Dual recognition of herpes simplex viruses by TLR2 and TLR9 in dendritic cells. - Wikidata - thesis-toulmin - TriplyDB

Dual recognition of herpes simplex viruses by TLR2 and TLR9 in dendritic cells.

Dual recognition of herpes simplex viruses by TLR2 and TLR9 in dendritic cells.

http://www.wikidata.org/entity/Q35768621

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Innate immune sensing of DNA viruses Understanding natural herpes simplex virus immunity to inform next-generation vaccine design Potential function of miRNAs in herpetic stromal keratitis The role of dendritic cells in immunity against primary herpes simplex virus infections Toll-like receptors: key players in antiviral immunity TLR cross-talk confers specificity to innate immunity Th1 Disabled Function in Response to TLR4 Stimulation of Monocyte-Derived DC from Patients Chronically-Infected by Hepatitis C Virus Toll-like receptors regulation of viral infection and disease☆Intrinsic Cellular Defenses against Virus Infection by Antiviral Type I Interferon A promiscuous lipid-binding protein diversifies the subcellular sites of toll-like receptor signal transduction.Herpesviruses dUTPases: A New Family of Pathogen-Associated Molecular Pattern (PAMP) Proteins with Implications for Human Disease Differential regulation of toll-like receptor-2, toll-like receptor-4, CD16 and human leucocyte antigen-DR on peripheral blood monocytes during mild and severe dengue fever.The virion host shut-off (vhs) protein blocks a TLR-independent pathway of herpes simplex virus type 1 recognition in human and mouse dendritic cells.Influence of perineurial cells and Toll-like receptors 2 and 9 on Herpes simplex type 1 entry to the central nervous system in rat encephalitis Herpes simplex type I (HSV-1) infection of the nervous system: is an immune response a good thing?Involvement of TLR2 in recognition of acute gammaherpesvirus-68 infection.Immunobiology of herpes simplex virus and cytomegalovirus infections of the fetus and newborn Optimal CD8 T-cell response against Encephalitozoon cuniculi is mediated by Toll-like receptor 4 upregulation by dendritic cells.Activation of NF-κB via endosomal Toll-like receptor 7 (TLR7) or TLR9 suppresses murine herpesvirus 68 reactivation.Herpes simplex virus immediate-early ICP0 protein inhibits Toll-like receptor 2-dependent inflammatory responses and NF-kappaB signaling Human herpesviruses-encoded dUTPases: a family of proteins that modulate dendritic cell function and innate immunity Toll-like receptor (TLR) 2 and TLR9 expressed in trigeminal ganglia are critical to viral control during herpes simplex virus 1 infection Milk matters: soluble Toll-like receptor 2 (sTLR2) in breast milk significantly inhibits HIV-1 infection and inflammation Identification of an important immunological difference between virulent varicella-zoster virus and its avirulent vaccine: viral disruption of dendritic cell instruction The herpes simplex virus 1-encoded envelope glycoprotein B activates NF-κB through the Toll-like receptor 2 and MyD88/TRAF6-dependent signaling pathway.In vitro synergism of trifluorothymidine and ganciclovir against HSV-1.TRAM is required for TLR2 endosomal signaling to type I IFN induction Epstein-Barr virus encoded dUTPase containing exosomes modulate innate and adaptive immune responses in human dendritic cells and peripheral blood mononuclear cells TLR4-MyD88/Mal-NF-kB axis is involved in infection of HSV-2 in human cervical epithelial cells.Human metapneumovirus M2-2 protein inhibits innate immune response in monocyte-derived dendritic cells HSV-2 increases TLR4-dependent phosphorylated IRFs and IFN-β induction in cervical epithelial cells dsRNA sensors and plasmacytoid dendritic cells in host defense and autoimmunity.Role of miR-155 in the pathogenesis of herpetic stromal keratitis.Evasion of early antiviral responses by herpes simplex viruses.Rapid genome assembly and comparison decode intrastrain variation in human alphaherpesviruses.Inflammatory monocyte effector mechanisms.The seroprevalence and salivary shedding of herpesviruses in Behçet's syndrome and recurrent aphthous stomatitis Role of specific innate immune responses in herpes simplex virus infection of the central nervous system IL-17A differentially regulates corneal vascular endothelial growth factor (VEGF)-A and soluble VEGF receptor 1 expression and promotes corneal angiogenesis after herpes simplex virus infection Chlamydial Pre-Infection Protects from Subsequent Herpes Simplex Virus-2 Challenge in a Murine Vaginal Super-Infection Model.

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Dual recognition of herpes simplex viruses by TLR2 and TLR9 in dendritic cells.

http://www.wikidata.org/entity/Q35768621

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年论文

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2006年论文

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name

Dual recognition of herpes simplex viruses by TLR2 and TLR9 in dendritic cells.

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Dual recognition of herpes simplex viruses by TLR2 and TLR9 in dendritic cells.

@en

type

label

Dual recognition of herpes simplex viruses by TLR2 and TLR9 in dendritic cells.

@ast

Dual recognition of herpes simplex viruses by TLR2 and TLR9 in dendritic cells.

@en

prefLabel

Dual recognition of herpes simplex viruses by TLR2 and TLR9 in dendritic cells.

@ast

Dual recognition of herpes simplex viruses by TLR2 and TLR9 in dendritic cells.

@en

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PNAS.0605102103

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Proceedings of the National Academy of Sciences of the United States of America

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Dual recognition of herpes simplex viruses by TLR2 and TLR9 in dendritic cells.

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Akiko Iwasaki

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Ayuko Sato

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Melissa M Linehan

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P2860

A Toll-like receptor recognizes bacterial DNA

Toll-like receptors 9 and 3 as essential components of innate immune defense against mouse cytomegalovirus infection

Differential roles of MDA5 and RIG-I helicases in the recognition of RNA viruses

Viruses and interferon: a fight for supremacy

Comparative usage of herpesvirus entry mediator A and nectin-1 by laboratory strains and clinical isolates of herpes simplex virus

Integral role of IRF-5 in the gene induction programme activated by Toll-like receptors

Differential roles of TLR2 and TLR4 in recognition of gram-negative and gram-positive bacterial cell wall components

IRF-7 is the master regulator of type-I interferon-dependent immune responses

The repertoire for pattern recognition of pathogens by the innate immune system is defined by cooperation between toll-like receptors

Hyporesponsiveness to vaccination with Borrelia burgdorferi OspA in humans and in TLR1- and TLR2-deficient mice

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103

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