Advanced glycation end products stimulate osteoblast apoptosis via the MAP kinase and cytosolic apoptotic pathways. - Wikidata - thesis-toulmin - TriplyDB

Advanced glycation end products stimulate osteoblast apoptosis via the MAP kinase and cytosolic apoptotic pathways.

Advanced glycation end products stimulate osteoblast apoptosis via the MAP kinase and cytosolic apoptotic pathways.

http://www.wikidata.org/entity/Q35878806

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Oxidative Stress and Dietary Fat Type in Relation to Periodontal Disease Diabetes mellitus related bone metabolism and periodontal disease Childhood obesity, bone development, and cardiometabolic risk factors Advanced glycation end products play adverse proinflammatory activities in osteoporosis Advanced glycation end products and diabetic complications Advanced Glycation End Products, Diabetes, and Bone Strength Diabetes, biochemical markers of bone turnover, diabetes control, and bone RAGE-dependent activation of gene expression of superoxide dismutase and vanins by AGE-rich extracts in mice cardiac tissue and murine cardiac fibroblasts.Osteoporosis and type 2 diabetes mellitus: what do we know, and what we can do?Precursor of advanced glycation end products mediates ER-stress-induced caspase-3 activation of human dermal fibroblasts through NAD(P)H oxidase 4 Evaluation of circulating sRAGE in osteoporosis according to BMI, adipokines and fracture risk: a pilot observational study.Anti-inflammatory Actions of Adjunctive Tetracyclines and Other Agents in Periodontitis and Associated Comorbidities.In situ accumulation of advanced glycation endproducts (AGEs) in bone matrix and its correlation with osteoclastic bone resorption.MiR-378 overexpression attenuates high glucose-suppressed osteogenic differentiation through targeting CASP3 and activating PI3K/Akt signaling pathway.Advanced glycation end products induce human corneal epithelial cells apoptosis through generation of reactive oxygen species and activation of JNK and p38 MAPK pathways.Diabetic complications and dysregulated innate immunity.Advanced glycation end products (AGEs) and their receptor (RAGE) induce apoptosis of periodontal ligament fibroblasts.FOXO1 differentially regulates both normal and diabetic wound healing.Effect of advanced glycosylation end products on apoptosis in human adipose tissue-derived stem cells in vitro.Effect of targeted delivery of bone morphogenetic protein-2 on bone formation in type 1 diabetes Resistance Training in Type II Diabetes Mellitus: Impact on Areas of Metabolic Dysfunction in Skeletal Muscle and Potential Impact on Bone.Pathophysiological role of enhanced bone marrow adipogenesis in diabetic complications.Influence of high glucose and advanced glycation end-products (ages) levels in human osteoblast-like cells gene expression.Advanced Glycation End Products Affect Osteoblast Proliferation and Function by Modulating Autophagy Via the Receptor of Advanced Glycation End Products/Raf Protein/Mitogen-activated Protein Kinase/Extracellular Signal-regulated Kinase Kinase/Extrac Augmented LPS responsiveness in type 1 diabetes-derived osteoclasts.Periodontal disease: the influence of metabolic syndrome Diabetes and Its Effect on Bone and Fracture Healing.Immunology of Osteoporosis: A Mini-Review.Transamidation by transglutaminase 2 transforms S100A11 calgranulin into a procatabolic cytokine for chondrocytes.Activation of the acquired immune response reduces coupled bone formation in response to a periodontal pathogen.Bone mechanical properties and changes with osteoporosis.Diabetes Stimulates Osteoclastogenesis by Acidosis-Induced Activation of Transient Receptor Potential Cation Channels.Association of hand or knee osteoarthritis with diabetes mellitus in a population of Hispanics from Puerto Rico RAGE and its ligands in bone metabolism.Type II collagen levels correlate with mineralization by articular cartilage vesicles.Advanced glycation end products induce cell cycle arrest and proinflammatory changes in osteoarthritic fibroblast-like synovial cells Metabolic syndrome and periodontitis: is oxidative stress a common link?Evaluation of bone mineral density among type 2 diabetes mellitus patients in South Karnataka Review: The diabetic bone: a cellular and molecular perspective.Diabetes mellitus and periodontitis: a tale of two common interrelated diseases.

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P2860

Advanced glycation end products stimulate osteoblast apoptosis via the MAP kinase and cytosolic apoptotic pathways.

http://www.wikidata.org/entity/Q35878806

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Advanced glycation end product ...... cytosolic apoptotic pathways.

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Advanced glycation end product ...... cytosolic apoptotic pathways.

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Advanced glycation end product ...... cytosolic apoptotic pathways.

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Advanced glycation end product ...... cytosolic apoptotic pathways.

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J.BONE.2006.09.011

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P1476

Advanced glycation end product ...... d cytosolic apoptotic pathways

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P2093

Christine M MacLellan

http://www.w3.org/2001/XMLSchema#string

Coy Boyd

http://www.w3.org/2001/XMLSchema#string

Dana T Graves

http://www.w3.org/2001/XMLSchema#string

Mani Alikhani

http://www.w3.org/2001/XMLSchema#string

Markos Raptis

http://www.w3.org/2001/XMLSchema#string

Nicole Pischon

http://www.w3.org/2001/XMLSchema#string

Rongkun Liu

http://www.w3.org/2001/XMLSchema#string

Zoubin Alikhani

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P2860

Non-enzymatic glycosylation of a type I collagen matrix: effects on osteoblastic development and oxidative stress

N(epsilon)-(carboxymethyl)lysine adducts of proteins are ligands for receptor for advanced glycation end products that activate cell signaling pathways and modulate gene expression

Mitogen-activated protein kinase pathways mediated by ERK, JNK, and p38 protein kinases

NF-kappaB antiapoptosis: induction of TRAF1 and TRAF2 and c-IAP1 and c-IAP2 to suppress caspase-8 activation

Alterations of cartilage and collagen expression during fracture healing in experimental diabetes.

2-ammonio-6-(3-oxidopyridinium-1-yl)hexanoate (OP-lysine) is a newly identified advanced glycation end product in cataractous and aged human lenses.

JNK/SAPK is required in nitric oxide-induced apoptosis in osteoblasts.

Osteoblast-derived PTHrP is a potent endogenous bone anabolic agent that modifies the therapeutic efficacy of administered PTH 1-34.

The biology of the receptor for advanced glycation end products and its ligands.

Pathogenesis of bone fragility in women and men.

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345-353

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10.1016/J.BONE.2006.09.011

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2

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40

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Philip C Trackman

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2006-10-24T00:00:00Z

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6731107

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17064973

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apoptotic process

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1913208

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