Soluble amyloid-β oligomers as synaptotoxins leading to cognitive impairment in Alzheimer's disease
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Fundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's disease in focal cerebral ischemic ratsFundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's diseaseDo Microglia Default on Network Maintenance in Alzheimer's Disease?Synaptopathies: synaptic dysfunction in neurological disorders - A review from students to studentsCellular levels of Grb2 and cytoskeleton stability are correlated in a neurodegenerative scenario.Isoflurane anesthesia promotes cognitive impairment by inducing expression of β-amyloid protein-related factors in the hippocampus of aged rats.Identifying N-linked glycan moiety and motifs in the cysteine-rich domain critical for N-glycosylation and intracellular trafficking of SR-AI and MARCO.Astaxanthin Protects Primary Hippocampal Neurons against Noxious Effects of Aβ-Oligomers.Protective effect of melatonin on soluble Aβ1-42-induced memory impairment, astrogliosis, and synaptic dysfunction via the Musashi1/Notch1/Hes1 signaling pathway in the rat hippocampus.Polymorphism of fibrillar structures depending on the size of assembled Aβ17-42 peptides.Protein Tyrosine Phosphatase 1B (PTP1B): A Potential Target for Alzheimer's Therapy?The Biological Function of the Prion Protein: A Cell Surface Scaffold of Signaling ModulesDietary arachidonic acid increases deleterious effects of amyloid-β oligomers on learning abilities and expression of AMPA receptors: putative role of the ACSL4-cPLA2 balance.The Amyloid Cascade Hypothesis in Alzheimer's Disease: It's Time to Change Our Mind.Kinesin-1 inhibits the aggregation of amyloid-β peptide as detected by fluorescence cross-correlation spectroscopy.Ion channel regulation by β-secretase BACE1 - enzymatic and non-enzymatic effects beyond Alzheimer's disease.Proteomic differences in amyloid plaques in rapidly progressive and sporadic Alzheimer's disease.Phosphorylation of the amyloid β-peptide at Ser26 stabilizes oligomeric assembly and increases neurotoxicity.Reelin: Neurodevelopmental Architect and Homeostatic Regulator of Excitatory Synapses.Tau Spread, Apolipoprotein E, Inflammation, and More: Rapidly Evolving Basic Science in Alzheimer Disease.The Associations between a Capsaicin-Rich Diet and Blood Amyloid-β Levels and Cognitive Function.Amyloid-β Impairs Vesicular Secretion in Neuronal and Astrocyte Peptidergic Transmission.Aberrant Co-localization of Synaptic Proteins Promoted by Alzheimer's Disease Amyloid-β Peptides: Protective Effect of Human Serum Albumin.Comparative pathobiology of β-amyloid and the unique susceptibility of humans to Alzheimer's diseaseCitalopram Ameliorates Impairments in Spatial Memory and Synaptic Plasticity in Female 3xTgAD MicePPAR Gamma Coactivator 1 Beta (PGC-1β) Reduces Mammalian Target of Rapamycin (mTOR) Expression via a SIRT1-Dependent Mechanism in Neurons.The Aβ oligomer eliminating D-enantiomeric peptide RD2 improves cognition without changing plaque pathology.Neurons derived from sporadic Alzheimer's disease iPSCs reveal elevated TAU hyperphosphorylation, increased amyloid levels, and GSK3B activation.BACE1 Function and Inhibition: Implications of Intervention in the Amyloid Pathway of Alzheimer's Disease Pathology.Large-Scale Oral Treatment Study with the Four Most Promising D3-Derivatives for the Treatment of Alzheimer's Disease.Taurine Directly Binds to Oligomeric Amyloid-β and Recovers Cognitive Deficits in Alzheimer Model Mice.Amyloid Beta monomers regulate cyclic adenosine monophosphate response element binding protein functions by activating type-1 insulin-like growth factor receptors in neuronal cells.Interaction of amyloid-β (Aβ) oligomers with neurexin 2α and neuroligin 1 mediates synapse damage and memory loss in mice.Thrombospondin-1 secreted by human umbilical cord blood-derived mesenchymal stem cells rescues neurons from synaptic dysfunction in Alzheimer's disease model.The Drosophila adult neuromuscular junction as a model for unravelling amyloid peptide influence on synapse dynamics.Challenges for Alzheimer's Disease Therapy: Insights from Novel Mechanisms Beyond Memory Defects.Focused ultrasound as a novel strategy for Alzheimer disease therapeutics.Mesenchymal stem cells and cell-derived extracellular vesicles protect hippocampal neurons from oxidative stress and synapse damage induced by amyloid-β oligomers.Apomorphine Therapy for Neuronal Insulin Resistance in a Mouse Model of Alzheimer's Disease.Counteracting the effects of TNF receptor-1 has therapeutic potential in Alzheimer's disease.
P2860
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P2860
Soluble amyloid-β oligomers as synaptotoxins leading to cognitive impairment in Alzheimer's disease
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Soluble amyloid-β oligomers as ...... airment in Alzheimer's disease
@ast
Soluble amyloid-β oligomers as ...... airment in Alzheimer's disease
@en
type
label
Soluble amyloid-β oligomers as ...... airment in Alzheimer's disease
@ast
Soluble amyloid-β oligomers as ...... airment in Alzheimer's disease
@en
prefLabel
Soluble amyloid-β oligomers as ...... airment in Alzheimer's disease
@ast
Soluble amyloid-β oligomers as ...... airment in Alzheimer's disease
@en
P2093
P2860
P356
P1476
Soluble amyloid-β oligomers as ...... airment in Alzheimer's disease
@en
P2093
Fernanda G De Felice
Mauricio M Oliveira
Sergio T Ferreira
P2860
P356
10.3389/FNCEL.2015.00191
P577
2015-05-26T00:00:00Z