Ablation of XP-V gene causes adipose tissue senescence and metabolic abnormalities.
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Neuroendorine and Epigentic Mechanisms Subserving Autonomic Imbalance and HPA Dysfunction in the Metabolic SyndromePI3K-resistant GSK3 controls adiponectin formation and protects from metabolic syndromeHepatitis B virus X protein (HBx)-induced abnormalities of nucleic acid metabolism revealed by (1)H-NMR-based metabonomicsEnhanced expression of Survivin has distinct roles in adipocyte homeostasisThe ominous triad of adipose tissue dysfunction: inflammation, fibrosis, and impaired angiogenesis.Palmitic acid, but not high-glucose, induced myocardial apoptosis is alleviated by N‑acetylcysteine due to attenuated mitochondrial-derived ROS accumulation-induced endoplasmic reticulum stress.
P2860
Ablation of XP-V gene causes adipose tissue senescence and metabolic abnormalities.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
Ablation of XP-V gene causes adipose tissue senescence and metabolic abnormalities.
@ast
Ablation of XP-V gene causes adipose tissue senescence and metabolic abnormalities.
@en
type
label
Ablation of XP-V gene causes adipose tissue senescence and metabolic abnormalities.
@ast
Ablation of XP-V gene causes adipose tissue senescence and metabolic abnormalities.
@en
prefLabel
Ablation of XP-V gene causes adipose tissue senescence and metabolic abnormalities.
@ast
Ablation of XP-V gene causes adipose tissue senescence and metabolic abnormalities.
@en
P2093
P2860
P356
P1476
Ablation of XP-V gene causes adipose tissue senescence and metabolic abnormalities.
@en
P2093
Kai-ming Chou
Robert A Harris
Yih-Wen Chen
Zafer Hatahet
P2860
P304
P356
10.1073/PNAS.1506954112
P407
P577
2015-08-03T00:00:00Z