Tristetraprolin inhibits Ras-dependent tumor vascularization by inducing vascular endothelial growth factor mRNA degradation.
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Sublytic membrane-attack-complex (MAC) activation alters regulated rather than constitutive vascular endothelial growth factor (VEGF) secretion in retinal pigment epithelium monolayersHallmarks of cancer and AU-rich elementsRole of hypoxia and vascular endothelial growth factors in lymphangiogenesisKaposin-B enhances the PROX1 mRNA stability during lymphatic reprogramming of vascular endothelial cells by Kaposi's sarcoma herpes virus.Stability of the LATS2 tumor suppressor gene is regulated by tristetraprolinRegulation of the mRNA half-life in breast cancerPost-transcriptional control during chronic inflammation and cancer: a focus on AU-rich elements.Coordinated expression of tristetraprolin post-transcriptionally attenuates mitogenic induction of the oncogenic Ser/Thr kinase Pim-1.Downregulation of the AU-rich RNA-binding protein ZFP36 in chronic HBV patients: implications for anti-inflammatory therapy.EGF activates TTP expression by activation of ELK-1 and EGR-1 transcription factorsSuppression of laser-induced choroidal neovascularization by intravitreal injection of tristetraprolin.Microarray profiling of mononuclear peripheral blood cells identifies novel candidate genes related to chemoradiation response in rectal cancer.Transcriptome sequencing reveals differences between primary and secondary hair follicle-derived dermal papilla cells of the Cashmere goat (Capra hircus).Caffeine mediates sustained inactivation of breast cancer-associated myofibroblasts via up-regulation of tumor suppressor genes.Hypoxia-inducible factor-1α mRNA: a new target for destabilization by tristetraprolin in endothelial cells.Drosophila eyes absent is a novel mRNA target of the tristetraprolin (TTP) protein DTIS11The RNA-binding zinc-finger protein tristetraprolin regulates AU-rich mRNAs involved in breast cancer-related processes.Tristetraprolin regulation of interleukin-22 productionHsp70 is a novel posttranscriptional regulator of gene expression that binds and stabilizes selected mRNAs containing AU-rich elements.p16INK4A represses breast stromal fibroblasts migration/invasion and their VEGF-A-dependent promotion of angiogenesis through Akt inhibition.Antagonistic function of the RNA-binding protein HuR and miR-200b in post-transcriptional regulation of vascular endothelial growth factor-A expression and angiogenesis.mTOR regulates cellular iron homeostasis through tristetraprolin.Genome-wide analysis identifies interleukin-10 mRNA as target of tristetraprolinGene regulation by RNA binding proteins and microRNAs in angiogenesis.Tristetraprolin (TTP): interactions with mRNA and proteins, and current thoughts on mechanisms of action.TIS11 family proteins and their roles in posttranscriptional gene regulation.Three Residues Make an Evolutionary Switch for Folding and RNA-Destabilizing Activity in the TTP Family of ProteinsInflammation: cytokines and RNA-based regulation.RNA binding protein, tristetraprolin in a murine model of recurrent pregnancy loss.Targeting the pro-angiogenic forms of VEGF or inhibiting their expression as anti-cancer strategies.Tristetraprolin: roles in cancer and senescence.The role of the 3' untranslated region in post-transcriptional regulation of protein expression in mammalian cells.Multiple functions of tristetraprolin/TIS11 RNA-binding proteins in the regulation of mRNA biogenesis and degradation.Role of hypoxia and vascular endothelial growth factors in lymphangiogenesis.Post-transcriptional regulation of cytokine and growth factor signaling in cancer.A synonymous polymorphism of the Tristetraprolin (TTP) gene, an AU-rich mRNA-binding protein, affects translation efficiency and response to Herceptin treatment in breast cancer patients.Constitutive ERK activity induces downregulation of tristetraprolin, a major protein controlling interleukin8/CXCL8 mRNA stability in melanoma cells.Casein kinase 2 regulates the mRNA-destabilizing activity of tristetraprolin.The mRNA-destabilizing protein tristetraprolin is suppressed in many cancers, altering tumorigenic phenotypes and patient prognosis.Identification of DEAD-box RNA helicase 6 (DDX6) as a cellular modulator of vascular endothelial growth factor expression under hypoxia.
P2860
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P2860
Tristetraprolin inhibits Ras-dependent tumor vascularization by inducing vascular endothelial growth factor mRNA degradation.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Tristetraprolin inhibits Ras-d ...... rowth factor mRNA degradation.
@ast
Tristetraprolin inhibits Ras-d ...... rowth factor mRNA degradation.
@en
type
label
Tristetraprolin inhibits Ras-d ...... rowth factor mRNA degradation.
@ast
Tristetraprolin inhibits Ras-d ...... rowth factor mRNA degradation.
@en
prefLabel
Tristetraprolin inhibits Ras-d ...... rowth factor mRNA degradation.
@ast
Tristetraprolin inhibits Ras-d ...... rowth factor mRNA degradation.
@en
P2093
P2860
P356
P1476
Tristetraprolin inhibits Ras-d ...... rowth factor mRNA degradation.
@en
P2093
Cercina Onesto
Christoph Moroni
Emmanuelle Stebe
Gilles Pagès
Khadija Essafi-Benkhadir
P2860
P304
P356
10.1091/MBC.E07-06-0570
P577
2007-09-12T00:00:00Z