Molecular mechanisms responsible for the selective and low-grade induction of proinflammatory mediators in murine macrophages by lipopolysaccharide
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Fundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's disease in focal cerebral ischemic ratsFundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's diseaseRole of gut microbiota in the modulation of atherosclerosis-associated immune responseNeuroinflammation in Alzheimer's diseaseHigh Fat High Cholesterol Diet (Western Diet) Aggravates Atherosclerosis, Hyperglycemia and Renal Failure in Nephrectomized LDL Receptor Knockout Mice: Role of Intestine Derived LipopolysaccharideToll-Interacting Protein in Resolving and Non-Resolving Inflammation.Molecular and cellular mechanisms responsible for cellular stress and low-grade inflammation induced by a super-low dose of endotoxinInnate immune programing by endotoxin and its pathological consequencesAlteration of lysosome fusion and low-grade inflammation mediated by super-low-dose endotoxin.Myeloid cell-derived inducible nitric oxide synthase suppresses M1 macrophage polarization.Super-low dose endotoxin pre-conditioning exacerbates sepsis mortality.Protein Phosphatase 2A in Lipopolysaccharide-Induced Cyclooxygenase-2 Expression in Murine Lymphatic Endothelial Cells.Critical role of phospholipase A2 group IID in age-related susceptibility to severe acute respiratory syndrome-CoV infection.An Exploratory Investigation of Endotoxin Levels in Novice Long Distance Triathletes, and the Effects of a Multi-Strain Probiotic/Prebiotic, Antioxidant Intervention.Molecular mechanisms responsible for the reduced expression of cholesterol transporters from macrophages by low-dose endotoxinLow-grade inflammatory polarization of monocytes impairs wound healingMorphine induced exacerbation of sepsis is mediated by tempering endotoxin tolerance through modulation of miR-146a.IRAK4 kinase activity is not required for induction of endotoxin tolerance but contributes to TLR2-mediated toleranceMicroglia and macrophages of the central nervous system: the contribution of microglia priming and systemic inflammation to chronic neurodegeneration.Deacetylation by SIRT1 Reprograms Inflammation and CancerDeficiency in Toll-interacting protein (Tollip) skews inflamed yet incompetent innate leukocytes in vivo during DSS-induced septic colitisThe persistence of low-grade inflammatory monocytes contributes to aggravated atherosclerosis.Intranasal immunization of lambs with serine/threonine phosphatase 2A against gastrointestinal nematodes.Tollip, an early regulator of the acute inflammatory response in the substantia nigraCellular mosaicism for X-linked polymorphisms and IRAK1 expression presents a distinct phenotype and improves survival following sepsisMitochondria: sensors and mediators of innate immune receptor signaling.Microglial priming in neurodegenerative disease.The Characteristics and Function of Bacterial Lipopolysaccharides and Their Endotoxic Potential in Humans.Chasing a ghost?--Issues with the determination of circulating levels of endotoxin in human blood.Dynamic modulation of innate immunity programming and memory.Tollip Deficiency Alters Atherosclerosis and Steatosis by Disrupting Lipophagy.Preventing Overheating: Tight Control of Gut Innate Immunity in Health and Disease.Differential effects of low and high doses of lipoteichoic acid on lipopolysaccharide-induced interleukin-6 production.Programming and memory dynamics of innate leukocytes during tissue homeostasis and inflammation.Trained innate immunity as a mechanistic link between sepsis and atherosclerosis.Potent suppression of arginase 1 expression in murine macrophages by low dose endotoxin.Inherent X-Linked Genetic Variability and Cellular Mosaicism Unique to Females Contribute to Sex-Related Differences in the Innate Immune Response.Retinal cell type-specific prevention of ischemia-induced damages by LPS-TLR4 signaling through microglia.The Traditional Medicinal Plants Cuphea calophylla, Tibouchina kingii, and Pseudelephantopus spiralis Attenuate Inflammatory and Oxidative Mediators.The High Costs of Low-Grade Inflammation: Persistent Fatigue as a Consequence of Reduced Cellular-Energy Availability and Non-adaptive Energy Expenditure.
P2860
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P2860
Molecular mechanisms responsible for the selective and low-grade induction of proinflammatory mediators in murine macrophages by lipopolysaccharide
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Molecular mechanisms responsib ...... rophages by lipopolysaccharide
@ast
Molecular mechanisms responsib ...... rophages by lipopolysaccharide
@en
type
label
Molecular mechanisms responsib ...... rophages by lipopolysaccharide
@ast
Molecular mechanisms responsib ...... rophages by lipopolysaccharide
@en
prefLabel
Molecular mechanisms responsib ...... rophages by lipopolysaccharide
@ast
Molecular mechanisms responsib ...... rophages by lipopolysaccharide
@en
P2093
P2860
P356
P1476
Molecular mechanisms responsib ...... rophages by lipopolysaccharide
@en
P2093
Bianca Baker
Daniel G S Capelluto
Trevor Glaros
Urmila Maitra
P2860
P304
P356
10.4049/JIMMUNOL.1200857
P407
P50
P577
2012-06-15T00:00:00Z