Proinflammatory protein CARD9 is essential for infiltration of monocytic fibroblast precursors and cardiac fibrosis caused by Angiotensin II infusion.
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CARD9 as a potential target in cardiovascular diseaseBioengineering of injectable encapsulated aggregates of pluripotent stem cells for therapy of myocardial infarctionExpression profiling of long noncoding RNAs and the dynamic changes of lncRNA-NR024118 and Cdkn1c in angiotensin II-treated cardiac fibroblastsZinc rescues obesity-induced cardiac hypertrophy via stimulating metallothionein to suppress oxidative stress-activated BCL10/CARD9/p38 MAPK pathway.Ischemia-reperfusion injury leads to distinct temporal cardiac remodeling in normal versus diabetic miceReciprocal interaction between macrophages and T cells stimulates IFN-γ and MCP-1 production in Ang II-induced cardiac inflammation and fibrosisMacrophage-stimulated cardiac fibroblast production of IL-6 is essential for TGF β/Smad activation and cardiac fibrosis induced by angiotensin IISenescent cardiac fibroblast is critical for cardiac fibrosis after myocardial infarction.The effect of a preparation of minerals, vitamins and trace elements on the cardiac gene expression pattern in male diabetic rats.Neuroprotective Effect of Scutellarin on Ischemic Cerebral Injury by Down-Regulating the Expression of Angiotensin-Converting Enzyme and AT1 ReceptorInterleukin-6/signal transducer and activator of transcription 3 (STAT3) pathway is essential for macrophage infiltration and myoblast proliferation during muscle regeneration.Monocyte and macrophage contributions to cardiac remodeling.CARD9 knockout ameliorates myocardial dysfunction associated with high fat diet-induced obesity.The risk of heart failure and cardiometabolic complications in obesity may be masked by an apparent healthy status of normal blood glucose.A CARD9 polymorphism is associated with decreased likelihood of persistent conjugated hyperbilirubinemia in intestinal failure.Angiotensin II upregulated the expression of microRNA-224 but not microRNA-21 in adult rat cardiac fibroblastsZinc Prevents the Development of Diabetic Cardiomyopathy in db/db Mice.CYP epoxygenase 2J2 prevents cardiac fibrosis by suppression of transmission of pro-inflammation from cardiomyocytes to macrophages.Angiotensin II induced differentially expressed microRNAs in adult rat cardiac fibroblasts.Contribution of CARD9-mediated signalling to wound healing in skin.Molecular and physiological roles of the adaptor protein CARD9 in immunity.Macrophage-derived exosomes induce inflammatory factors in endothelial cells under hypertensive conditions.The role of KCa3.1 channels in cardiac fibrosis induced by pressure overload in rats.Inhibition of Toll-like receptor 2 reduces cardiac fibrosis by attenuating macrophage-mediated inflammation.
P2860
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P2860
Proinflammatory protein CARD9 is essential for infiltration of monocytic fibroblast precursors and cardiac fibrosis caused by Angiotensin II infusion.
description
2011 nî lūn-bûn
@nan
2011 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մարտին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Proinflammatory protein CARD9 ...... ed by Angiotensin II infusion.
@ast
Proinflammatory protein CARD9 ...... ed by Angiotensin II infusion.
@en
type
label
Proinflammatory protein CARD9 ...... ed by Angiotensin II infusion.
@ast
Proinflammatory protein CARD9 ...... ed by Angiotensin II infusion.
@en
prefLabel
Proinflammatory protein CARD9 ...... ed by Angiotensin II infusion.
@ast
Proinflammatory protein CARD9 ...... ed by Angiotensin II infusion.
@en
P2093
P2860
P356
P1476
Proinflammatory protein CARD9 ...... ed by Angiotensin II infusion.
@en
P2093
Guanming Qi
Jiao Zheng
Jingyuan Ren
Jizhong Cheng
P2860
P304
P356
10.1038/AJH.2011.42
P577
2011-03-24T00:00:00Z