Mutations in the N termini of herpes simplex virus type 1 and 2 gDs alter functional interactions with the entry/fusion receptors HVEM, nectin-2, and 3-O-sulfated heparan sulfate but not with nectin-1.
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Differences in the N termini of herpes simplex virus type 1 and 2 gDs that influence functional interactions with the human entry receptor Nectin-2 and an entry receptor expressed in Chinese hamster ovary cellsCharacterization of heparan sulphate 3-O-sulphotransferase isoform 6 and its role in assisting the entry of herpes simplex virus type 1Evolutionarily divergent herpesviruses modulate T cell activation by targeting the herpesvirus entry mediator cosignaling pathwayFour viruses, two bacteria, and one receptor: membrane cofactor protein (CD46) as pathogens' magnetViral entry mechanisms: cellular and viral mediators of herpes simplex virus entryThe principal neuronal gD-type 3-O-sulfotransferases and their products in central and peripheral nervous system tissuesStructure of Herpes Simplex Virus Glycoprotein D Bound to the Human Receptor Nectin-1Random mutagenesis of the gene encoding a viral ligand for multiple cell entry receptors to obtain viral mutants altered for receptor usage.Plasmodium falciparum BAEBL binds to heparan sulfate proteoglycans on the human erythrocyte surface.Heparan sulfate 3-O-sulfation: a rare modification in search of a function.Functional interaction between herpes simplex virus type 2 gD and HVEM transiently dampens local chemokine production after murine mucosal infectionHerpes virus fusion and entry: a story with many characters.Structure of unliganded HSV gD reveals a mechanism for receptor-mediated activation of virus entry.The nectin-1alpha transmembrane domain, but not the cytoplasmic tail, influences cell fusion induced by HSV-1 glycoproteins.A novel function of heparan sulfate in the regulation of cell-cell fusion.Generation of neutralizing aptamers against herpes simplex virus type 2: potential components of multivalent microbicides.Herpesvirus entry: an updateHerpesvirus entry mediator and nectin-1 mediate herpes simplex virus 1 infection of the murine cornea.Nectin-2-mediated entry of a syncytial strain of herpes simplex virus via pH-independent fusion with the plasma membrane of Chinese hamster ovary cells.Fusing structure and function: a structural view of the herpesvirus entry machinery.The soluble amino-terminal region of HVEM mediates efficient herpes simplex virus type 1 infection of gD receptor-negative cells.Herpes B virus utilizes human nectin-1 but not HVEM or PILRα for cell-cell fusion and virus entry.The amino terminus of varicella-zoster virus (VZV) glycoprotein E is required for binding to insulin-degrading enzyme, a VZV receptor.Alternative entry receptors for herpes simplex virus and their roles in diseaseHerpesvirus entry mediator on radiation-resistant cell lineages promotes ocular herpes simplex virus 1 pathogenesis in an entry-independent manner.Herpesvirus entry mediator is a serotype specific determinant of pathogenesis in ocular herpes.Effective treatment of an orthotopic xenograft model of human glioblastoma using an EGFR-retargeted oncolytic herpes simplex virusAn HSV-1 gD mutant virus as an entry-impaired live virus vaccine.B Virus (Macacine Herpesvirus 1) Divergence: Variations in Glycoprotein D from Clinical and Laboratory Isolates Diversify Virus Entry Strategies.Displacement of the C terminus of herpes simplex virus gD is sufficient to expose the fusion-activating interfaces on gD.Biography of Patricia G. Spear.Mutations in herpes simplex virus glycoprotein D that prevent cell entry via nectins and alter cell tropism.Murine Corneal Inflammation and Nerve Damage After Infection With HSV-1 Are Promoted by HVEM and Ameliorated by Immune-Modifying Nanoparticle Therapy.Induction of conformational changes at the N-terminus of herpes simplex virus glycoprotein D upon binding to HVEM and nectin-1Overexpression of herpes simplex virus glycoprotein K (gK) alters expression of HSV receptors in ocularly-infected micePotential nectin-1 binding site on herpes simplex virus glycoprotein d.Interaction of herpes simplex virus type 2 (HSV-2) glycoprotein D with the host cell surface is sufficient to induce Chlamydia trachomatis persistence.Herpes simplex virus infects most cell types in vitro: clues to its successMembers of 3-O-Sulfotransferases (3-OST) Family: A Valuable Tool from Zebrafish to Humans for Understanding Herpes Simplex Virus Entry.Interactions between herpesvirus entry mediator (TNFRSF14) and latency-associated transcript during herpes simplex virus 1 latency.
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Mutations in the N termini of herpes simplex virus type 1 and 2 gDs alter functional interactions with the entry/fusion receptors HVEM, nectin-2, and 3-O-sulfated heparan sulfate but not with nectin-1.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Mutations in the N termini of ...... sulfate but not with nectin-1.
@ast
Mutations in the N termini of ...... sulfate but not with nectin-1.
@en
type
label
Mutations in the N termini of ...... sulfate but not with nectin-1.
@ast
Mutations in the N termini of ...... sulfate but not with nectin-1.
@en
prefLabel
Mutations in the N termini of ...... sulfate but not with nectin-1.
@ast
Mutations in the N termini of ...... sulfate but not with nectin-1.
@en
P2093
P2860
P1433
P1476
Mutations in the N termini of ...... sulfate but not with nectin-1
@en
P2093
Patricia G Spear
P2860
P304
P356
10.1128/JVI.77.17.9221-9231.2003
P407
P577
2003-09-01T00:00:00Z