Redox modulation of FAK controls melanoma survival--role of NOX4.
about
Ubiquitin C-terminal hydrolase-L1 increases cancer cell invasion by modulating hydrogen peroxide generated via NADPH oxidase 4CRISPR-Cas9 Mediated NOX4 Knockout Inhibits Cell Proliferation and Invasion in HeLa CellsTargeting the Myofibroblastic Cancer-Associated Fibroblast Phenotype Through Inhibition of NOX4.Microradiopharmaceutical for Metastatic Melanoma.Reactive oxygen species generation mediated by NADPH oxidase and PI3K/Akt pathways contribute to invasion of Streptococcus agalactiae in human endothelial cells.
P2860
Redox modulation of FAK controls melanoma survival--role of NOX4.
description
2014 nî lūn-bûn
@nan
2014 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Redox modulation of FAK controls melanoma survival--role of NOX4.
@ast
Redox modulation of FAK controls melanoma survival--role of NOX4.
@en
type
label
Redox modulation of FAK controls melanoma survival--role of NOX4.
@ast
Redox modulation of FAK controls melanoma survival--role of NOX4.
@en
prefLabel
Redox modulation of FAK controls melanoma survival--role of NOX4.
@ast
Redox modulation of FAK controls melanoma survival--role of NOX4.
@en
P2093
P2860
P1433
P1476
Redox modulation of FAK controls melanoma survival--role of NOX4
@en
P2093
Christina Barja-Fidalgo
Cristiane Ribeiro-Pereira
Francisco R Laurindo
Maria Augusta Arruda
Mariele de Jesus Souza
P2860
P304
P356
10.1371/JOURNAL.PONE.0099481
P407
P577
2014-06-09T00:00:00Z