain tumor progression Microfluidic isolation of cancer-cell-derived microvesicles from hetergeneous extracellular shed vesicle populations.Minireview: Emerging Roles for Extracellular Vesicles in Diabetes and Related Metabolic Disorders.High Levels of EBV-Encoded RNA 1 (EBER1) Trigger Interferon and Inflammation-Related Genes in Keratinocytes Expressing HPV16 E6/E7 Sorting it out: regulation of exosome loading Inhibition of cancer cell growth by ruthenium complexes Microvesicles as potential ovarian cancer biomarkers.Cathepsin B mediates the pH-dependent proinvasive activity of tumor-shed microvesicles.The human ovarian cancer cell line CABA I: A peculiar genetic evolution Emerging roles of extracellular vesicles in the adaptive response of tumour cells to microenvironmental stress.Silencing of atp6v1c1 prevents breast cancer growth and bone metastasis.

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P2860

Bioavailability of VEGF in tumor-shed vesicles depends on vesicle burst induced by acidic pH.

http://www.wikidata.org/entity/Q35052151

description

2006 nî lūn-bûn

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2006 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի փետրվարին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Bioavailability of VEGF in tum ...... le burst induced by acidic pH.

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Bioavailability of VEGF in tum ...... le burst induced by acidic pH.

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type

label

Bioavailability of VEGF in tum ...... le burst induced by acidic pH.

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Bioavailability of VEGF in tum ...... le burst induced by acidic pH.

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prefLabel

Bioavailability of VEGF in tum ...... le burst induced by acidic pH.

@ast

Bioavailability of VEGF in tum ...... le burst induced by acidic pH.

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Bioavailability of VEGF in tum ...... le burst induced by acidic pH.

@en

P2093

Antonio Pavan

http://www.w3.org/2001/XMLSchema#string

Daniela Marchetti

http://www.w3.org/2001/XMLSchema#string

Danilo Millimaggi

http://www.w3.org/2001/XMLSchema#string

Ilaria Giusti

http://www.w3.org/2001/XMLSchema#string

Patrizia Borsotti

http://www.w3.org/2001/XMLSchema#string

Sandra D'Ascenzo

http://www.w3.org/2001/XMLSchema#string

P2860

The biology of VEGF and its receptors

Angiogenesis in cancer and other diseases

Why do cancers have high aerobic glycolysis?

Angiogenesis in cancer, vascular, rheumatoid and other disease

Regulation of angiogenesis by hypoxia: role of the HIF system

Tumorigenesis and the angiogenic switch

Role of the vascular endothelial growth factor pathway in tumor growth and angiogenesis

Dendritic cells or their exosomes are effective biotherapies of cancer.

Expulsion of small molecules in vesicles shed by cancer cells: association with gene expression and chemosensitivity profiles.

Rous-Whipple Award Lecture. How tumors make bad blood vessels and stroma.

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96-103

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scholarly article

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10.1593/NEO.05583

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2

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8

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Raffaella Giavazzi

Giulia Taraboletti

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2006-02-01T00:00:00Z

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7169577

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16611402

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1578512

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