The UPEC pore-forming toxin α-hemolysin triggers proteolysis of host proteins to disrupt cell adhesion, inflammatory, and survival pathways.
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Subversion of Host Innate Immunity by Uropathogenic Escherichia coliVirulence and Fitness Determinants of Uropathogenic Escherichia coliUrinary tract infections: epidemiology, mechanisms of infection and treatment optionsUropathogenic E. coli Exploit CEA to Promote Colonization of the Urogenital Tract MucosaPathogenesis of Streptococcus urinary tract infection depends on bacterial strain and β-hemolysin/cytolysin that mediates cytotoxicity, cytokine synthesis, inflammation and virulenceBacteria differentially induce degradation of Bcl-xL, a survival protein, by human plateletsDFI-seq identification of environment-specific gene expression in uropathogenic Escherichia coli.Cytotoxic Necrotizing Factor-1 (CNF1) does not promote E. coli infection in a murine model of ascending pyelonephritisPathogenesis of human diffusely adhering Escherichia coli expressing Afa/Dr adhesins (Afa/Dr DAEC): current insights and future challengesA conserved PapB family member, TosR, regulates expression of the uropathogenic Escherichia coli RTX nonfimbrial adhesin TosA while conserved LuxR family members TosE and TosF suppress motility.How to make it in the urinary tract: a tutorial by Escherichia coliBacterial lysis liberates the neutrophil migration suppressor YbcL from the periplasm of uropathogenic Escherichia coliFNR regulates expression of important virulence factors contributing to pathogenicity of uropathogenic Escherichia coli.Clinical features of bacterial vaginosis in a murine model of vaginal infection with Gardnerella vaginalisUropathogenic Escherichia coli superinfection enhances the severity of mouse bladder infection.Dysregulation of Escherichia coli α-hemolysin expression alters the course of acute and persistent urinary tract infectionRole of Hypoxia Inducible Factor-1α (HIF-1α) in Innate Defense against Uropathogenic Escherichia coli Infection.Inhibition of Cyclooxygenase-2 Prevents Chronic and Recurrent Cystitis.Structure of a bacterial toxin-activating acyltransferase.Switching Rho GTPase activation into effective antibacterial defenses requires the caspase-1/IL-1beta signaling axis.Establishment and Characterization of UTI and CAUTI in a Mouse Model.Uropathogenic Escherichia coli Releases Extracellular Vesicles That Are Associated with RNACampylobacter jejuni increases flagellar expression and adhesion of noninvasive Escherichia coli: effects on enterocytic Toll-like receptor 4 and CXCL-8 expression.Cytotoxic necrotizing factor 1 and hemolysin from uropathogenic Escherichia coli elicit different host responses in the murine bladderAssociation of O-Antigen Serotype with the Magnitude of Initial Systemic Cytokine Responses and Persistence in the Urinary Tract.Role of pore-forming toxins in neonatal sepsisMolecular Characterization of the Vacuolating Autotransporter Toxin in Uropathogenic Escherichia coli.Strengths and Limitations of Model Systems for the Study of Urinary Tract Infections and Related Pathologies.Uropathogenic Escherichia coli-Associated ExotoxinsLipopolysaccharide Domains Modulate Urovirulence.Natural killer cell-mediated host defense against uropathogenic E. coli is counteracted by bacterial hemolysinA-dependent killing of NK cells.Human milk oligosaccharides protect bladder epithelial cells against uropathogenic Escherichia coli invasion and cytotoxicity.Purine Biosynthesis Metabolically Constrains Intracellular Survival of Uropathogenic Escherichia coli.Invasion of Host Cells and Tissues by Uropathogenic Bacteria.Metabolomic analysis of siderophore cheater mutants reveals metabolic costs of expression in uropathogenic Escherichia coli.The RTX pore-forming toxin α-hemolysin of uropathogenic Escherichia coli: progress and perspectives.Uropathogenic Escherichia coli modulates innate immunity to suppress Th1-mediated inflammatory responses during infectious epididymitis.Urinary tract infection of mice to model human disease: Practicalities, implications and limitations.Cutaneous Immune Defenses Against Staphylococcus aureus Infections.The yin-yang driving urinary tract infection and how proteomics can enhance research, diagnostics, and treatment.
P2860
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P2860
The UPEC pore-forming toxin α-hemolysin triggers proteolysis of host proteins to disrupt cell adhesion, inflammatory, and survival pathways.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
The UPEC pore-forming toxin α- ...... matory, and survival pathways.
@ast
The UPEC pore-forming toxin α- ...... matory, and survival pathways.
@en
type
label
The UPEC pore-forming toxin α- ...... matory, and survival pathways.
@ast
The UPEC pore-forming toxin α- ...... matory, and survival pathways.
@en
prefLabel
The UPEC pore-forming toxin α- ...... matory, and survival pathways.
@ast
The UPEC pore-forming toxin α- ...... matory, and survival pathways.
@en
P2860
P1433
P1476
The UPEC pore-forming toxin α- ...... mmatory, and survival pathways
@en
P2093
Bijaya K Dhakal
P2860
P356
10.1016/J.CHOM.2011.12.003
P577
2012-01-01T00:00:00Z