The p53 inhibitor pifithrin-α can stimulate fibrosis in a rat model of ischemic acute kidney injury.
about
Renocardiac syndromes: physiopathology and treatment stratagemsTherapeutic translation in acute kidney injury: the epithelial/endothelial axisMiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis.Regulation of necrotic cell death: p53, PARP1 and cyclophilin D-overlapping pathways of regulated necrosis?Recent advances in the understanding of acute kidney injury.Tubular p53 regulates multiple genes to mediate AKISustained oxidative stress causes late acute renal failure via duplex regulation on p38 MAPK and Akt phosphorylation in severely burned rats.Targeted deletion of p53 in the proximal tubule prevents ischemic renal injuryAstaxanthin attenuates early acute kidney injury following severe burns in rats by ameliorating oxidative stress and mitochondrial-related apoptosisEffects of hydrogen-rich saline on early acute kidney injury in severely burned rats by suppressing oxidative stress induced apoptosis and inflammation.Protective effect of quinacrine against glycerol-induced acute kidney injury in rats.Podocyte p53 Limits the Severity of Experimental Alport Syndrome.p53 is renoprotective after ischemic kidney injury by reducing inflammation.Novel application of complementary imaging techniques to examine in vivo glucose metabolism in the kidneyInhibition of Toll-Like Receptor 4 Signaling Mitigates Microvascular Loss but Not Fibrosis in a Model of Ischemic Acute Kidney InjuryRegulation of lung injury and fibrosis by p53-mediated changes in urokinase and plasminogen activator inhibitor-1.Melatonin prevents acute kidney injury in severely burned rats via the activation of SIRT1.Numb contributes to renal fibrosis by promoting tubular epithelial cell cycle arrest at G2/MA PTBA small molecule enhances recovery and reduces postinjury fibrosis after aristolochic acid-induced kidney injury.p53 induces miR199a-3p to suppress SOCS7 for STAT3 activation and renal fibrosis in UUO.Molecular basis of organ fibrosis: potential therapeutic approaches.Hypoxia as a key player in the AKI-to-CKD transition.A perspective on chronic kidney disease progression.Mitochondria Protection after Acute Ischemia Prevents Prolonged Upregulation of IL-1β and IL-18 and Arrests CKD.AKI on CKD: heightened injury, suppressed repair, and the underlying mechanisms.Cancer driver mutations in endometriosis: Variations on the major theme of fibrogenesis
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The p53 inhibitor pifithrin-α can stimulate fibrosis in a rat model of ischemic acute kidney injury.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
The p53 inhibitor pifithrin-α ...... ischemic acute kidney injury.
@ast
The p53 inhibitor pifithrin-α ...... ischemic acute kidney injury.
@en
type
label
The p53 inhibitor pifithrin-α ...... ischemic acute kidney injury.
@ast
The p53 inhibitor pifithrin-α ...... ischemic acute kidney injury.
@en
prefLabel
The p53 inhibitor pifithrin-α ...... ischemic acute kidney injury.
@ast
The p53 inhibitor pifithrin-α ...... ischemic acute kidney injury.
@en
P2093
P2860
P1476
The p53 inhibitor pifithrin-α ...... f ischemic acute kidney injury
@en
P2093
Erik M Mai
Henry E Mang
Melissa D Anderson
Nicole L Knipe
Pierre C Dagher
So-Young Lee
Stephanie C Karozos
Zoya Plotkin
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P304
P356
10.1152/AJPRENAL.00317.2011
P577
2011-11-02T00:00:00Z