about
Aliphatic hydrocarbonsMicroRNA-155 suppresses activation-induced cytidine deaminase-mediated Myc-Igh translocationMutations, kataegis and translocations in B cells: understanding AID promiscuous activityPreclinical validation of interleukin 6 as a therapeutic target in multiple myelomaEffects of Gene Dose, Chromatin, and Network Topology on Expression in Drosophila melanogasterA novel mouse model for multiple myeloma (MOPC315.BM) that allows noninvasive spatiotemporal detection of osteolytic diseasePTIP promotes chromatin changes critical for immunoglobulin class switch recombinationMcl-1 is essential for the survival of plasma cells.Gene expression profiling reveals different pathways related to Abl and other genes that cooperate with c-Myc in a model of plasma cell neoplasia.A mutant collagen XIII alters intestinal expression of immune response genes and predisposes transgenic mice to develop B-cell lymphomas.(18)F-FDG-PET/CT imaging in an IL-6- and MYC-driven mouse model of human multiple myeloma affords objective evaluation of plasma cell tumor progression and therapeutic response to the proteasome inhibitor ixazomib.A library of gene expression signatures to illuminate normal and pathological lymphoid biology.Heterologous tissue culture expression signature predicts human breast cancer prognosis.Myc dynamically and preferentially relocates to a transcription factory occupied by IghNOTCH is part of the transcriptional network regulating cell growth and survival in mouse plasmacytomasAID produces DNA double-strand breaks in non-Ig genes and mature B cell lymphomas with reciprocal chromosome translocations.IL-6 and MYC collaborate in plasma cell tumor formation in mice.Glycogen Synthase Kinase-3 regulates multiple myeloma cell growth and bortezomib-induced cell deathPromiscuous MYC locus rearrangements hijack enhancers but mostly super-enhancers to dysregulate MYC expression in multiple myeloma.Plasmacytomagenesis in Eμ-v-abl transgenic mice is accelerated when apoptosis is restrained.Global gene expression profiling in mouse plasma cell tumor precursor and bystander cells reveals potential intervention targets for plasma cell neoplasia.Msh6 protects mature B cells from lymphoma by preserving genomic stability.Distinct granuloma responses in C57BL/6J and BALB/cByJ mice in response to pristane.Targeting the oncogene B lymphoma deregulator IgH 3' regulatory region does not impede the in vivo inflammatory response in mice.Anaplastic plasmacytoma of mouse--establishing parallels between subtypes of mouse and human plasma cell neoplasia.Translocation of an antibody transgene requires AID and occurs by interchromosomal switching to all switch regions except the mu switch region.Constitutive NF-kappaB activation confers interleukin 6 (IL6) independence and resistance to dexamethasone and Janus kinase inhibitor INCB018424 in murine plasmacytoma cells.Translocation-capture sequencing reveals the extent and nature of chromosomal rearrangements in B lymphocytesDifferential development of oil granulomas induced by pristane injection in galectin-3 deficient miceMouse model of endemic Burkitt translocations reveals the long-range boundaries of Ig-mediated oncogene deregulationcDNA-library testing identifies transforming genes cooperating with c-myc in mouse pre-B cells.Genomic instability and mouse microRNAs.Anaplastic plasmacytomas: relationships to normal memory B cells and plasma cell neoplasms of immunodeficient and autoimmune mice.Oncogenic Myc translocations are independent of chromosomal location and orientation of the immunoglobulin heavy chain locusAID expression levels determine the extent of cMyc oncogenic translocations and the incidence of B cell tumor development.53BP1 alters the landscape of DNA rearrangements and suppresses AID-induced B cell lymphoma.A der(8)t(8;11) chromosome in the Karpas-620 myeloma cell line expresses only cyclin D1: yet both cyclin D1 and MYC are repositioned in close proximity to the 3'IGH enhancerAID is required for the chromosomal breaks in c-myc that lead to c-myc/IgH translocationsCharacterization of MYC translocations in multiple myeloma cell lines.Effects of acute and chronic inflammation on B-cell development and differentiation.
P2860
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P2860
description
2003 nî lūn-bûn
@nan
2003 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Neoplastic development in plasma cells.
@ast
Neoplastic development in plasma cells.
@en
type
label
Neoplastic development in plasma cells.
@ast
Neoplastic development in plasma cells.
@en
prefLabel
Neoplastic development in plasma cells.
@ast
Neoplastic development in plasma cells.
@en
P2860
P1476
Neoplastic development in plasma cells.
@en
P2093
Michael Potter
P2860
P304
P356
10.1034/J.1600-065X.2003.00061.X
P577
2003-08-01T00:00:00Z