Parathyroid hormone receptor signaling in osteocytes increases the expression of fibroblast growth factor-23 in vitro and in vivo - Wikidata - thesis-toulmin - TriplyDB

Parathyroid hormone receptor signaling in osteocytes increases the expression of fibroblast growth factor-23 in vitro and in vivo

Parathyroid hormone receptor signaling in osteocytes increases the expression of fibroblast growth factor-23 in vitro and in vivo

http://www.wikidata.org/entity/Q35196515

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Effects of PTH on osteocyte function Skeletal secretion of FGF-23 regulates phosphate and vitamin D metabolism Bone disease in pediatric chronic kidney disease Fibroblast Growth Factor 23: A New Dimension to Diseases of Calcium-Phosphorus Metabolism Role of FGF23 in vitamin D and phosphate metabolism: implications in chronic kidney disease Cardiovascular Risk Factors and Chronic Kidney Disease-FGF23: A Key Molecule in the Cardiovascular Disease Deletion of PTH rescues skeletal abnormalities and high osteopontin levels in Klotho-/- mice Functional diversity of fibroblast growth factors in bone formation Relationship between Fibroblast Growth Factor 23 and Biochemical and Bone Histomorphometric Alterations in a Chronic Kidney Disease Rat Model Undergoing Parathyroidectomy Fibroblast growth factor 23 enhances renal klotho abundance 1,25-dihydroxyvitamin D(3) regulation of fibroblast growth factor-23 expression in bone cells: evidence for primary and secondary mechanisms modulated by leptin and interleukin-6 Fibroblast growth factor (Fgf) 23 gene transcription depends on actin cytoskeleton reorganization.Fibroblast growth factor 23 and Klotho: physiology and pathophysiology of an endocrine network of mineral metabolism Osteocytes mediate the anabolic actions of canonical Wnt/β-catenin signaling in bone.The use of fibroblast growth factor 23 testing in patients with kidney disease.Parathyroid hormone receptor signaling induces bone resorption in the adult skeleton by directly regulating the RANKL gene in osteocytes.FGF23 protein expression in coronary arteries is associated with impaired kidney function.Hypophosphatemic rickets: revealing novel control points for phosphate homeostasis.The human response to acute enteral and parenteral phosphate loads Renal phosphate wasting in the absence of adenylyl cyclase 6 Coupling fibroblast growth factor 23 production and cleavage: iron deficiency, rickets, and kidney disease.Anti-sclerostin antibody treatment in a rat model of progressive renal osteodystrophy.Osteocytic protein expression response to doxercalciferol therapy in pediatric dialysis patients.GNAS Spectrum of Disorders.Effect of Cinacalcet and Vitamin D Analogs on Fibroblast Growth Factor-23 during the Treatment of Secondary Hyperparathyroidism.Cell autonomous requirement of connexin 43 for osteocyte survival: consequences for endocortical resorption and periosteal bone formation.Arterial calcification and bone physiology: role of the bone-vascular axis.Metabolic acidosis increases fibroblast growth factor 23 in neonatal mouse bone Update on fibroblast growth factor 23 in chronic kidney disease.FGF23 is elevated in multiple myeloma and increases heparanase expression by tumor cells.Osteocytes and Skeletal Pathophysiology A Prospective Cohort Study of Mineral Metabolism After Kidney Transplantation.Parathyroid hormone 1 receptor is essential to induce FGF23 production and maintain systemic mineral ion homeostasis.Bovine parathyroid hormone enhances osteoclast bone resorption by modulating V-ATPase through PTH1R Renal Phosphate Reabsorption is Correlated with the Increase in Lumbar Bone Mineral Density in Patients Receiving Once-Weekly Teriparatide.Ablation of the Stimulatory G Protein α-Subunit in Renal Proximal Tubules Leads to Parathyroid Hormone-Resistance With Increased Renal Cyp24a1 mRNA Abundance and Reduced Serum 1,25-Dihydroxyvitamin D.Interactions between calcium and phosphorus in the regulation of the production of fibroblast growth factor 23 in vivo.Cardiovascular diseases in older patients with osteoporotic hip fracture: prevalence, disturbances in mineral and bone metabolism, and bidirectional links Fibroblast growth factor 23 and bone mineralisation.Systemic Control of Bone Homeostasis by FGF23 Signaling.

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P2860

Parathyroid hormone receptor signaling in osteocytes increases the expression of fibroblast growth factor-23 in vitro and in vivo

http://www.wikidata.org/entity/Q35196515

description

2011 nî lūn-bûn

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2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2011 թվականի հունիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Parathyroid hormone receptor s ...... factor-23 in vitro and in vivo

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Parathyroid hormone receptor s ...... factor-23 in vitro and in vivo

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type

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Parathyroid hormone receptor s ...... factor-23 in vitro and in vivo

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Parathyroid hormone receptor s ...... factor-23 in vitro and in vivo

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Parathyroid hormone receptor s ...... factor-23 in vitro and in vivo

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Parathyroid hormone receptor s ...... factor-23 in vitro and in vivo

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J.BONE.2011.06.025

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P1476

Parathyroid hormone receptor s ...... factor-23 in vitro and in vivo

@en

P2093

Emily Farrow

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Kenneth E White

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Lilian I Plotkin

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Nicoletta Bivi

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Virginia Lezcano

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Yumie Rhee

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P2860

Overexpression of fibroblast growth factor 23 suppresses osteoblast differentiation and matrix mineralization in vitro

Control of RANKL gene expression

Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia

Control of bone mass and remodeling by PTH receptor signaling in osteocytes

Proximal tubular phosphate reabsorption: molecular mechanisms

Human fibroblast growth factor-23 mutants suppress Na+-dependent phosphate co-transport activity and 1alpha,25-dihydroxyvitamin D3 production

Chronic elevation of parathyroid hormone in mice reduces expression of sclerostin by osteocytes: a novel mechanism for hormonal control of osteoblastogenesis

Klotho converts canonical FGF receptor into a specific receptor for FGF23

Genetic evidence of serum phosphate-independent functions of FGF-23 on bone

Homozygous ablation of fibroblast growth factor-23 results in hyperphosphatemia and impaired skeletogenesis, and reverses hypophosphatemia in Phex-deficient mice

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51464878

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