DeltaNp63α protein triggers epithelial-mesenchymal transition and confers stem cell properties in normal human keratinocytes.
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Therapeutic potential of mesenchymal stem cells for oral and systemic diseasesSensitive Tumorigenic Potential Evaluation of Adult Human Multipotent Neural Cells Immortalized by hTERT Gene TransductionΔNp63α and microRNAs: leveraging the epithelial-mesenchymal transitionTranscription factor p63 regulates key genes and wound repair in human airway epithelial basal cellsTrp63 is regulated by STAT5 in mammary tissue and subject to differentiation in cancer.Kaposi's sarcoma-associated herpesvirus ORF18 and ORF30 are essential for late gene expression during lytic replication.Periostin contributes to the acquisition of multipotent stem cell-like properties in human mammary epithelial cells and breast cancer cells.Enrichment of the Cancer Stem Phenotype in Sphere Cultures of Prostate Cancer Cell Lines Occurs through Activation of Developmental Pathways Mediated by the Transcriptional Regulator ΔNp63αThe p63 Gene Is Regulated by Grainyhead-like 2 (GRHL2) through Reciprocal Feedback and Determines the Epithelial Phenotype in Human KeratinocytesThe p63 protein isoform ΔNp63α inhibits epithelial-mesenchymal transition in human bladder cancer cells: role of MIR-205.The TP53 fertility network.Conditional ablation of p63 indicates that it is essential for embryonic development of the central nervous system.ΔNp63α Silences a miRNA Program to Aberrantly Initiate a Wound-Healing Program That Promotes TGFβ-Induced MetastasisPotentially malignant disorders of the oral cavity: current practice and future directions in the clinic and laboratory.Pulp-dentin Regeneration: Current State and Future Prospects.Basal cells of the human airways acquire mesenchymal traits in idiopathic pulmonary fibrosis and in culture.Dentin and dental pulp regeneration by the patient's endogenous cells.Lacrimal Gland Repair after Short-term Obstruction of Excretory Duct in Rabbits.NRP1-positive lung cancer cells possess tumor-initiating properties.Expression of the FGFR2c mesenchymal splicing variant in human keratinocytes inhibits differentiation and promotes invasion.Remodeling of epithelial cells and basement membranes in a corneal deficiency model with long-term follow-up.
P2860
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P2860
DeltaNp63α protein triggers epithelial-mesenchymal transition and confers stem cell properties in normal human keratinocytes.
description
2011 nî lūn-bûn
@nan
2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
DeltaNp63α protein triggers ep ...... in normal human keratinocytes.
@ast
DeltaNp63α protein triggers ep ...... in normal human keratinocytes.
@en
type
label
DeltaNp63α protein triggers ep ...... in normal human keratinocytes.
@ast
DeltaNp63α protein triggers ep ...... in normal human keratinocytes.
@en
prefLabel
DeltaNp63α protein triggers ep ...... in normal human keratinocytes.
@ast
DeltaNp63α protein triggers ep ...... in normal human keratinocytes.
@en
P2093
P2860
P356
P1476
DeltaNp63α protein triggers ep ...... in normal human keratinocytes.
@en
P2093
Ki-Hyuk Shin
No-Hee Park
Reuben H Kim
P2860
P304
38757-38767
P356
10.1074/JBC.M111.244939
P407
P577
2011-08-31T00:00:00Z