IGFBP-3 may trigger osteoarthritis by inducing apoptosis of chondrocytes through Nur77 translocation

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IGFBP-3 may trigger osteoarthritis by inducing apoptosis of chondrocytes through Nur77 translocation

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2015年論文

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IGFBP-3 may trigger osteoarthr ...... es through Nur77 translocation

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IGFBP-3 may trigger osteoarthr ...... es through Nur77 translocation

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IGFBP-3 may trigger osteoarthr ...... es through Nur77 translocation

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IGFBP-3 may trigger osteoarthr ...... es through Nur77 translocation

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IGFBP-3 may trigger osteoarthr ...... es through Nur77 translocation

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IGFBP-3 may trigger osteoarthr ...... es through Nur77 translocation

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P1476

IGFBP-3 may trigger osteoarthr ...... es through Nur77 translocation

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P2093

Hao-Huan Li

http://www.w3.org/2001/XMLSchema#string

Zhun Wei

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P2860

A current review of molecular mechanisms regarding osteoarthritis and pain

Cellular internalization of insulin-like growth factor binding protein-3: distinct endocytic pathways facilitate re-uptake and nuclear localization

IGF binding proteins in cancer: mechanistic and clinical insights

Dual effects of IGFBP-3 on endothelial cell apoptosis and survival: involvement of the sphingolipid signaling pathways

The IGF axis and programmed cell death.

Toward regeneration of articular cartilage.

Nuclear export and mitochondrial and endoplasmic reticulum localization of IGF-binding protein 3 regulate its apoptotic properties.

Genome-wide association and functional studies identify a role for IGFBP3 in hip osteoarthritis.

Growth factor inhibition: potential role in the etiopathogenesis of osteoarthritis.

p53 and Nur77/TR3 - transcription factors that directly target mitochondria for cell death induction.

P304

15599-15610

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P31

scholarly article

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2015-12-01T00:00:00Z

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P698

26884829

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P921

osteoarthritis

P932

4730042

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