Lack of recognition by global-genome nucleotide excision repair accounts for the high mutagenicity and persistence of aristolactam-DNA adducts. - Wikidata - thesis-toulmin - TriplyDB

Lack of recognition by global-genome nucleotide excision repair accounts for the high mutagenicity and persistence of aristolactam-DNA adducts.

Lack of recognition by global-genome nucleotide excision repair accounts for the high mutagenicity and persistence of aristolactam-DNA adducts.

http://www.wikidata.org/entity/Q35860570

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Aristolochic acid-associated urothelial cancer in Taiwan Analysis of TP53 mutation spectra reveals the fingerprint of the potent environmental carcinogen, aristolochic acid Balkan endemic nephropathy: an update on its aetiology Nucleotide excision repair in humans Hypermutation in human cancer genomes: footprints and mechanisms TP53 and lacZ mutagenesis induced by 3-nitrobenzanthrone in Xpa-deficient human TP53 knock-in mouse embryo fibroblasts Ribonucleotides as nucleotide excision repair substrates.Aristolochic acid exposure in Romania and implications for renal cell carcinoma Nucleotide excision repair efficiencies of bulky carcinogen-DNA adducts are governed by a balance between stabilizing and destabilizing interactions Modelling mutational landscapes of human cancers in vitro.The relationships between XPC binding to conformationally diverse DNA adducts and their excision by the human NER system: is there a correlation?Bioactivation of the human carcinogen aristolochic acid Formalin-fixed paraffin-embedded tissue as a source for quantitation of carcinogen DNA adducts: aristolochic acid as a prototype carcinogen Genome-wide mutational signatures of aristolochic acid and its application as a screening tool.Mutational signature of aristolochic acid exposure as revealed by whole-exome sequencing.Adenine-DNA adduct derived from the nitroreduction of 6-nitrochrysene is more resistant to nucleotide excision repair than guanine-DNA adducts.New Approaches for Biomonitoring Exposure to the Human Carcinogen Aristolochic Acid.The efficiencies of damage recognition and excision correlate with duplex destabilization induced by acetylaminofluorene adducts in human nucleotide excision repair.Biomonitoring of aristolactam-DNA adducts in human tissues using ultra-performance liquid chromatography/ion-trap mass spectrometry.Adenine-DNA adducts derived from the highly tumorigenic Dibenzo[a,l]pyrene are resistant to nucleotide excision repair while guanine adducts are not Sulfotransferase-1A1-dependent bioactivation of aristolochic acid I and N-hydroxyaristolactam I in human cells.The growing use of herbal medicines: issues relating to adverse reactions and challenges in monitoring safety Human formalin-fixed paraffin-embedded tissues: an untapped specimen for biomonitoring of carcinogen DNA adducts by mass spectrometry.Mechanisms of base substitution mutagenesis in cancer genomes.Aristolochic acid nephropathy: Harbinger of a global iatrogenic disease.Potential genotoxicity of traditional chinese medicinal plants and phytochemicals: an overview.Naturally occurring aristolochic acid analogues and their toxicities.Historical perspective on the DNA damage response.Transcription-coupled repair: an update.Adenine versus guanine DNA adducts of aristolochic acids: role of the carcinogen-purine linkage in the differential global genomic repair propensity.Aristolochic Acid in the Etiology of Renal Cell Carcinoma.Altered minor-groove hydrogen bonds in DNA block transcription elongation by T7 RNA polymerase.Repair-Resistant DNA Lesions.DNA Adducts Formed by Aristolochic Acid Are Unique Biomarkers of Exposure and Explain the Initiation Phase of Upper Urothelial Cancer.Enhanced spontaneous DNA twisting/bending fluctuations unveiled by fluorescence lifetime distributions promote mismatch recognition by the Rad4 nucleotide excision repair complex.Aristolochic acids and their derivatives are widely implicated in liver cancers in Taiwan and throughout Asia.A Carcinogen-induced mouse model recapitulates the molecular alterations of human muscle invasive bladder cancer.Evidence of exposure to aristolochic acid in patients with urothelial cancer from a Balkan endemic nephropathy region of Romania.Exceptionally long-term persistence of DNA adducts formed by carcinogenic aristolochic acid I in renal tissue from patients with aristolochic acid nephropathy

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P2860

Lack of recognition by global-genome nucleotide excision repair accounts for the high mutagenicity and persistence of aristolactam-DNA adducts.

http://www.wikidata.org/entity/Q35860570

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2011 nî lūn-bûn

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Lack of recognition by global- ...... e of aristolactam-DNA adducts.

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Lack of recognition by global- ...... e of aristolactam-DNA adducts.

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Lack of recognition by global- ...... e of aristolactam-DNA adducts.

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Lack of recognition by global- ...... e of aristolactam-DNA adducts.

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Lack of recognition by global- ...... e of aristolactam-DNA adducts.

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Arthur P Grollman

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Francis Johnson

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Jung-Eun Yeo

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Orlando D Schärer

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Radha R Bonala

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Victoria S Sidorenko

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P2860

The ATPase domain but not the acidic region of Cockayne syndrome group B gene product is essential for DNA repair

Structural basis of UV DNA-damage recognition by the DDB1-DDB2 complex

Xeroderma pigmentosum group C protein complex is the initiator of global genome nucleotide excision repair

Xeroderma pigmentosum group F caused by a defect in a structure-specific DNA repair endonuclease

Two-step recognition of DNA damage for mammalian nucleotide excision repair: Directional binding of the XPC complex and DNA strand scanning

Aristolochic acid and the etiology of endemic (Balkan) nephropathy

A common mutational pattern in Cockayne syndrome patients from xeroderma pigmentosum group G: implications for a second XPG function

Site-specific incorporation of N-(deoxyguanosin-8-yl)-2-acetylaminofluorene (dG-AAF) into oligonucleotides using modified 'ultra-mild' DNA synthesis

CPD damage recognition by transcribing RNA polymerase II

Recognition of DNA damage by the Rad4 nucleotide excision repair protein

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2494-2505

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10.1093/NAR/GKR1095

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40

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51837947

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22121226

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3315299

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