Contributions of mature granule cells to structural plasticity in temporal lobe epilepsy.
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Excessive activation of mTOR in postnatally generated granule cells is sufficient to cause epilepsy.Hippocampal granule cell pathology in epilepsy - a possible structural basis for comorbidities of epilepsy?Abnormalities of granule cell dendritic structure are a prominent feature of the intrahippocampal kainic acid model of epilepsy despite reduced postinjury neurogenesisMorphological alterations in newly born dentate gyrus granule cells that emerge after status epilepticus contribute to make them less excitableBlockade of excitatory synaptogenesis with proximal dendrites of dentate granule cells following rapamycin treatment in a mouse model of temporal lobe epilepsy.PTEN deletion from adult-generated dentate granule cells disrupts granule cell mossy fiber axon structure.Combined role of seizure-induced dendritic morphology alterations and spine loss in newborn granule cells with mossy fiber sprouting on the hyperexcitability of a computer model of the dentate gyrusClonal Analysis of Newborn Hippocampal Dentate Granule Cell Proliferation and Development in Temporal Lobe EpilepsyThe Ever-Changing Morphology of Hippocampal Granule Neurons in Physiology and PathologyImpact of rapamycin on status epilepticus induced hippocampal pathology and weight gain.Accumulation of abnormal adult-generated hippocampal granule cells predicts seizure frequency and severity.Neurosteroid interactions with synaptic and extrasynaptic GABA(A) receptors: regulation of subunit plasticity, phasic and tonic inhibition, and neuronal network excitability.Morphological changes among hippocampal dentate granule cells exposed to early kindling-epileptogenesis.Mechanisms regulating neuronal excitability and seizure development following mTOR pathway hyperactivation.Remodeling of dendrites and spines in the C1q knockout model of genetic epilepsy.PTEN deletion increases hippocampal granule cell excitability in male and female mice.Signaling Pathways and Cellular Mechanisms Regulating Mossy Fiber Sprouting in the Development of Epilepsy.Contributions of Adult-Generated Granule Cells to Hippocampal Pathology in Temporal Lobe Epilepsy: A Neuronal Bestiary
P2860
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P2860
Contributions of mature granule cells to structural plasticity in temporal lobe epilepsy.
description
2011 nî lūn-bûn
@nan
2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Contributions of mature granule cells to structural plasticity in temporal lobe epilepsy.
@ast
Contributions of mature granule cells to structural plasticity in temporal lobe epilepsy.
@en
type
label
Contributions of mature granule cells to structural plasticity in temporal lobe epilepsy.
@ast
Contributions of mature granule cells to structural plasticity in temporal lobe epilepsy.
@en
prefLabel
Contributions of mature granule cells to structural plasticity in temporal lobe epilepsy.
@ast
Contributions of mature granule cells to structural plasticity in temporal lobe epilepsy.
@en
P2093
P2860
P1433
P1476
Contributions of mature granule cells to structural plasticity in temporal lobe epilepsy.
@en
P2093
A W Loepke
B L Murphy
M S Hester
N Garcia-Cairasco
O W de Castro
S C Danzer
V R Santos
P2860
P304
P356
10.1016/J.NEUROSCIENCE.2011.09.034
P407
P577
2011-09-19T00:00:00Z