CXCR2-dependent mucosal neutrophil influx protects against colitis-associated diarrhea caused by an attaching/effacing lesion-forming bacterial pathogen.
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Disruptions of Host Immunity and Inflammation by Giardia Duodenalis: Potential Consequences for Co-Infections in the Gastro-Intestinal TractInnate immune signaling in defense against intestinal microbesCXCL9 contributes to antimicrobial protection of the gut during citrobacter rodentium infection independent of chemokine-receptor signalingGiardia duodenalis infection reduces granulocyte infiltration in an in vivo model of bacterial toxin-induced colitis and attenuates inflammation in human intestinal tissueMultivariate modeling identifies neutrophil- and Th17-related factors as differential serum biomarkers of chronic murine colitis.Lymphotoxin beta receptor signaling in intestinal epithelial cells orchestrates innate immune responses against mucosal bacterial infection.Neutrophil elastase causes tissue damage that decreases host tolerance to lung infection with burkholderia species.Rac2-deficiency leads to exacerbated and protracted colitis in response to Citrobacter rodentium infectionChemokine receptor CXCR2 mediates bacterial clearance rather than neutrophil recruitment in a murine model of pneumonic plagueEpithelial phosphatidylinositol-3-kinase signaling is required for β-catenin activation and host defense against Citrobacter rodentium infection.Guanylate cyclase C limits systemic dissemination of a murine enteric pathogen.A balanced IL-1β activity is required for host response to Citrobacter rodentium infection.Innate immune responses to systemic Acinetobacter baumannii infection in mice: neutrophils, but not interleukin-17, mediate host resistanceConstitutive intestinal NF-κB does not trigger destructive inflammation unless accompanied by MAPK activation.Interleukin-1 (IL-1) signaling in intestinal stromal cells controls KC/ CXCL1 secretion, which correlates with recruitment of IL-22- secreting neutrophils at early stages of Citrobacter rodentium infection.Tir Triggers Expression of CXCL1 in Enterocytes and Neutrophil Recruitment during Citrobacter rodentium InfectionSalmonella-induced Diarrhea Occurs in the Absence of IL-8 Receptor (CXCR2)-Dependent Neutrophilic InflammationIL-17A promotes protective IgA responses and expression of other potential effectors against the lumen-dwelling enteric parasite Giardia.Citrobacter rodentium mouse model of bacterial infection.The sphingosine-1-phosphate analogue FTY720 impairs mucosal immunity and clearance of the enteric pathogen Citrobacter rodentium.Enhanced susceptibility to Citrobacter rodentium infection in microRNA-155-deficient miceHVEM: An unusual TNF receptor family member important for mucosal innate immune responses to microbes.IL-22-producing neutrophils contribute to antimicrobial defense and restitution of colonic epithelial integrity during colitis.Yersinia enterocolitica YopH-Deficient Strain Activates Neutrophil Recruitment to Peyer's Patches and Promotes Clearance of the Virulent Strain.Altered expression and localization of ion transporters contribute to diarrhea in mice with Salmonella-induced enteritis.Bacterial virulence factor inhibits caspase-4/11 activation in intestinal epithelial cells.Enteric pathogens and gut function: Role of cytokines and STATsAlways one step ahead: How pathogenic bacteria use the type III secretion system to manipulate the intestinal mucosal immune system.Crosstalk between the intestinal microbiota and the innate immune system in intestinal homeostasis and inflammatory bowel disease.Microbiome-mediated neutrophil recruitment via CXCR2 and protection from amebic colitis.Tpl2 promotes innate cell recruitment and effector T cell differentiation to limit Citrobacter rodentium burden and dissemination.Interleukin-8, CXCL1, and MicroRNA miR-146a Responses to Probiotic Escherichia coli Nissle 1917 and Enteropathogenic E. coli in Human Intestinal Epithelial T84 and Monocytic THP-1 Cells after Apical or Basolateral Infection.Ficolin-A/2, acting as a new regulator of macrophage polarization, mediates the inflammatory response in experimental mouse colitis.NLRC4 expression in intestinal epithelial cells mediates protection against an enteric pathogen.Cxcr2 signaling and the microbiome suppress inflammation, bile duct injury, and the phenotype of experimental biliary atresia.Tpl2 promotes neutrophil trafficking, oxidative burst, and bacterial killing.Steroid Receptor Coactivator 3 Contributes to Host Defense against Enteric Bacteria by Recruiting Neutrophils via Upregulation of CXCL2 Expression.Platelet-Derived CCL5 Regulates CXC Chemokine Formation and Neutrophil Recruitment in Acute Experimental Colitis.Properdin Regulation of Complement Activation Affects Colitis in Interleukin 10 Gene-Deficient Mice.A deficiency in the autophagy gene Atg16L1 enhances resistance to enteric bacterial infection.
P2860
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P2860
CXCR2-dependent mucosal neutrophil influx protects against colitis-associated diarrhea caused by an attaching/effacing lesion-forming bacterial pathogen.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
CXCR2-dependent mucosal neutro ...... on-forming bacterial pathogen.
@ast
CXCR2-dependent mucosal neutro ...... on-forming bacterial pathogen.
@en
type
label
CXCR2-dependent mucosal neutro ...... on-forming bacterial pathogen.
@ast
CXCR2-dependent mucosal neutro ...... on-forming bacterial pathogen.
@en
prefLabel
CXCR2-dependent mucosal neutro ...... on-forming bacterial pathogen.
@ast
CXCR2-dependent mucosal neutro ...... on-forming bacterial pathogen.
@en
P2093
P2860
P356
P1476
CXCR2-dependent mucosal neutro ...... ion-forming bacterial pathogen
@en
P2093
Declan F McCole
Elaine Hanson
Lars Eckmann
Martina E Spehlmann
P2860
P304
P356
10.4049/JIMMUNOL.0900600
P407
P50
P577
2009-08-12T00:00:00Z