MiR-196a promotes pancreatic cancer progression by targeting nuclear factor kappa-B-inhibitor alpha - Wikidata - thesis-toulmin - TriplyDB

MiR-196a promotes pancreatic cancer progression by targeting nuclear factor kappa-B-inhibitor alpha

MiR-196a promotes pancreatic cancer progression by targeting nuclear factor kappa-B-inhibitor alpha

http://www.wikidata.org/entity/Q35087835

about

miR-196, an Emerging Cancer Biomarker for Digestive Tract Cancers Managing Pancreatic Adenocarcinoma: A Special Focus in MicroRNA Gene Therapy Noncoding RNAs and pancreatic cancer MicroRNA as new tools for prostate cancer risk assessment and therapeutic intervention: results from clinical data set and patients' samples Upregulation of microRNA-196a and microRNA-196b cooperatively correlate with aggressive progression and unfavorable prognosis in patients with colorectal cancer Increased expression of microRNA-196a predicts poor prognosis in human ovarian carcinoma Small nucleolar RNA U91 is a new internal control for accurate microRNAs quantification in pancreatic cancer.MiRNA-891a-5p mediates HIV-1 Tat and KSHV Orf-K1 synergistic induction of angiogenesis by activating NF-κB signaling Mining for Candidate Genes Related to Pancreatic Cancer Using Protein-Protein Interactions and a Shortest Path Approach.High Expression of MicroRNA-196a Indicates Poor Prognosis in Resected Pancreatic Neuroendocrine Tumor.Role of miR-196 and its target gene HoxB8 in the development and proliferation of human colorectal cancer and the impact of neoadjuvant chemotherapy with FOLFOX4 on their expression.Loss of ZG16 is regulated by miR-196a and contributes to stemness and progression of colorectal cancer.MicroRNA-196a2 Biomarker and Targetome Network Analysis in Solid Tumors.miRNA profiling in gastrointestinal stromal tumors: implication as diagnostic and prognostic markers.MicroRNAs as biomarkers and prospective therapeutic targets in colon and pancreatic cancers.A comprehensive study of smoking-specific microRNA alterations in head and neck squamous cell carcinoma.Downregulation of microRNA-196a inhibits human liver cancer cell proliferation and invasion by targeting FOXO1.Improving accuracy of RNA-based diagnosis and prognosis of oral cancer by using noninvasive methods.MicroRNA-140 regulates cell growth and invasion in pancreatic duct adenocarcinoma by targeting iASPP.MicroRNA in pancreatic cancer.MicroRNA-196a overexpression promotes cell proliferation and inhibits cell apoptosis through PTEN/Akt/FOXO1 pathway.MicroRNA-196a promotes an oncogenic effect in head and neck cancer cells by suppressing annexin A1 and enhancing radioresistance.The role of microRNA-196a in tumorigenesis, tumor progression, and prognosis.Expression patterns of microRNA-218 and its potential functions by targeting CIP2A and BMI1 genes in melanoma.Epigenetics of epithelial-to-mesenchymal transition in pancreatic carcinoma.HIV-1 Vpr Inhibits Kaposi's Sarcoma-Associated Herpesvirus Lytic Replication by Inducing MicroRNA miR-942-5p and Activating NF-κB Signaling Donor miR-196a-2 polymorphism is associated with hepatocellular carcinoma recurrence after liver transplantation in a Han Chinese population.MiR-142 modulates human pancreatic cancer proliferation and invasion by targeting hypoxia-inducible factor 1 (HIF-1α) in the tumor microenvironments Plasma exosome miR-196a and miR-1246 are potential indicators of localized pancreatic cancer.Evaluation of miRNA-196a2 and apoptosis-related target genes: ANXA1, DFFA and PDCD4 expression in gastrointestinal cancer patients: A pilot study.Dysregulation of Mir-196b in Head and Neck Cancers Leads to Pleiotropic Effects in the Tumor Cells and Surrounding Stromal Fibroblasts.Application of non-invasive low strength pulsed electric field to EGCG treatment synergistically enhanced the inhibition effect on PANC-1 cells.MicroRNA-300 promotes apoptosis and inhibits proliferation, migration, invasion and epithelial-mesenchymal transition via the Wnt/β-catenin signaling pathway by targeting CUL4B in pancreatic cancer cells.Noncoding RNAs Regulating NF-κB Signaling.miR-196a Is Able to Restore the Aggressive Phenotype of Annexin A1 Knock-Out in Pancreatic Cancer Cells by CRISPR/Cas9 Genome Editing

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MiR-196a promotes pancreatic cancer progression by targeting nuclear factor kappa-B-inhibitor alpha

http://www.wikidata.org/entity/Q35087835

description

2014 nî lūn-bûn

@nan

2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

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2014年論文

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2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

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2014年论文

@wuu

name

MiR-196a promotes pancreatic c ...... factor kappa-B-inhibitor alpha

@ast

MiR-196a promotes pancreatic c ...... factor kappa-B-inhibitor alpha

@en

type

label

MiR-196a promotes pancreatic c ...... factor kappa-B-inhibitor alpha

@ast

MiR-196a promotes pancreatic c ...... factor kappa-B-inhibitor alpha

@en

prefLabel

MiR-196a promotes pancreatic c ...... factor kappa-B-inhibitor alpha

@ast

MiR-196a promotes pancreatic c ...... factor kappa-B-inhibitor alpha

@en

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MiR-196a promotes pancreatic c ...... factor kappa-B-inhibitor alpha

@en

P2093

Fengting Huang

http://www.w3.org/2001/XMLSchema#string

Herui Yao

http://www.w3.org/2001/XMLSchema#string

Jian Tang

http://www.w3.org/2001/XMLSchema#string

Shineng Zhang

http://www.w3.org/2001/XMLSchema#string

Wenbo Chen

http://www.w3.org/2001/XMLSchema#string

Wenjie Cheng

http://www.w3.org/2001/XMLSchema#string

Xiaohong Zhuang

http://www.w3.org/2001/XMLSchema#string

Yanyan Zhuang

http://www.w3.org/2001/XMLSchema#string

P2860

IkappaBalpha and IkappaBalpha /NF-kappa B complexes are retained in the cytoplasm through interaction with a novel partner, RasGAP SH3-binding protein 2

Distant metastasis occurs late during the genetic evolution of pancreatic cancer

I kappaB alpha physically interacts with a cytoskeleton-associated protein through its signal response domain

MicroRNAs in stress signaling and human disease

Epithelial-mesenchymal transitions in development and disease

Cancer statistics, 2013

MicroRNA expression patterns to differentiate pancreatic adenocarcinoma from normal pancreas and chronic pancreatitis

Norcantharidin facilitates LPS-mediated immune responses by up-regulation of AKT/NF-κB signaling in macrophages

SIRT1 activators suppress inflammatory responses through promotion of p65 deacetylation and inhibition of NF-κB activity

Complex networks orchestrate epithelial-mesenchymal transitions

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